Rachel Byron

7.5k total citations
8 papers, 505 citations indexed

About

Rachel Byron is a scholar working on Molecular Biology, Genetics and Pediatrics, Perinatology and Child Health. According to data from OpenAlex, Rachel Byron has authored 8 papers receiving a total of 505 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 2 papers in Genetics and 1 paper in Pediatrics, Perinatology and Child Health. Recurrent topics in Rachel Byron's work include Genomics and Chromatin Dynamics (5 papers), RNA modifications and cancer (4 papers) and Hemoglobinopathies and Related Disorders (2 papers). Rachel Byron is often cited by papers focused on Genomics and Chromatin Dynamics (5 papers), RNA modifications and cancer (4 papers) and Hemoglobinopathies and Related Disorders (2 papers). Rachel Byron collaborates with scholars based in United States, Sri Lanka and United Kingdom. Rachel Byron's co-authors include Mark Groudine, M. A. Bender, Tobias Ragoczy, Agnes Telling, Daniel E. Sabath, Harvey A. Greisman, D. J. Weatherall, Anuja Premawardhena, Jian Xu and Stuart H. Orkin and has published in prestigious journals such as New England Journal of Medicine, Nature Communications and Genes & Development.

In The Last Decade

Rachel Byron

7 papers receiving 499 citations

Peers

Rachel Byron

MB

GENET

HEMAT

GENET

PPCH

AeRi Kim South Korea

Yea Woon Kim South Korea

Duangporn Jamsai Australia

Nicole Hamagami United States

Saurabh K. Bhardwaj United States

S. Pruzina United States

Jon Kerry United Kingdom

AeRi Kim South Korea

Asuka Kamio Japan

Ninad M. Walavalkar United States

AeRi Kim South Korea

Rachel Byron

407 ×1.0

MB

83 ×0.6

GENET

32 ×0.4

HEMAT

38 ×0.6

GENET

31 ×0.6

PPCH

Citations per year, relative to Rachel Byron Rachel Byron (= 1×) peers AeRi Kim

Countries citing papers authored by Rachel Byron

Since Specialization

Citations

This map shows the geographic impact of Rachel Byron's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Rachel Byron with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Rachel Byron more than expected).

Fields of papers citing papers by Rachel Byron

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Rachel Byron. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Rachel Byron. The network helps show where Rachel Byron may publish in the future.

Co-authorship network of co-authors of Rachel Byron

This figure shows the co-authorship network connecting the top 25 collaborators of Rachel Byron. A scholar is included among the top collaborators of Rachel Byron based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Rachel Byron. Rachel Byron is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Halow, Jessica, Rachel Byron, Megan S. Hogan, et al.. (2021). Tissue context determines the penetrance of regulatory DNA variation. Nature Communications. 12(1). 2850–2850. 9 indexed citations

2.

Bender, M. A., Tobias Ragoczy, Jongjoo Lee, et al.. (2012). The hypersensitive sites of the murine β-globin locus control region act independently to affect nuclear localization and transcriptional elongation. Blood. 119(16). 3820–3827. 35 indexed citations

3.

Sankaran, Vijay G., Jian Xu, Rachel Byron, et al.. (2011). A Functional Element Necessary for Fetal Hemoglobin Silencing. New England Journal of Medicine. 365(9). 807–814. 133 indexed citations

4.

Bender, M. A., Davide Baù, Tobias Ragoczy, et al.. (2011). Integrating Structural and Functional Studies Leads to a New Model of β-Globin Activation That Suggests Distinct Initiation and Maintenance States. Blood. 118(21). 349–349. 1 indexed citations

5.

Fromm, George, Rachel Byron, Jennifer Fields, et al.. (2009). Histone hyperacetylation within the β-globin locus is context-dependent and precedes high-level gene expression. Blood. 114(16). 3479–3488. 14 indexed citations

6.

Ragoczy, Tobias, M. A. Bender, Agnes Telling, Rachel Byron, & Mark Groudine. (2006). The locus control region is required for association of the murine β-globin locus with engaged transcription factories during erythroid maturation. Genes & Development. 20(11). 1447–1457. 263 indexed citations

7.

Bender, M. A., Rachel Byron, Tobias Ragoczy, et al.. (2006). Flanking HS-62.5 and 3′ HS1, and regions upstream of the LCR, are not required for β-globin transcription. Blood. 108(4). 1395–1401. 50 indexed citations

8.

Bender, M. A., Tobias Ragoczy, Rachel Byron, Agnes Telling, & Mark Groudine. (2005). Nuclear Dynamics and Gene Activation during Erythroid Maturation.. Blood. 106(11). 819–819.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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