Ralph Schwenzer

983 total citations
9 papers, 873 citations indexed

About

Ralph Schwenzer is a scholar working on Molecular Biology, Cancer Research and Immunology. According to data from OpenAlex, Ralph Schwenzer has authored 9 papers receiving a total of 873 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 7 papers in Cancer Research and 4 papers in Immunology. Recurrent topics in Ralph Schwenzer's work include NF-κB Signaling Pathways (7 papers), Cell death mechanisms and regulation (6 papers) and interferon and immune responses (2 papers). Ralph Schwenzer is often cited by papers focused on NF-κB Signaling Pathways (7 papers), Cell death mechanisms and regulation (6 papers) and interferon and immune responses (2 papers). Ralph Schwenzer collaborates with scholars based in Germany, Switzerland and Austria. Ralph Schwenzer's co-authors include Harald Wajant, G Schubert, Peter Scheurich, Elvira Haas, Frank Mühlenbeck, Matthias Grell, Pascal Schneider, Nathalie Peters, Roland M. Schmid and Susanne Liptay and has published in prestigious journals such as Journal of Biological Chemistry, Current Biology and European Journal of Immunology.

In The Last Decade

Ralph Schwenzer

9 papers receiving 855 citations

Peers

Ralph Schwenzer
Dirk De Valck Belgium
Frank Mühlenbeck Germany
Astrid Strelow Germany
Anneke Dörrie Germany
Nisha H. Kabra United States
Uta Schaefer Germany
Adam B. Agranoff United States
Jonathan C. Betts United States
Thomas Raven United Kingdom
Felix Freuler Switzerland
Dirk De Valck Belgium
Ralph Schwenzer
Citations per year, relative to Ralph Schwenzer Ralph Schwenzer (= 1×) peers Dirk De Valck

Countries citing papers authored by Ralph Schwenzer

Since Specialization
Citations

This map shows the geographic impact of Ralph Schwenzer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ralph Schwenzer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ralph Schwenzer more than expected).

Fields of papers citing papers by Ralph Schwenzer

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ralph Schwenzer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ralph Schwenzer. The network helps show where Ralph Schwenzer may publish in the future.

Co-authorship network of co-authors of Ralph Schwenzer

This figure shows the co-authorship network connecting the top 25 collaborators of Ralph Schwenzer. A scholar is included among the top collaborators of Ralph Schwenzer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ralph Schwenzer. Ralph Schwenzer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

9 of 9 papers shown
1.
Schwenzer, Ralph, et al.. (2008). Implementation of a semi-automated processing system for DNA profiling of forensic casework samples. Forensic science international. Genetics supplement series. 1(1). 83–85. 4 indexed citations
2.
Schwenzer, Ralph, et al.. (2008). Implementation of a robotized real-time PCR setup for the use of the Quantifiler™ Human DNA Quantification Kit. Forensic science international. Genetics supplement series. 1(1). 68–70. 3 indexed citations
3.
Henkler, Frank, Bernd Baumann, Mariola Fotin‐Mleczek, et al.. (2003). Caspase-mediated Cleavage Converts the Tumor Necrosis Factor (TNF) Receptor-associated Factor (TRAF)-1 from a Selective Modulator of TNF Receptor Signaling to a General Inhibitor of NF-κB Activation. Journal of Biological Chemistry. 278(31). 29216–29230. 38 indexed citations
4.
Wajant, Harald, Elvira Haas, Ralph Schwenzer, et al.. (2000). Inhibition of Death Receptor-mediated Gene Induction by a Cycloheximide-sensitive Factor Occurs at the Level of or Upstream of Fas-associated Death Domain Protein (FADD). Journal of Biological Chemistry. 275(32). 24357–24366. 154 indexed citations
5.
Mühlenbeck, Frank, Pascal Schneider, Jean-Luc Bodmer, et al.. (2000). The Tumor Necrosis Factor-related Apoptosis-inducing Ligand Receptors TRAIL-R1 and TRAIL-R2 Have Distinct Cross-linking Requirements for Initiation of Apoptosis and Are Non-redundant in JNK Activation. Journal of Biological Chemistry. 275(41). 32208–32213. 128 indexed citations
6.
Schneider, Pascal, Ralph Schwenzer, Elvira Haas, et al.. (1999). TWEAK can induce cell death via endogenous TNF and TNF receptor 1. European Journal of Immunology. 29(6). 1785–1792. 132 indexed citations
7.
Schwenzer, Ralph, Susanne Liptay, G Schubert, et al.. (1999). The Human Tumor Necrosis Factor (TNF) Receptor-associated Factor 1 Gene (TRAF1) Is Up-regulated by Cytokines of the TNF Ligand Family and Modulates TNF-induced Activation of NF-κB and c-Jun N-terminal Kinase. Journal of Biological Chemistry. 274(27). 19368–19374. 154 indexed citations
8.
Wajant, Harald, Franz‐Josef Johannes, Elvira Haas, et al.. (1998). Dominant-negative FADD inhibits TNFR60-, Fas/Apo1-  and TRAIL-R/Apo2-mediated cell death but not gene induction. Current Biology. 8(2). 113–116. 135 indexed citations
9.
Mühlenbeck, Frank, Elvira Haas, Ralph Schwenzer, et al.. (1998). TRAIL/Apo2L Activates c-Jun NH2-terminal Kinase (JNK) via Caspase-dependent and Caspase-independent Pathways. Journal of Biological Chemistry. 273(49). 33091–33098. 125 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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