Philip Koh

1.3k total citations
8 papers, 1.1k citations indexed

About

Philip Koh is a scholar working on Molecular Biology, Nephrology and Genetics. According to data from OpenAlex, Philip Koh has authored 8 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 2 papers in Nephrology and 2 papers in Genetics. Recurrent topics in Philip Koh's work include Renal and related cancers (2 papers), Chronic Kidney Disease and Diabetes (2 papers) and Kruppel-like factors research (2 papers). Philip Koh is often cited by papers focused on Renal and related cancers (2 papers), Chronic Kidney Disease and Diabetes (2 papers) and Kruppel-like factors research (2 papers). Philip Koh collaborates with scholars based in Australia, United Kingdom and United States. Philip Koh's co-authors include Mark E. Cooper, Karin Jandeleit‐Dahm, Phillip Kantharidis, Bo Wang, Catherine E. Winbanks, Merlin C. Thomas, Michal Herman‐Edelstein, Anna M.D. Watson, Wendy C. Burns and Paul Gregorevic and has published in prestigious journals such as Nucleic Acids Research, Molecular and Cellular Biology and Diabetes.

In The Last Decade

Philip Koh

8 papers receiving 1.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Philip Koh Australia 7 730 529 184 102 102 8 1.1k
Xiaoyan Bai China 19 605 0.8× 399 0.8× 187 1.0× 92 0.9× 316 3.1× 55 1.2k
Cuiyan Xin United States 5 669 0.9× 439 0.8× 249 1.4× 106 1.0× 101 1.0× 7 1.0k
Piera Trionfini Italy 10 496 0.7× 304 0.6× 170 0.9× 47 0.5× 54 0.5× 19 798
Sumanth Putta United States 11 1.4k 1.9× 1.1k 2.1× 294 1.6× 166 1.6× 90 0.9× 13 1.9k
Jose L. Viñas Canada 13 549 0.8× 272 0.5× 196 1.1× 114 1.1× 70 0.7× 20 895
Jessica M. Overstreet United States 15 633 0.9× 190 0.4× 321 1.7× 125 1.2× 183 1.8× 16 1.3k
Vivek Kaimal United States 9 803 1.1× 744 1.4× 98 0.5× 238 2.3× 92 0.9× 11 1.2k
Xiangchen Gu China 11 678 0.9× 341 0.6× 203 1.1× 81 0.8× 94 0.9× 24 1.1k
Allie M. Roach United States 8 390 0.5× 192 0.4× 203 1.1× 62 0.6× 76 0.7× 10 676

Countries citing papers authored by Philip Koh

Since Specialization
Citations

This map shows the geographic impact of Philip Koh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Philip Koh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Philip Koh more than expected).

Fields of papers citing papers by Philip Koh

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Philip Koh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Philip Koh. The network helps show where Philip Koh may publish in the future.

Co-authorship network of co-authors of Philip Koh

This figure shows the co-authorship network connecting the top 25 collaborators of Philip Koh. A scholar is included among the top collaborators of Philip Koh based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Philip Koh. Philip Koh is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Funnell, Alister P. W., Laura J. Norton, Ka Sin Mak, et al.. (2012). The CACCC-Binding Protein KLF3/BKLF Represses a Subset of KLF1/EKLF Target Genes and Is Required for Proper Erythroid Maturation In Vivo. Molecular and Cellular Biology. 32(16). 3281–3292. 35 indexed citations
2.
Wang, Bo, Radko Komers, Catherine E. Winbanks, et al.. (2011). Suppression of microRNA-29 Expression by TGF-β1 Promotes Collagen Expression and Renal Fibrosis. Journal of the American Society of Nephrology. 23(2). 252–265. 426 indexed citations
3.
Koh, Philip, et al.. (2010). PCAF interacts with XBP-1S and mediates XBP-1S-dependent transcription. Nucleic Acids Research. 39(2). 429–439. 24 indexed citations
4.
Wang, Bo, Michal Herman‐Edelstein, Philip Koh, et al.. (2010). E-Cadherin Expression Is Regulated by miR-192/215 by a Mechanism That Is Independent of the Profibrotic Effects of Transforming Growth Factor-β. Diabetes. 59(7). 1794–1802. 221 indexed citations
5.
Wang, Bo, Philip Koh, Catherine E. Winbanks, et al.. (2010). miR-200a Prevents Renal Fibrogenesis Through Repression of TGF-β2 Expression. Diabetes. 60(1). 280–287. 296 indexed citations
6.
Koı̈tka, Audrey, Zemin Cao, Philip Koh, et al.. (2009). Angiotensin II subtype 2 receptor blockade and deficiency attenuate the development of atherosclerosis in an apolipoprotein E-deficient mouse model of diabetes. Diabetologia. 53(3). 584–592. 30 indexed citations
7.
Chew, Phyllis, Derek Y.C. Yuen, Philip Koh, et al.. (2009). Site-Specific Antiatherogenic Effect of the Antioxidant Ebselen in the Diabetic Apolipoprotein E–Deficient Mouse. Arteriosclerosis Thrombosis and Vascular Biology. 29(6). 823–830. 73 indexed citations
8.
Tikellis, Chris, Philip Koh, Wendy C. Burns, & Phillip Kantharidis. (2008). Quantitative Gene Expression Analysis in Kidney Tissues. Methods in molecular biology. 466. 83–107. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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