Pin Ouyang

909 total citations
30 papers, 774 citations indexed

About

Pin Ouyang is a scholar working on Molecular Biology, Cell Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Pin Ouyang has authored 30 papers receiving a total of 774 indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Molecular Biology, 9 papers in Cell Biology and 2 papers in Cellular and Molecular Neuroscience. Recurrent topics in Pin Ouyang's work include RNA Research and Splicing (15 papers), RNA modifications and cancer (7 papers) and RNA and protein synthesis mechanisms (7 papers). Pin Ouyang is often cited by papers focused on RNA Research and Splicing (15 papers), RNA modifications and cancer (7 papers) and RNA and protein synthesis mechanisms (7 papers). Pin Ouyang collaborates with scholars based in Taiwan, United States and China. Pin Ouyang's co-authors include S P Sugrue, Steve Leu, An-Hsun Chou, Hung‐Li Wang, Ying‐Ling Chen, Tu‐Hsueh Yeh, Stephen P. Sugrue, Pei‐Jen Lou, Ping Wang and Yujiang Geno Shi and has published in prestigious journals such as Journal of Biological Chemistry, The Journal of Cell Biology and Molecular and Cellular Biology.

In The Last Decade

Pin Ouyang

30 papers receiving 765 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Pin Ouyang Taiwan 15 630 148 122 85 58 30 774
Yuji Funakoshi Japan 18 786 1.2× 96 0.6× 211 1.7× 81 1.0× 92 1.6× 32 1.0k
Sang Mi Shim South Korea 13 404 0.6× 59 0.4× 172 1.4× 50 0.6× 54 0.9× 13 661
Astrid Rohrbeck Germany 15 292 0.5× 69 0.5× 84 0.7× 76 0.9× 86 1.5× 30 482
Isabelle Cornez Germany 10 501 0.8× 183 1.2× 156 1.3× 35 0.4× 126 2.2× 12 904
Ken Matsumoto Japan 16 484 0.8× 64 0.4× 135 1.1× 82 1.0× 61 1.1× 29 685
Noga Guttmann‐Raviv Israel 14 729 1.2× 251 1.7× 133 1.1× 74 0.9× 41 0.7× 19 830
Adele G. Woolley New Zealand 13 436 0.7× 73 0.5× 53 0.4× 106 1.2× 78 1.3× 17 613
Marilyn Parra United States 19 1.0k 1.6× 134 0.9× 273 2.2× 111 1.3× 79 1.4× 24 1.4k
Norio Takizawa United States 13 599 1.0× 48 0.3× 297 2.4× 60 0.7× 56 1.0× 20 813
José Piedra Spain 7 867 1.4× 85 0.6× 318 2.6× 39 0.5× 88 1.5× 9 1.0k

Countries citing papers authored by Pin Ouyang

Since Specialization
Citations

This map shows the geographic impact of Pin Ouyang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Pin Ouyang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Pin Ouyang more than expected).

Fields of papers citing papers by Pin Ouyang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Pin Ouyang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Pin Ouyang. The network helps show where Pin Ouyang may publish in the future.

