Peter H. Heidemann

1.0k total citations
18 papers, 607 citations indexed

About

Peter H. Heidemann is a scholar working on Molecular Biology, Genetics and Surgery. According to data from OpenAlex, Peter H. Heidemann has authored 18 papers receiving a total of 607 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 9 papers in Genetics and 5 papers in Surgery. Recurrent topics in Peter H. Heidemann's work include Lysosomal Storage Disorders Research (4 papers), Glycosylation and Glycoproteins Research (3 papers) and Cellular transport and secretion (3 papers). Peter H. Heidemann is often cited by papers focused on Lysosomal Storage Disorders Research (4 papers), Glycosylation and Glycoproteins Research (3 papers) and Cellular transport and secretion (3 papers). Peter H. Heidemann collaborates with scholars based in Germany, United States and Switzerland. Peter H. Heidemann's co-authors include Martin Hasilik, Georg F. Hoffmann, Christian Thiel, Verena Peters, Christian Körner, Kurt Von Figura, Torben Lübke, Stefan Höning, Eric G. Berger and Dirk Schnabel and has published in prestigious journals such as Journal of Clinical Investigation, PLoS ONE and The Journal of Clinical Endocrinology & Metabolism.

In The Last Decade

Peter H. Heidemann

18 papers receiving 585 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Peter H. Heidemann Germany 12 360 234 130 80 64 18 607
Yi-Fen Lee United States 16 397 1.1× 179 0.8× 71 0.5× 54 0.7× 68 1.1× 20 740
Asta Valančiūtė France 11 281 0.8× 69 0.3× 61 0.5× 26 0.3× 124 1.9× 15 706
Paolo Lova Italy 11 141 0.4× 74 0.3× 74 0.6× 59 0.7× 65 1.0× 13 631
Maureen A. Nickols United States 9 284 0.8× 50 0.2× 20 0.2× 89 1.1× 49 0.8× 10 572
Yi Ming Mu China 9 405 1.1× 43 0.2× 62 0.5× 62 0.8× 46 0.7× 11 623
L L Shelly United States 12 397 1.1× 262 1.1× 170 1.3× 39 0.5× 47 0.7× 13 985
Takehiko Murakami Japan 14 536 1.5× 62 0.3× 77 0.6× 178 2.2× 27 0.4× 20 805
Sanghamitra Sahoo United States 17 285 0.8× 228 1.0× 54 0.4× 22 0.3× 205 3.2× 21 801
China Nagano Japan 15 361 1.0× 154 0.7× 34 0.3× 25 0.3× 19 0.3× 79 630
Elena Kleymenova United States 14 400 1.1× 162 0.7× 64 0.5× 36 0.5× 20 0.3× 20 694

Countries citing papers authored by Peter H. Heidemann

Since Specialization
Citations

This map shows the geographic impact of Peter H. Heidemann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Peter H. Heidemann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Peter H. Heidemann more than expected).

Fields of papers citing papers by Peter H. Heidemann

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Peter H. Heidemann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Peter H. Heidemann. The network helps show where Peter H. Heidemann may publish in the future.

