Peter Schneiderat

1.2k total citations
19 papers, 598 citations indexed

About

Peter Schneiderat is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Clinical Biochemistry. According to data from OpenAlex, Peter Schneiderat has authored 19 papers receiving a total of 598 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 4 papers in Cellular and Molecular Neuroscience and 4 papers in Clinical Biochemistry. Recurrent topics in Peter Schneiderat's work include Muscle Physiology and Disorders (6 papers), Mitochondrial Function and Pathology (5 papers) and Metabolism and Genetic Disorders (4 papers). Peter Schneiderat is often cited by papers focused on Muscle Physiology and Disorders (6 papers), Mitochondrial Function and Pathology (5 papers) and Metabolism and Genetic Disorders (4 papers). Peter Schneiderat collaborates with scholars based in Germany, United Kingdom and United States. Peter Schneiderat's co-authors include Hanns Lochmüller, Rita Horváth, Benedikt Schoser, Maggie C. Walter, Ali Naini, Klaus Gempel, Beril Talim, Holger Prokisch, Haluk Topaloğlu and Ayşegül Tokatlı and has published in prestigious journals such as PLoS ONE, Brain and Neurology.

In The Last Decade

Peter Schneiderat

19 papers receiving 589 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Peter Schneiderat Germany 13 487 190 97 89 60 19 598
Jorida Çoku United States 12 459 0.9× 204 1.1× 40 0.4× 49 0.6× 26 0.4× 18 525
Stephanie Demuth Germany 9 270 0.6× 32 0.2× 25 0.3× 40 0.4× 25 0.4× 11 407
Lucia Valente Italy 10 540 1.1× 251 1.3× 14 0.1× 6 0.1× 51 0.8× 11 644
Y. Fujita Japan 6 686 1.4× 214 1.1× 17 0.2× 20 0.2× 34 0.6× 17 809
Akihiko Miyauchi Japan 11 189 0.4× 85 0.4× 36 0.4× 5 0.1× 45 0.8× 24 326
Tomoko Lee Japan 14 317 0.7× 61 0.3× 16 0.2× 3 0.0× 42 0.7× 43 400
Andrea Lim Singapore 11 283 0.6× 20 0.1× 12 0.1× 10 0.1× 11 0.2× 15 474
Patrizia Annunziata Italy 13 191 0.4× 56 0.3× 53 0.5× 3 0.0× 9 0.1× 19 407
Nobuko Matsushita Japan 10 599 1.2× 94 0.5× 15 0.2× 4 0.0× 58 1.0× 12 808
Elena Perli Italy 11 411 0.8× 117 0.6× 8 0.1× 5 0.1× 28 0.5× 14 529

Countries citing papers authored by Peter Schneiderat

Since Specialization
Citations

This map shows the geographic impact of Peter Schneiderat's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Peter Schneiderat with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Peter Schneiderat more than expected).

Fields of papers citing papers by Peter Schneiderat

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Peter Schneiderat. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Peter Schneiderat. The network helps show where Peter Schneiderat may publish in the future.