Co-authorship network of co-authors of Pin Ouyang

This figure shows the co-authorship network connecting the top 25 collaborators of Pin Ouyang. A scholar is included among the top collaborators of Pin Ouyang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Pin Ouyang. Pin Ouyang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Chu, Pao‐Hsien, et al.. (2022). Cell cycle expression of FLJ25439, a cytokinesis-associated protein, is mediated by D-box recognition and APC/C-Cdc20 regulated degradation. Biochemical and Biophysical Research Communications. 630. 151–157. 2 indexed citations
2.
Chu, Pao‐Hsien, et al.. (2019). Ectopically expressed pNO40 suppresses ribosomal RNA synthesis by inhibiting UBF-dependent transcription activation. Biochemical and Biophysical Research Communications. 516(2). 381–387. 5 indexed citations
3.
Tam, Bjorn T., Angus P. Yu, Eric W. Tam, et al.. (2018). Ablation of Bax and Bak protects skeletal muscle against pressure-induced injury. Scientific Reports. 8(1). 3689–3689. 12 indexed citations
4.
Chu, Pao‐Hsien, et al.. (2018). TSG101 interacts with the androgen receptor and attenuates its expression through the endosome/lysosome pathway. Biochemical and Biophysical Research Communications. 503(1). 157–164. 3 indexed citations
5.
Chu, Pao‐Hsien, et al.. (2017). Ribosomal protein pNO40 mediates nucleolar sequestration of SR family splicing factors and its overexpression impairs mRNA metabolism. Cellular Signalling. 32. 12–23. 8 indexed citations
6.
Wu, Chih‐Ching, et al.. (2015). Target disruption of ribosomal protein pNO40 accelerates aging and impairs osteogenic differentiation of mesenchymal stem cells. Biochemical and Biophysical Research Communications. 469(4). 903–910. 6 indexed citations
7.
Hsu, Shu-Yuan, et al.. (2013). Transgenic mice expressing mutant Pinin exhibit muscular dystrophy, nebulin deficiency and elevated expression of slow-type muscle fiber genes. Biochemical and Biophysical Research Communications. 443(1). 313–320. 4 indexed citations
8.
Hsu, Shu-Yuan, Yi‐Chuan Cheng, Hung‐Yu Shih, & Pin Ouyang. (2012). Dissection of the role of Pinin in the development of zebrafish posterior pharyngeal cartilages. Histochemistry and Cell Biology. 138(1). 127–140. 8 indexed citations
10.
Hsu, Shu-Yuan, Yen‐Jung Chen, & Pin Ouyang. (2011). Pnn and SR family proteins are differentially expressed in mouse central nervous system. Histochemistry and Cell Biology. 135(4). 361–373. 8 indexed citations
11.
Chang, Yu‐Chen, et al.. (2010). Characterization of centrosomal proteins Cep55 and pericentrin in intercellular bridges of mouse testes. Journal of Cellular Biochemistry. 109(6). 1274–1285. 16 indexed citations
12.
Chou, An-Hsun, et al.. (2008). Polyglutamine-expanded ataxin-3 causes cerebellar dysfunction of SCA3 transgenic mice by inducing transcriptional dysregulation. Neurobiology of Disease. 31(1). 89–101. 143 indexed citations
13.
Ouyang, Pin, et al.. (2006). Loss of Pnn expression attenuates expression levels of SR family splicing factors and modulates alternative pre-mRNA splicing in vivo. Biochemical and Biophysical Research Communications. 341(2). 663–671. 12 indexed citations
14.
Leu, Steve & Pin Ouyang. (2006). Spatial and temporal expression profile of pinin during mouse development. Gene Expression Patterns. 6(6). 620–631. 11 indexed citations
15.
Leu, Steve, et al.. (2004). Over-expression of SR-cyclophilin, an interaction partner of nuclear pinin, releases SR family splicing factors from nuclear speckles. Biochemical and Biophysical Research Communications. 321(3). 638–647. 24 indexed citations
16.
Wang, Ping, Pei‐Jen Lou, Steve Leu, & Pin Ouyang. (2002). Modulation of alternative pre-mRNA splicing in vivo by pinin. Biochemical and Biophysical Research Communications. 294(2). 448–455. 54 indexed citations
17.
Shi, Yujiang Geno, Pin Ouyang, & Stephen P. Sugrue. (2000). Characterization of the gene encoding pinin/DRS/memA and evidence for its potential tumor suppressor function. Oncogene. 19(2). 289–297. 43 indexed citations
18.
Ouyang, Pin. (1999). Antibodies Differentiate Desmosome-Form and Nucleus-Form Pinin: Evidence That Pinin Is a Moonlighting Protein with Dual Location at the Desmosome and within the Nucleus. Biochemical and Biophysical Research Communications. 263(1). 192–200. 32 indexed citations
19.
Ouyang, Pin. (1998). Anin VitroModel to Study Mesenchymal– Epithelial Transformation. Biochemical and Biophysical Research Communications. 246(3). 771–776. 20 indexed citations
20.
Ouyang, Pin, Yen‐Yi Zhen, & Stephen P. Sugrue. (1997). Cloning and analysis of cDNA encoding murine pinin. Gene. 197(1-2). 115–120. 11 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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