Raekil Park

3.0k total citations · 1 hit paper
84 papers, 2.3k citations indexed

About

Raekil Park is a scholar working on Molecular Biology, Epidemiology and Sensory Systems. According to data from OpenAlex, Raekil Park has authored 84 papers receiving a total of 2.3k indexed citations (citations by other indexed papers that have themselves been cited), including 44 papers in Molecular Biology, 22 papers in Epidemiology and 15 papers in Sensory Systems. Recurrent topics in Raekil Park's work include Hearing, Cochlea, Tinnitus, Genetics (15 papers), Autophagy in Disease and Therapy (15 papers) and Peroxisome Proliferator-Activated Receptors (13 papers). Raekil Park is often cited by papers focused on Hearing, Cochlea, Tinnitus, Genetics (15 papers), Autophagy in Disease and Therapy (15 papers) and Peroxisome Proliferator-Activated Receptors (13 papers). Raekil Park collaborates with scholars based in South Korea, United States and Australia. Raekil Park's co-authors include Hong‐Seob So, Channy Park, Seong‐Kyu Choe, Joon No Lee, Se-Jin Kim, Hyung‐Jin Kim, Hun‐Taeg Chung, Hyun‐Ock Pae, David Lim and Young‐Myeong Kim and has published in prestigious journals such as Blood, PLoS ONE and Circulation Research.

In The Last Decade

Raekil Park

82 papers receiving 2.3k citations

Hit Papers

Mitochondria-associated programmed cell death as a therap... 2023 2026 2024 2025 2023 50 100 150

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Raekil Park South Korea 30 1.2k 383 313 271 229 84 2.3k
Raekil Park South Korea 31 1.4k 1.2× 334 0.9× 343 1.1× 177 0.7× 274 1.2× 74 3.0k
Channy Park South Korea 26 918 0.8× 606 1.6× 200 0.6× 135 0.5× 123 0.5× 57 2.1k
Rong Ma United States 38 1.8k 1.5× 812 2.1× 186 0.6× 161 0.6× 368 1.6× 148 3.9k
Michael P. Neeper United States 20 922 0.8× 285 0.7× 518 1.7× 293 1.1× 426 1.9× 26 3.3k
Hong‐Seob So South Korea 39 2.1k 1.8× 687 1.8× 477 1.5× 343 1.3× 337 1.5× 119 4.4k
Maria Beatrice Morelli Italy 34 1.0k 0.9× 826 2.2× 295 0.9× 196 0.7× 247 1.1× 91 2.8k
Consuelo Amantini Italy 35 1.4k 1.2× 1.1k 2.8× 436 1.4× 321 1.2× 352 1.5× 118 3.7k
Xiaoning Zeng China 30 1.1k 1.0× 98 0.3× 541 1.7× 125 0.5× 392 1.7× 68 2.6k
Xinghua Gao China 25 1.2k 1.0× 107 0.3× 274 0.9× 146 0.5× 288 1.3× 57 2.7k
Kyung‐Jong Won South Korea 27 1.2k 1.1× 258 0.7× 256 0.8× 77 0.3× 580 2.5× 93 2.7k

Countries citing papers authored by Raekil Park

Since Specialization
Citations

This map shows the geographic impact of Raekil Park's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Raekil Park with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Raekil Park more than expected).

Fields of papers citing papers by Raekil Park

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Raekil Park. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Raekil Park. The network helps show where Raekil Park may publish in the future.