Co-authorship network of co-authors of Peter Schneiderat

This figure shows the co-authorship network connecting the top 25 collaborators of Peter Schneiderat. A scholar is included among the top collaborators of Peter Schneiderat based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Peter Schneiderat. Peter Schneiderat is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Kroidl, Inge, Peter Schneiderat, Katharina Müller, et al.. (2021). Vaccine breakthrough infection and onward transmission of SARS-CoV-2 Beta (B.1.351) variant, Bavaria, Germany, February to March 2021. Eurosurveillance. 26(30). 12 indexed citations
2.
Schrank, Bertold, Benedikt Schoser, Thomas Klopstock, et al.. (2017). Lifetime exercise intolerance with lactic acidosis as key manifestation of novel compound heterozygous ACAD9 mutations causing complex I deficiency. Neuromuscular Disorders. 27(5). 473–476. 10 indexed citations
3.
Oliveira, Cibely Cristine Fontes de, et al.. (2017). Bioenergetic Impairment in Congenital Muscular Dystrophy Type 1A and Leigh Syndrome Muscle Cells. Scientific Reports. 7(1). 25 indexed citations
4.
Schneiderat, Peter, et al.. (2015). Generation of a human iPSC line from a patient with a defect of intergenomic communication. Stem Cell Research. 16(1). 120–123. 5 indexed citations
5.
Meinke, Peter, Peter Schneiderat, Vlastimil Sršeň, et al.. (2014). Abnormal proliferation and spontaneous differentiation of myoblasts from a symptomatic female carrier of X-linked Emery–Dreifuss muscular dystrophy. Neuromuscular Disorders. 25(2). 127–136. 14 indexed citations
6.
Schneiderat, Peter, Wolfram Kreß, Bernd Rautenstrauß, et al.. (2013). Facioscapulohumeral muscular dystrophy and Charcot-Marie-Tooth neuropathy 1A - evidence for “double trouble” overlapping syndromes. BMC Medical Genetics. 14(1). 92–92. 17 indexed citations
7.
Stevens, Elizabeth, Silvia Torelli, Lucy Feng, et al.. (2013). Flow Cytometry for the Analysis of α-Dystroglycan Glycosylation in Fibroblasts from Patients with Dystroglycanopathies. PLoS ONE. 8(7). e68958–e68958. 13 indexed citations
8.
Harafuji, Naoe, Peter Schneiderat, Maggie C. Walter, & Yiwen Chen. (2013). miR-411 is up-regulated in FSHD myoblasts and suppresses myogenic factors. Orphanet Journal of Rare Diseases. 8(1). 55–55. 41 indexed citations
9.
Yoon, Soonsang, Guido Stadler, Mary Lou Beermann, et al.. (2013). Immortalized myogenic cells from congenital muscular dystrophy type1A patients recapitulate aberrant caspase activation in pathogenesis: a new tool for MDC1A research. Skeletal Muscle. 3(1). 28–28. 8 indexed citations
10.
Büchner, Boriana, Constanze Gallenmüller, Klaus A. Kuhn, et al.. (2012). Das Deutsche Netzwerk für mitochondriale Erkrankungen (mitoNET). Medizinische Genetik. 24(3). 193–199. 2 indexed citations
11.
Reilich, Peter, Sabine Krause, Nicolai Schramm, et al.. (2011). A novel mutation in the myotilin gene (MYOT) causes a severe form of limb girdle muscular dystrophy 1A (LGMD1A). Journal of Neurology. 258(8). 1437–1444. 22 indexed citations
12.
Reilich, Peter, Nicolai Schramm, Benedikt Schoser, et al.. (2010). Facioscapulohumeral muscular dystrophy presenting with unusual phenotypes and atypical morphological features of vacuolar myopathy. Journal of Neurology. 257(7). 1108–1118. 20 indexed citations
13.
Walter, Maggie C., Birgit Czermin, Stefanie Bulst, et al.. (2010). Late-onset ptosis and myopathy in a patient with a heterozygous insertion in POLG2. Journal of Neurology. 257(9). 1517–1523. 35 indexed citations
14.
Lochmüller, Hanns & Peter Schneiderat. (2010). Biobanking in Rare Disorders. Advances in experimental medicine and biology. 686. 105–113. 13 indexed citations
15.
Erol, İlknur, et al.. (2009). Demyelinating disease of central and peripheral nervous systems associated with a A8344G mutation in tRNALys. Neuromuscular Disorders. 19(4). 275–278. 19 indexed citations
16.
Horváth, Rita, Andreas Bender, Angela Abicht, et al.. (2009). Heteroplasmic mutation in the anticodon-stem of mitochondrial tRNAVal causing MNGIE-like gastrointestinal dysmotility and cachexia. Journal of Neurology. 256(5). 810–815. 23 indexed citations
17.
Gempel, Klaus, Haluk Topaloğlu, Beril Talim, et al.. (2007). The myopathic form of coenzyme Q10 deficiency is caused by mutations in the electron-transferring-flavoprotein dehydrogenase (ETFDH) gene. Brain. 130(8). 2037–2044. 231 indexed citations
18.
Horváth, Rita, Peter Schneiderat, Benedikt Schoser, et al.. (2006). Coenzyme Q10 deficiency and isolated myopathy. Neurology. 66(2). 253–255. 83 indexed citations
19.
Braess, Jan, et al.. (2004). Functional analysis of apoptosis induction in acute myeloid leukaemia‐relevance of karyotype and clinical treatment response. British Journal of Haematology. 126(3). 338–347. 5 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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