Qingwei Ji

2.3k total citations
60 papers, 1.8k citations indexed

About

Qingwei Ji is a scholar working on Immunology, Cardiology and Cardiovascular Medicine and Molecular Biology. According to data from OpenAlex, Qingwei Ji has authored 60 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 32 papers in Immunology, 18 papers in Cardiology and Cardiovascular Medicine and 14 papers in Molecular Biology. Recurrent topics in Qingwei Ji's work include Atherosclerosis and Cardiovascular Diseases (25 papers), T-cell and B-cell Immunology (9 papers) and Cytokine Signaling Pathways and Interactions (7 papers). Qingwei Ji is often cited by papers focused on Atherosclerosis and Cardiovascular Diseases (25 papers), T-cell and B-cell Immunology (9 papers) and Cytokine Signaling Pathways and Interactions (7 papers). Qingwei Ji collaborates with scholars based in China, Japan and United States. Qingwei Ji's co-authors include Yingzhong Lin, Ying Shi, Qiutang Zeng, Ying Huang, Bangwei Wu, Zhen Wang, Qiutang Zeng, Kunwu Yu, Jun Wan and Min Guo and has published in prestigious journals such as Journal of the American College of Cardiology, PLoS ONE and Frontiers in Immunology.

In The Last Decade

Qingwei Ji

58 papers receiving 1.8k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Qingwei Ji China 28 827 550 433 361 237 60 1.8k
Mengyang Liao China 22 749 0.9× 613 1.1× 457 1.1× 177 0.5× 266 1.1× 37 1.6k
Anton Gisterå Sweden 20 1.3k 1.5× 611 1.1× 226 0.5× 408 1.1× 294 1.2× 39 2.1k
Tom Seijkens Netherlands 24 1.1k 1.3× 444 0.8× 318 0.7× 342 0.9× 152 0.6× 52 1.9k
De-xiu Bu United States 14 674 0.8× 454 0.8× 265 0.6× 265 0.7× 211 0.9× 17 1.8k
Roslynn A. Stirzaker United States 8 848 1.0× 555 1.0× 263 0.6× 475 1.3× 325 1.4× 8 1.8k
Yingzhong Lin China 24 603 0.7× 356 0.6× 308 0.7× 251 0.7× 134 0.6× 56 1.3k
Matthias Canault France 21 445 0.5× 515 0.9× 232 0.5× 199 0.6× 176 0.7× 42 1.6k
Esther Peña Spain 26 388 0.5× 831 1.5× 445 1.0× 339 0.9× 397 1.7× 60 1.9k
Giovanni Cimmino Italy 26 415 0.5× 523 1.0× 725 1.7× 307 0.9× 524 2.2× 110 2.1k
Lindsey A. MacFarlane United States 21 756 0.9× 526 1.0× 156 0.4× 334 0.9× 467 2.0× 46 2.1k

Countries citing papers authored by Qingwei Ji

Since Specialization
Citations

This map shows the geographic impact of Qingwei Ji's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Qingwei Ji with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Qingwei Ji more than expected).

Fields of papers citing papers by Qingwei Ji

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Qingwei Ji. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Qingwei Ji. The network helps show where Qingwei Ji may publish in the future.