Co-authorship network of co-authors of Raekil Park

This figure shows the co-authorship network connecting the top 25 collaborators of Raekil Park. A scholar is included among the top collaborators of Raekil Park based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Raekil Park. Raekil Park is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wei, Xiaofan, et al.. (2025). Pexophagy and Oxidative Stress: Focus on Peroxisomal Proteins and Reactive Oxygen Species (ROS) Signaling Pathways. Antioxidants. 14(2). 126–126. 5 indexed citations
3.
Kwon, Hyug Moo, et al.. (2024). TonEBP degradation is essential for microtubule nucleation and regrowth. Biochemical and Biophysical Research Communications. 734. 150791–150791. 1 indexed citations
4.
Kim, Joon, Seung Hyun Yong, Raekil Park, et al.. (2024). Spatial profiling of non-small cell lung cancer provides insights into tumorigenesis and immunotherapy response. Communications Biology. 7(1). 930–930. 2 indexed citations
5.
Park, Channy, et al.. (2024). TMEM135 deficiency improves hepatic steatosis by suppressing CD36 in a SIRT1-dependent manner. Molecular Metabolism. 92. 102080–102080.
6.
Bhandari, Sushil, Yong‐Il Kim, Yunju Jo, et al.. (2023). Loss of pex5 sensitizes zebrafish to fasting due to deregulated mitochondria, mTOR, and autophagy. Cellular and Molecular Life Sciences. 80(3). 69–69. 5 indexed citations
7.
Nguyễn, Thanh Tùng, Shibo Wei, Ha Nguyen Thi Thu, et al.. (2023). Mitochondria-associated programmed cell death as a therapeutic target for age-related disease. Experimental & Molecular Medicine. 55(8). 1595–1619. 159 indexed citations breakdown →
8.
Cho, Sung Woo, et al.. (2023). Pharmacologic Activation of Angiotensin-Converting Enzyme II Alleviates Diabetic Cardiomyopathy in db/db Mice by Reducing Reactive Oxidative Stress. Diabetes & Metabolism Journal. 47(4). 487–499. 7 indexed citations
11.
Lee, Joon No, et al.. (2021). Catalase deficiency induces reactive oxygen species mediated pexophagy and cell death in the liver during prolonged fasting. BioFactors. 47(1). 112–125. 22 indexed citations
12.
Lee, Juyeon, et al.. (2021). The Peroxisomal Localization of Hsd17b4 Is Regulated by Its Interaction with Phosphatidylserine. Molecules and Cells. 44(4). 214–222. 5 indexed citations
13.
14.
Wei, Xiaofan, et al.. (2021). Knockdown of PEX16 Induces Autophagic Degradation of Peroxisomes. International Journal of Molecular Sciences. 22(15). 7989–7989. 7 indexed citations
15.
Wei, Xiaofan, et al.. (2020). Dimethyloxaloylglycine induces pexophagy in a HIF-2α dependent manner involving autophagy receptor p62. Biochemical and Biophysical Research Communications. 525(1). 46–52. 5 indexed citations
16.
Kim, Yong‐Il, Dong‐Kyu Lee, Sushil Bhandari, et al.. (2019). Slc25a17 acts as a peroxisomal coenzyme A transporter and regulates multiorgan development in zebrafish. Journal of Cellular Physiology. 235(1). 151–165. 22 indexed citations
17.
Park, Channy, Hye-Won Lim, Sung K. Moon, & Raekil Park. (2019). Pyridoxine Preferentially Induces Auditory Neuropathy Through Mitochondrial Dysfunction and Endoplasmic Reticulum Stress-Mediated Apoptosis. Annals of Otology Rhinology & Laryngology. 128(6_suppl). 117S–124S. 7 indexed citations
18.
So, Hong‐Seob, Hyung‐Jin Kim, Jeong-Han Lee, et al.. (2007). Cisplatin Cytotoxicity of Auditory Cells Requires Secretions of Proinflammatory Cytokines via Activation of ERK and NF-κB. Journal of the Association for Research in Otolaryngology. 8(3). 338–355. 199 indexed citations
19.
Kim, Hyungjin, Hong‐Seob So, Jeong‐Han Lee, et al.. (2006). Heme oxygenase-1 attenuates the cisplatin-induced apoptosis of auditory cells via down-regulation of reactive oxygen species generation. Free Radical Biology and Medicine. 40(10). 1810–1819. 67 indexed citations
20.
Yu, Hyeon‐Hee, et al.. (2006). Protective Effect of Ursolic Acid from Corni fructus on the Hydrogen Peroxide-induced Damage of HEI-OC1 Auditory Cells. Journal of Physiology & Pathology in Korean Medicine. 20(6). 1524–1529. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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