Co-authorship network of co-authors of Peter H. Heidemann

This figure shows the co-authorship network connecting the top 25 collaborators of Peter H. Heidemann. A scholar is included among the top collaborators of Peter H. Heidemann based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Peter H. Heidemann. Peter H. Heidemann is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Vogt, P. H., Markus Bettendorf, Petra Frank‐Herrmann, et al.. (2022). Sex chromosome DSD individuals with mosaic 45,X0 and aberrant Y chromosomes in 46,XY cells: distinct gender phenotypes and germ cell tumour risks§. Systems Biology in Reproductive Medicine. 68(4). 247–257. 4 indexed citations
2.
Dunstheimer, Désirée, Jürgen Klammt, Daniela Friebe, et al.. (2012). Clinical and Functional Characterization of a Patient Carrying a Compound Heterozygous Pericentrin Mutation and a Heterozygous IGF1 Receptor Mutation. PLoS ONE. 7(5). e38220–e38220. 15 indexed citations
3.
Raue, Friedhelm, J. Pichl, Dirk Schnabel, et al.. (2011). Activating mutations in the calcium-sensing receptor: genetic and clinical spectrum in 25 patients with autosomal dominant hypocalcaemia - a German survey. Clinical Endocrinology. 75(6). 760–765. 64 indexed citations
4.
Köhler, Birgit, Lin Lin, Bruno Ferraz‐de‐Souza, et al.. (2007). Five novel mutations in steroidogenic factor 1 (SF1,NR5A1) in 46,XY patients with severe underandrogenization but without adrenal insufficiency. Human Mutation. 29(1). 59–64. 106 indexed citations
5.
Hehr, Ute, et al.. (2004). Wide phenotypic variability in families with holoprosencephaly and a sonic hedgehog mutation. European Journal of Pediatrics. 163(7). 347–52. 40 indexed citations
6.
Binder, Gerhard, et al.. (2004). Insulin Tolerance Test Causes Hypokalaemia and Can Provoke Cardiac Arrhythmias. Hormone Research in Paediatrics. 62(2). 84–87. 28 indexed citations
7.
Karges, Wölfram, Beate Karges, Leopold Ludwig, & Peter H. Heidemann. (2003). Bi-allelic inactivation of the MEN1 tumor suppressor gene in human grade II astrocytoma☆. Cancer Letters. 196(1). 23–27. 2 indexed citations
8.
Thiel, Christian, Torben Lübke, Martin Hasilik, et al.. (2002). Deficiency of UDP-galactose:N-acetylglucosamine β-1,4-galactosyltransferase I causes the congenital disorder of glycosylation type IId. Journal of Clinical Investigation. 109(6). 725–733. 76 indexed citations
9.
Thiel, Christian, Torben Lübke, Martin Hasilik, et al.. (2002). Deficiency of UDP-galactose:N-acetylglucosamine β-1,4-galactosyltransferase I causes the congenital disorder of glycosylation type IId. Journal of Clinical Investigation. 109(6). 725–733. 103 indexed citations
10.
Thiel, Christian, Torben Lübke, Martin Hasilik, et al.. (2002). Deficiency of UDP-galactose:N-acetylglucosamine β-1,4-galactosyltransferase I causes the congenital disorder of glycosylation type IId. Journal of Clinical Investigation. 109(6). 725–733. 11 indexed citations
11.
Apel, Thomas W., et al.. (2001). Phaeochromocytoma associated with a de novo VHL mutation as form fruste of von Hippel-Lindau disease. European Journal of Pediatrics. 160(7). 421–424. 6 indexed citations
12.
Bender, Bernhard U., Andrzej Januszewicz, Roland Gärtner, et al.. (1997). Functioning Thoracic Paraganglioma: Association with Von Hippel-Lindau Syndrome*. The Journal of Clinical Endocrinology & Metabolism. 82(10). 3356–3360. 16 indexed citations
13.
Balda, B.-R., A. Konstantinow, Hans Starz, Astrid Gnekow, & Peter H. Heidemann. (1996). Extracorporeal photochemotherapy as an effective treatment modality in chronic graft‐versus‐host disease. Journal of the European Academy of Dermatology and Venereology. 7(2). 155–162. 14 indexed citations
14.
Seo, Hee‐Chan, et al.. (1995). A nonsense mutation in two German patients with fucosidosis. Human Mutation. 6(2). 184–185. 5 indexed citations
15.
16.
Heidemann, Peter H., James L. Wittliff, Robert E. Calhoon, & Elizabeth S. Boylan. (1981). Influence of prenatal exposure to diethylstilbestrol on estrogen and progestin binding proteins in uteri and d1methylbenzanthracene‐induced mammary tumors of the rat. Journal of Toxicology and Environmental Health. 8(4). 667–686. 4 indexed citations
17.
Kute, Timothy E., et al.. (1978). Molecular heterogeneity of cytosolic forms of estrogen receptors from human breast tumors.. Munich Personal RePEc Archive (Ludwig Maximilian University of Munich). 38(11 Pt 2). 4307–13. 47 indexed citations
18.
Heidemann, Peter H. & P. Stubbe. (1978). Serum 3,5,3′-Triiodothyronine, Thyroxine, and Thyrotropin in Hypothyroid Infants with Congenital Goiter and the Response to Iodine. The Journal of Clinical Endocrinology & Metabolism. 47(1). 189–192. 12 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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