Co-authorship network of co-authors of Qingwei Ji

This figure shows the co-authorship network connecting the top 25 collaborators of Qingwei Ji. A scholar is included among the top collaborators of Qingwei Ji based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Qingwei Ji. Qingwei Ji is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ding, Y., et al.. (2025). NLRP3 inflammasome in cardiovascular diseases: an update. Frontiers in Immunology. 16. 1550226–1550226. 8 indexed citations
3.
Ji, Qingwei, et al.. (2025). The link between the atherogenic index of plasma and the risk of hypertension: Analysis from NHANES 2017–2020. PLoS ONE. 20(1). e0317116–e0317116. 1 indexed citations
4.
Lu, Xiyi, Qingwei Ji, Heng Pan, et al.. (2024). IL-23p19 deficiency reduces M1 macrophage polarization and improves stress-induced cardiac remodeling by alleviating macrophage ferroptosis in mice. Biochemical Pharmacology. 222. 116072–116072. 9 indexed citations
5.
Ji, Qingwei, Mengmeng Zhao, Zihui Zheng, et al.. (2024). IL-12p40 deletion reduces M1 macrophage polarization and alleviates cardiac remodeling via regulating Th17 cells differentiation, but not γδT 17 cells, in TAC mice. European Journal of Pharmacology. 974. 176602–176602.
6.
Li, Shicheng, et al.. (2023). Inhibiting the MAPK pathway improves heart failure with preserved ejection fraction induced by salt-sensitive hypertension. Biomedicine & Pharmacotherapy. 170. 115987–115987. 9 indexed citations
7.
Peng, Yudong, Kai Meng, Meian He, et al.. (2020). Clinical Characteristics and Prognosis of 244 Cardiovascular Patients Suffering From Coronavirus Disease in Wuhan, China. Journal of the American Heart Association. 9(19). e016796–e016796. 27 indexed citations
8.
Ye, Jing, Yuan Wang, Zhen Wang, et al.. (2020). The Expression of IL-12 Family Members in Patients with Hypertension and Its Association with the Occurrence of Carotid Atherosclerosis. Mediators of Inflammation. 2020. 1–10. 23 indexed citations
9.
Shi, Lei, Qingwei Ji, Ling Liu, et al.. (2020). IL‐22 produced by Th22 cells aggravates atherosclerosis development in ApoE −/− mice by enhancing DC‐induced Th17 cell proliferation. Journal of Cellular and Molecular Medicine. 24(5). 3064–3078. 22 indexed citations
10.
Ye, Jing, Yuan Wang, Zhen Wang, et al.. (2020). Roles and Mechanisms of Interleukin-12 Family Members in Cardiovascular Diseases: Opportunities and Challenges. Frontiers in Pharmacology. 11. 129–129. 46 indexed citations
11.
Ye, Jing, Zhen Wang, Di Ye, et al.. (2019). Increased Interleukin-11 Levels Are Correlated with Cardiac Events in Patients with Chronic Heart Failure. Mediators of Inflammation. 2019. 1–8. 39 indexed citations
12.
Tong, Shan, Qingwei Ji, Yu Du, et al.. (2019). Sfrp5/Wnt Pathway: A Protective Regulatory System in Atherosclerotic Cardiovascular Disease. Journal of Interferon & Cytokine Research. 39(8). 472–482. 43 indexed citations
13.
Ye, Jing, Yuan Wang, Zhen Wang, et al.. (2018). Circulating Th1, Th2, Th9, Th17, Th22, and Treg Levels in Aortic Dissection Patients. Mediators of Inflammation. 2018. 1–10. 59 indexed citations
14.
Ye, Jing, Zhen Wang, Ying Huang, et al.. (2018). The clinical potential of IL-12/IL-35 in treating chemotherapy drug-induced cardiac injury – Authors' reply. EBioMedicine. 35. 4–5. 1 indexed citations
15.
Ye, Jing, Menglong Wang, Huimin Jiang, et al.. (2017). Increased levels of interleukin-22 in thoracic aorta and plasma from patients with acute thoracic aortic dissection. Clinica Chimica Acta. 486. 395–401. 28 indexed citations
16.
Ji, Qingwei, Yu Du, Zhijian Wang, et al.. (2017). Human epicardial adipose tissue-derived and circulating secreted frizzled-related protein 4 (SFRP4) levels are increased in patients with coronary artery disease. Cardiovascular Diabetology. 16(1). 133–133. 29 indexed citations
17.
Chai, Meng, Qingwei Ji, Haitao Zhang, et al.. (2015). The Protective Effect of Interleukin-37 on Vascular Calcification and Atherosclerosis in Apolipoprotein E-Deficient Mice with Diabetes. Journal of Interferon & Cytokine Research. 35(7). 530–539. 50 indexed citations
18.
Meng, Kai, Qiutang Zeng, Qinghua Lu, et al.. (2015). Valsartan Attenuates Atherosclerosis via Upregulating the Th2 Immune Response in Prolonged Angiotensin II-Treated ApoE−/− Mice. Molecular Medicine. 21(1). 143–153. 27 indexed citations
19.
Meng, Kai, Yucheng Zhong, Xiaobo Mao, et al.. (2014). Impairment of Circulating CD4+CD25+GARP+Regulatory T Cells in Patients with Acute Coronary Syndrome. Cellular Physiology and Biochemistry. 33(3). 621–632. 18 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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