Priska Kaufmann

993 total citations
36 papers, 777 citations indexed

About

Priska Kaufmann is a scholar working on Molecular Biology, Cognitive Neuroscience and Pharmacology. According to data from OpenAlex, Priska Kaufmann has authored 36 papers receiving a total of 777 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 6 papers in Cognitive Neuroscience and 6 papers in Pharmacology. Recurrent topics in Priska Kaufmann's work include Complement system in diseases (6 papers), Sleep and Wakefulness Research (6 papers) and Sleep and related disorders (5 papers). Priska Kaufmann is often cited by papers focused on Complement system in diseases (6 papers), Sleep and Wakefulness Research (6 papers) and Sleep and related disorders (5 papers). Priska Kaufmann collaborates with scholars based in Switzerland, Germany and United States. Priska Kaufmann's co-authors include Michael Török, Jasper Dingemanse, Stephan Krähenbühl, Karin Brecht, Anja Zahno, Stephan Krähenbühl, K. Waldhauser, Paul Roberts, Rodolfo Gasser and Shirin Bruderer and has published in prestigious journals such as Hepatology, Journal of Hepatology and Journal of Lipid Research.

In The Last Decade

Priska Kaufmann

35 papers receiving 760 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Priska Kaufmann Switzerland 13 307 151 151 127 114 36 777
Xuemei Wang China 18 237 0.8× 65 0.4× 46 0.3× 41 0.3× 105 0.9× 37 731
Eileen Holovac United States 6 268 0.9× 122 0.8× 26 0.2× 128 1.0× 30 0.3× 10 872
Malek Kechrid United States 5 232 0.8× 76 0.5× 28 0.2× 97 0.8× 30 0.3× 9 712
Chiara Bencini Italy 13 157 0.5× 94 0.6× 109 0.7× 88 0.7× 93 0.8× 23 581
B. Scherer Germany 18 390 1.3× 232 1.5× 112 0.7× 161 1.3× 100 0.9× 40 1.4k
Venkatesh Pilla Reddy United States 19 325 1.1× 28 0.2× 66 0.4× 37 0.3× 207 1.8× 56 1.1k
Csaba Mátyás Hungary 22 348 1.1× 476 3.2× 50 0.3× 221 1.7× 68 0.6× 37 1.4k
Youguang Gao China 13 306 1.0× 45 0.3× 78 0.5× 55 0.4× 45 0.4× 19 736
Robert Fischer Germany 16 385 1.3× 441 2.9× 48 0.3× 124 1.0× 124 1.1× 36 1.6k
C D Malis United States 11 353 1.1× 113 0.7× 74 0.5× 138 1.1× 46 0.4× 14 949

Countries citing papers authored by Priska Kaufmann

Since Specialization
Citations

This map shows the geographic impact of Priska Kaufmann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Priska Kaufmann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Priska Kaufmann more than expected).

Fields of papers citing papers by Priska Kaufmann

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Priska Kaufmann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Priska Kaufmann. The network helps show where Priska Kaufmann may publish in the future.

