Qinlong Gu

1.6k total citations
40 papers, 1.3k citations indexed

About

Qinlong Gu is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Qinlong Gu has authored 40 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 31 papers in Molecular Biology, 13 papers in Oncology and 13 papers in Cancer Research. Recurrent topics in Qinlong Gu's work include MicroRNA in disease regulation (9 papers), Cancer-related molecular mechanisms research (9 papers) and Circular RNAs in diseases (7 papers). Qinlong Gu is often cited by papers focused on MicroRNA in disease regulation (9 papers), Cancer-related molecular mechanisms research (9 papers) and Circular RNAs in diseases (7 papers). Qinlong Gu collaborates with scholars based in China, United States and Israel. Qinlong Gu's co-authors include Bingya Liu, Jianfang Li, Zhenggang Zhu, Liping Su, Yingyan Yu, Min Yan, Beiqin Yu, Xuehua Chen, Zhenggang Zhu and Ying Qu and has published in prestigious journals such as SHILAP Revista de lepidopterología, PLoS ONE and Scientific Reports.

In The Last Decade

Qinlong Gu

40 papers receiving 1.3k citations

Peers

Qinlong Gu
Chad A. Glazer United States
Karen D. Cowden Dahl United States
Chun-Ju Chang United States
Mohit Sachdeva United States
Alfiya Safina United States
Chad A. Glazer United States
Qinlong Gu
Citations per year, relative to Qinlong Gu Qinlong Gu (= 1×) peers Chad A. Glazer

Countries citing papers authored by Qinlong Gu

Since Specialization
Citations

This map shows the geographic impact of Qinlong Gu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Qinlong Gu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Qinlong Gu more than expected).

Fields of papers citing papers by Qinlong Gu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Qinlong Gu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Qinlong Gu. The network helps show where Qinlong Gu may publish in the future.

Co-authorship network of co-authors of Qinlong Gu

This figure shows the co-authorship network connecting the top 25 collaborators of Qinlong Gu. A scholar is included among the top collaborators of Qinlong Gu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Qinlong Gu. Qinlong Gu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Yang, Zhongyin, Chen Li, Chao Yan, et al.. (2018). KIF14 promotes tumor progression and metastasis and is an independent predictor of poor prognosis in human gastric cancer. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1865(1). 181–192. 64 indexed citations
2.
Zang, Mingde, Lei Hu, Shu Cao, et al.. (2017). Dual role of carcinoembryonic antigen-related cell adhesion molecule 6 expression in predicting the overall survival of gastric cancer patients. Scientific Reports. 7(1). 10773–10773. 8 indexed citations
3.
Zang, Mingde, Yunqiang Zhang, Baogui Zhang, et al.. (2015). CEACAM6 promotes tumor angiogenesis and vasculogenic mimicry in gastric cancer via FAK signaling. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1852(5). 1020–1028. 59 indexed citations
4.
Zang, Mingde, Baogui Zhang, Yunqiang Zhang, et al.. (2014). CEACAM6 Promotes Gastric Cancer Invasion and Metastasis by Inducing Epithelial-Mesenchymal Transition via PI3K/AKT Signaling Pathway. PLoS ONE. 9(11). e112908–e112908. 53 indexed citations
5.
Yang, Zhongyin, He Jiang, Ying Qu, et al.. (2013). Metallopanstimulin-1 regulates invasion and migration of gastric cancer cells partially through integrin β4. Carcinogenesis. 34(12). 2851–2860. 35 indexed citations
6.
Yu, Beiqin, Xuehua Chen, Jianfang Li, et al.. (2013). Stromal fibroblasts in the microenvironment of gastric carcinomas promote tumor metastasis via upregulating TAGLN expression. BMC Cell Biology. 14(1). 17–17. 53 indexed citations
7.
Wang, Ming, Chenglong Li, Hui Nie, et al.. (2012). Down‐regulated miR‐625 suppresses invasion and metastasis of gastric cancer by targeting ILK. FEBS Letters. 586(16). 2382–2388. 60 indexed citations
8.
Zheng, Zhong, Xiangyi He, Jianfang Li, et al.. (2012). RhoGDI2 confers resistance to 5-fluorouracil in human gastric cancer cells. Oncology Letters. 5(1). 255–260. 11 indexed citations
9.
Zhang, Qing, Zhongyin Yang, Min Wei, et al.. (2012). Upregulated expression of LOX is a novel independent prognostic marker of worse outcome in gastric cancer patients after curative surgery. Oncology Letters. 5(3). 896–902. 21 indexed citations
10.
Wei, Min, Bingya Liu, Qinlong Gu, et al.. (2012). Stat6 cooperates with Sp1 in controlling breast cancer cell proliferation by modulating the expression of p21Cip1/WAF1 and p27Kip1. Cellular Oncology. 36(1). 79–93. 34 indexed citations
11.
Yu, Beiqin, Liping Su, Jianfang Li, et al.. (2012). microRNA expression signature of gastric cancer cells relative to normal gastric mucosa. Molecular Medicine Reports. 6(4). 821–826. 23 indexed citations
12.
Wei, Min, Li Zhu, Yafen Li, et al.. (2011). Knocking down cyclin D1b inhibits breast cancer cell growth and suppresses tumor development in a breast cancer model. Cancer Science. 102(8). 1537–1544. 18 indexed citations
13.
Wei, Min, Zhiwei Wang, Hongliang Yao, et al.. (2011). P27Kip1, regulated by glycogen synthase kinase-3β, results in HMBA-induced differentiation of human gastric cancer cells. BMC Cancer. 11(1). 109–109. 14 indexed citations
14.
Yang, Zhongyin, Ying Qu, Qing Zhang, et al.. (2011). Knockdown of metallopanstimulin‐1 inhibits NF‐κB signaling at different levels: The role of apoptosis induction of gastric cancer cells. International Journal of Cancer. 130(12). 2761–2770. 30 indexed citations
15.
Wei, Min, Bingya Liu, Liping Su, et al.. (2010). A Novel Plant Homeodomain Finger 10–Mediated Antiapoptotic Mechanism Involving Repression of Caspase-3 in Gastric Cancer Cells. Molecular Cancer Therapeutics. 9(6). 1764–1774. 21 indexed citations
16.
Wang, Lifeng, Lisa Rollins, Qinlong Gu, Si–Yi Chen, & Xue F. Huang. (2009). A Mage3/Heat Shock Protein70 DNA vaccine induces both innate and adaptive immune responses for the antitumor activity. Vaccine. 28(2). 561–570. 27 indexed citations
17.
Qu, Ying, Jianfang Li, Qu Cai, et al.. (2007). Over-expression of FRZB in gastric cancer cell suppresses proliferation and induces differentiation. Journal of Cancer Research and Clinical Oncology. 134(3). 353–364. 23 indexed citations
18.
Gu, Qinlong. (2007). Targeting hepatitis B virus antigens to dendritic cells by heat shock protein to improve DNA vaccine potency. World Journal of Gastroenterology. 13(44). 5911–5911. 7 indexed citations
19.
Li, Jianfang, Ying Qu, Xuehua Chen, et al.. (2006). [Expression of Ezrin in gastric carcinoma and its significance].. PubMed. 9(5). 433–5. 3 indexed citations
20.
Wang, Yunwei, Qinlong Gu, Bingya Liu, & Zhenggang Zhu. (2004). Perspectives of SEREX-defined antigens in diagnosis and immunotherapy for gastric cancer. Cancer Biology & Therapy. 3(9). 806–811. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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