Peng‐Cheng Wang

552 total citations
11 papers, 355 citations indexed

About

Peng‐Cheng Wang is a scholar working on Molecular Biology, Cancer Research and Pathology and Forensic Medicine. According to data from OpenAlex, Peng‐Cheng Wang has authored 11 papers receiving a total of 355 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 6 papers in Cancer Research and 2 papers in Pathology and Forensic Medicine. Recurrent topics in Peng‐Cheng Wang's work include Circular RNAs in diseases (7 papers), MicroRNA in disease regulation (6 papers) and Cardiovascular, Neuropeptides, and Oxidative Stress Research (1 paper). Peng‐Cheng Wang is often cited by papers focused on Circular RNAs in diseases (7 papers), MicroRNA in disease regulation (6 papers) and Cardiovascular, Neuropeptides, and Oxidative Stress Research (1 paper). Peng‐Cheng Wang collaborates with scholars based in China and United States. Peng‐Cheng Wang's co-authors include Zheng‐Jun Zhou, Li Mao, Shao‐Lai Zhou, Songyang Yu, Jia Li, Zhiqiang Hu, Xiaowu Huang, Jia Fan, Haoyang Xin and Ya Cao and has published in prestigious journals such as Nature Communications, Hepatology and Cancer Research.

In The Last Decade

Peng‐Cheng Wang

9 papers receiving 355 citations

Peers

Peng‐Cheng Wang
Chun‐Xiang Ye China
Youzhi Wu China
Zetian Chen China
Sara García-Duque Spain
Yunhui Liu China
Hao Jin China
Haoxian Zhou China
Guangcheng Guo China
Chun‐Xiang Ye China
Peng‐Cheng Wang
Citations per year, relative to Peng‐Cheng Wang Peng‐Cheng Wang (= 1×) peers Chun‐Xiang Ye

Countries citing papers authored by Peng‐Cheng Wang

Since Specialization
Citations

This map shows the geographic impact of Peng‐Cheng Wang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Peng‐Cheng Wang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Peng‐Cheng Wang more than expected).

Fields of papers citing papers by Peng‐Cheng Wang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Peng‐Cheng Wang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Peng‐Cheng Wang. The network helps show where Peng‐Cheng Wang may publish in the future.

Co-authorship network of co-authors of Peng‐Cheng Wang

This figure shows the co-authorship network connecting the top 25 collaborators of Peng‐Cheng Wang. A scholar is included among the top collaborators of Peng‐Cheng Wang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Peng‐Cheng Wang. Peng‐Cheng Wang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
1.
Xia, Yuhao, Peng‐Cheng Wang, Dibo Hou, et al.. (2025). High-accuracy protein complex structure modeling based on sequence-derived structure complementarity. Nature Communications. 16(1). 10182–10182.
2.
Wang, Peng‐Cheng, et al.. (2025). The Mitochondrial-targeting Drug SkQ1 Attenuates the Progression of Posttraumatic Osteoarthritis through Suppression of Mitochondrial Oxidative Stress. Current Molecular Pharmacology. 17. e18761429383749–e18761429383749. 2 indexed citations
3.
Yu, Songyang, Peng‐Cheng Wang, Jia Li, et al.. (2024). Tumor-associated macrophage-induced circMRCKα encodes a peptide to promote glycolysis and progression in hepatocellular carcinoma. Cancer Letters. 591. 216872–216872. 11 indexed citations
4.
Wei, Wei, Hui Zhang, Wei Yang, et al.. (2024). Dual electrical stimulation at spinal-muscular interface reconstructs spinal sensorimotor circuits after spinal cord injury. Nature Communications. 15(1). 619–619. 20 indexed citations
5.
Wang, Peng‐Cheng, et al.. (2024). E3 ubiquitin ligase BTBD3 inhibits tumorigenesis of colorectal cancer by regulating the TYRO3/Wnt/β-catenin signaling axis. Cancer Cell International. 24(1). 306–306.
6.
Yang, Xinyi, et al.. (2023). Harnessing ADAR-Mediated Site-Specific RNA Editing in Immune-Related Disease: Prediction and Therapeutic Implications. International Journal of Molecular Sciences. 25(1). 351–351. 1 indexed citations
7.
Li, Jia, Zhiqiang Hu, Songyang Yu, et al.. (2022). CircRPN2 Inhibits Aerobic Glycolysis and Metastasis in Hepatocellular Carcinoma. Cancer Research. 82(6). 1055–1069. 100 indexed citations
8.
Wang, Peng‐Cheng, et al.. (2022). Circular RNA circ_0079593 facilitates glioma development via modulating miR-324-5p/XBP1 axis. Metabolic Brain Disease. 37(7). 2389–2403. 3 indexed citations
9.
Ruan, Hang, Peng‐Cheng Wang, & Leng Han. (2022). Characterization of circular RNAs with advanced sequencing technologies in human complex diseases. Wiley Interdisciplinary Reviews - RNA. 14(1). e1759–e1759. 13 indexed citations
10.
Xu, Jianhua, et al.. (2020). circRAE1 promotes colorectal cancer cell migration and invasion by modulating miR-338-3p/TYRO3 axis. Cancer Cell International. 20(1). 430–430. 24 indexed citations
11.
Hu, Zhiqiang, Shao‐Lai Zhou, Jia Li, et al.. (2019). Circular RNA Sequencing Identifies CircASAP1 as a Key Regulator in Hepatocellular Carcinoma Metastasis. Hepatology. 72(3). 906–922. 181 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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