Co-authorship network of co-authors of Priska Kaufmann

This figure shows the co-authorship network connecting the top 25 collaborators of Priska Kaufmann. A scholar is included among the top collaborators of Priska Kaufmann based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Priska Kaufmann. Priska Kaufmann is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Berger, Benjamin, et al.. (2024). Value of Assessing 1‐Hydroxymidazolam in Drug‐Drug Interaction Studies with Midazolam as a Substrate of Cytochrome P450 3A. The Journal of Clinical Pharmacology. 64(9). 1123–1129. 2 indexed citations
3.
Dingemanse, Jasper, Stéphane Delahaye, Luboš Janů, et al.. (2023). Evaluation of the cytochrome P450 2C19 and 3A4 inhibition potential of the complement factor 5a receptor 1 antagonist ACT‐1014‐6470 in vitro and in vivo. Clinical and Translational Science. 16(7). 1220–1231. 2 indexed citations
4.
Krause, Andreas, et al.. (2023). Modeling time‐delayed concentration‐QT effects with ACT‐1014‐6470, a novel oral complement factor 5a receptor 1 (C5a1 receptor) antagonist. Pharmacology Research & Perspectives. 11(4). e01112–e01112. 2 indexed citations
5.
6.
Kaufmann, Priska, et al.. (2020). Multiple-dose clinical pharmacology of the selective orexin-1 receptor antagonist ACT-539313. Progress in Neuro-Psychopharmacology and Biological Psychiatry. 108. 110166–110166. 24 indexed citations
7.
Kaufmann, Priska, et al.. (2016). Absolute oral bioavailability of selexipag, a novel oral prostacyclin IP receptor agonist. European Journal of Clinical Pharmacology. 73(2). 151–156. 19 indexed citations
8.
Kaufmann, Priska, et al.. (2015). Pharmacokinetics and Tolerability of the Novel Oral Prostacyclin IP Receptor Agonist Selexipag. American Journal of Cardiovascular Drugs. 15(3). 195–203. 48 indexed citations
9.
Kaufmann, Priska, et al.. (2015). Effect of lopinavir/ritonavir on the pharmacokinetics of selexipag an oral prostacyclin receptor agonist and its active metabolite in healthy subjects. British Journal of Clinical Pharmacology. 80(4). 670–677. 29 indexed citations
10.
11.
Dingemanse, Jasper, et al.. (2011). PHARMACOKINETICS AND TOLERABILITY OF THE ORAL SELECTIVE PROSTACYCLIN RECEPTOR AGONIST SELEXIPAG AFTER MULTIPLE-DOSE ADMINISTRATION IN HEALTHY MALE SUBJECTS. Clinical Pharmacology & Therapeutics. 89. 88–89. 1 indexed citations
12.
Kaufmann, Priska, Michael Török, Anja Zahno, et al.. (2006). Toxicity of statins on rat skeletal muscle mitochondria. Cellular and Molecular Life Sciences. 63(19-20). 2415–2425. 201 indexed citations
13.
Kaufmann, Priska, Manuel Haschke, Michael Török, et al.. (2006). Mechanisms of Venoocclusive Disease Resulting From the Combination of Cyclophosphamide and Roxithromycin. Therapeutic Drug Monitoring. 28(6). 766–774. 5 indexed citations
14.
Porta, Francesca, Jukka Takala, Christian Weikert, et al.. (2006). Effect of endotoxin, dobutamine and dopamine on muscle mitochondrial respiration <I>in vitro</I>. Journal of Endotoxin Research. 12(6). 358–366. 9 indexed citations
15.
Haschke, Manuel, et al.. (2006). Hepatic Veno-Occlusive Disease Associated with Immunosuppressive Cyclophosphamide Dosing and Roxithromycin. Annals of Pharmacotherapy. 40(4). 767–770. 5 indexed citations
16.
Simmen, Urs, et al.. (2005). Preserved Pharmacological Activity of Hepatocytes-Treated Extracts of Valerian and St. John's Wort. Planta Medica. 71(7). 592–598. 8 indexed citations
17.
Kaufmann, Priska, et al.. (2005). Mechanisms of benzarone and benzbromarone‐induced hepatic toxicity†. Hepatology. 41(4). 925–935. 150 indexed citations
18.
Kaufmann, Priska, et al.. (2004). 78 Mechanisms of benzarone and benzbromarone induced hepatic toxicity. Journal of Hepatology. 40. 27–28. 6 indexed citations
19.
Kaufmann, Priska, Konstantin Beier, Michael Török, et al.. (2003). Mechanisms of liver steatosis in rats with systemic carnitine deficiency due to treatment with trimethylhydraziniumpropionate. Journal of Lipid Research. 44(1). 144–153. 41 indexed citations
20.
Kaufmann, Priska, et al.. (2003). Myocardial function and energy metabolism in carnitine-deficient rats. Cellular and Molecular Life Sciences. 60(4). 767–775. 22 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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