Philippe Saas

10.1k total citations · 2 hit papers
210 papers, 7.0k citations indexed

About

Philippe Saas is a scholar working on Immunology, Hematology and Molecular Biology. According to data from OpenAlex, Philippe Saas has authored 210 papers receiving a total of 7.0k indexed citations (citations by other indexed papers that have themselves been cited), including 146 papers in Immunology, 45 papers in Hematology and 41 papers in Molecular Biology. Recurrent topics in Philippe Saas's work include Immune Cell Function and Interaction (62 papers), T-cell and B-cell Immunology (42 papers) and Immunotherapy and Immune Responses (41 papers). Philippe Saas is often cited by papers focused on Immune Cell Function and Interaction (62 papers), T-cell and B-cell Immunology (42 papers) and Immunotherapy and Immune Responses (41 papers). Philippe Saas collaborates with scholars based in France, United States and Switzerland. Philippe Saas's co-authors include Pierre Tiberghien, Paul R. Walker, Pierre‐Yves Dietrich, Sylvain Perruche, Émilie Gaiffe, Jamal Bamoulid, Béatrice Gaugler, Christophe Ferrand, Christophe Borg and Éric Toussirot and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and Nature Medicine.

In The Last Decade

Philippe Saas

199 papers receiving 6.8k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Human Leukocyte Antigen-G5 Secretion by Human Mesenchymal Stem Cells Is Required to Suppress T Lymphocyte and Natural Killer Function and to Induce CD4+CD25highFOXP3+ Regulatory T Cells

2007 845 citations Christophe Borg, Philippe Saas et al. profile →
MCT1 lactate transporter blockade re-invigorates anti-tumor immunity through metabolic rewiring of dendritic cells in melanoma

2025 18 citations Stéphane Mouret, Mylène Pezet et al. Nature Communications profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Philippe Saas France	43	3.4k	1.8k	1.3k	1.0k	935	210	7.0k
Francesco Liotta Italy	49	5.7k 1.7×	2.0k 1.1×	1.4k 1.1×	1.7k 1.7×	644 0.7×	99	10.3k
José L. Cohen France	39	3.9k 1.1×	879 0.5×	918 0.7×	566 0.6×	1.3k 1.4×	124	6.2k
Benedetta Mazzinghi Italy	30	2.9k 0.8×	2.6k 1.4×	1.3k 1.1×	1.5k 1.5×	415 0.4×	55	7.4k
Shunichi Kato Japan	43	2.2k 0.6×	1.4k 0.8×	956 0.8×	1.1k 1.1×	3.0k 3.2×	171	6.2k
Elena Lazzeri Italy	46	3.2k 0.9×	2.7k 1.5×	1.8k 1.4×	428 0.4×	313 0.3×	99	8.2k
Gerd‐Rüdiger Burmester Germany	48	3.2k 0.9×	1.1k 0.6×	724 0.6×	1.1k 1.1×	1.4k 1.5×	154	8.7k
Lorenzo Cosmi Italy	55	7.5k 2.2×	2.0k 1.1×	2.2k 1.7×	1.6k 1.5×	747 0.8×	134	12.4k
Leendert A. Trouw Netherlands	55	4.7k 1.4×	1.5k 0.8×	511 0.4×	553 0.5×	1.2k 1.3×	218	9.8k
A. Richard Kitching Australia	56	4.3k 1.3×	2.1k 1.2×	647 0.5×	706 0.7×	467 0.5×	209	8.8k
H. Terence Cook United Kingdom	48	6.5k 1.9×	2.2k 1.2×	391 0.3×	802 0.8×	1.3k 1.4×	122	9.6k

Countries citing papers authored by Philippe Saas

Since Specialization

Citations

This map shows the geographic impact of Philippe Saas's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Philippe Saas with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Philippe Saas more than expected).

Fields of papers citing papers by Philippe Saas

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Philippe Saas. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Philippe Saas. The network helps show where Philippe Saas may publish in the future.

Co-authorship network of co-authors of Philippe Saas

This figure shows the co-authorship network connecting the top 25 collaborators of Philippe Saas. A scholar is included among the top collaborators of Philippe Saas based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Philippe Saas. Philippe Saas is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

a-Flin, Ekaterina Bourov, Thomas Decaens, Saadi Khochbin, et al.. (2025). Identification of immunogenic HLA-A*02:01 epitopes associated with HCC for immunotherapy development. Hepatology Communications. 9(3). 1 indexed citations

Chuffart, Florent, Thomas Decaens, Marie‐Noëlle Hilleret, et al.. (2025). HBV and HBsAg strongly reshape the phenotype, function, and metabolism of DCs according to patients’ clinical stage. Hepatology Communications. 9(2). 2 indexed citations

Bonnefoy, Francis, et al.. (2025). HUMANIN produced by human efferocytic macrophages promotes the resolution of inflammation. Cell Death and Disease. 16(1). 656–656. 1 indexed citations

Mouret, Stéphane, Mylène Pezet, Benoît Roubinet, et al.. (2025). MCT1 lactate transporter blockade re-invigorates anti-tumor immunity through metabolic rewiring of dendritic cells in melanoma. Nature Communications. 16(1). 1083–1083. 18 indexed citations breakdown →

Saas, Philippe, et al.. (2024). Glycans in melanoma: Drivers of tumour progression but sweet targets to exploit for immunotherapy. Immunology. 173(1). 33–52. 4 indexed citations

Saas, Philippe, et al.. (2024). « Fort comme la mort », où comment l’efferocytose contrôle la résolution de l’inflammation. médecine/sciences. 40(5). 428–436.

Roubinet, Benoît, Mylène Pezet, Michel Thépaut, et al.. (2023). Melanoma tumour‐derived glycans hijack dendritic cell subsets through C‐type lectin receptor binding. Immunology. 171(2). 286–311. 7 indexed citations

Saas, Philippe, et al.. (2023). Dendritic Cell Subsets in Melanoma: Pathophysiology, Clinical Prognosis and Therapeutic Exploitation. Cancers. 15(8). 2206–2206. 9 indexed citations

Barros, Jean-Paul Paı̈s de, et al.. (2023). Resolved Psoriasis with Abundant Oleic Acid in Stratum Corneum Exhibits Lower T-Cell–Driven IL-17 Signature. Journal of Investigative Dermatology. 143(11). 2145–2152.e6. 4 indexed citations

10.

Laheurte, Caroline, Laura Boullerot, Marine Malfroy, et al.. (2022). Interplay between plasmacytoid dendritic cells and tumor-specific T cells in peripheral blood influences long-term survival in non-small cell lung carcinoma. Cancer Immunology Immunotherapy. 72(3). 579–589. 5 indexed citations

11.

Audia, Sylvain, T. Moulinet, Marion Ciudad, et al.. (2018). Altered distribution and function of splenic innate lymphoid cells in adult chronic immune thrombocytopenia. Journal of Autoimmunity. 93. 139–144. 8 indexed citations

12.

Weil, Delphine, Jean-Paul Paı̈s de Barros, Guillaume Mourey, et al.. (2018). Circulating levels of 3‐hydroxymyristate, a direct quantification of endotoxaemia in noninfected cirrhotic patients. Liver International. 39(1). 106–114. 9 indexed citations

13.

Courivaud, Cécile, Jamal Bamoulid, Caroline Roubiou, et al.. (2014). Alloimmune Responses and Atherosclerotic Disease After Kidney Transplantation. Transplantation. 99(1). 220–225. 10 indexed citations

14.

Binda, Delphine, et al.. (2014). In vitro study of the impact of mechanical tension on the dermal fibroblast phenotype in the context of skin wound healing. Journal of Biomechanics. 47(14). 3555–3561. 36 indexed citations

15.

Godet, Yann, Elizabeth Fabre, Magalie Dosset, et al.. (2012). Analysis of Spontaneous Tumor-Specific CD4 T-cell Immunity in Lung Cancer Using Promiscuous HLA-DR Telomerase-Derived Epitopes: Potential Synergistic Effect with Chemotherapy Response. Clinical Cancer Research. 18(10). 2943–2953. 76 indexed citations

16.

Legrand, Faézeh, Frédéric Grenouillet, Fabrice Larosa, et al.. (2010). Diagnosis and treatment of digestive cryptosporidiosis in allogeneic haematopoietic stem cell transplant recipients: a prospective single centre study. Bone Marrow Transplantation. 46(6). 858–862. 29 indexed citations

17.

Toussirot, Éric, Philippe Saas, Marina Deschamps, et al.. (2009). Increased production of soluble CTLA-4 in patients with spondylarthropathies correlates with disease activity. Arthritis Research & Therapy. 11(4). R101–R101. 44 indexed citations

18.

Pallandre, Jean‐René, David Chalmers, Bernhard Ryffel, et al.. (2007). Natural killer cells prevent CD28-mediated Foxp3 transcription in CD4+CD25– T lymphocytes. Experimental Hematology. 35(3). 416–425. 42 indexed citations

19.

Perruche, Sylvain, R Angonin, Jean‐Yves Cahn, et al.. (2003). A Single Intravenous Infusion of Apoptotic Cells, An Alternative Cell-Based Therapy Approach Facilitating Hematopoietic Cell Engraftment, Did Not Induce Autoimmunity. Journal of Hematotherapy & Stem Cell Research. 12(4). 451–459. 11 indexed citations

20.

Sauce, Delphine, Anne Duperrier, Manoel de Carvalho, et al.. (2002). Influence of Ex Vivo Expansion and Retrovirus-Mediated Gene Transfer on Primary T Lymphocyte Phenotype and Functions. Journal of Hematotherapy & Stem Cell Research. 11(6). 929–940. 26 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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