Pengcheng Dou

556 total citations
18 papers, 446 citations indexed

About

Pengcheng Dou is a scholar working on Cancer Research, Rheumatology and Molecular Biology. According to data from OpenAlex, Pengcheng Dou has authored 18 papers receiving a total of 446 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Cancer Research, 7 papers in Rheumatology and 6 papers in Molecular Biology. Recurrent topics in Pengcheng Dou's work include Osteoarthritis Treatment and Mechanisms (7 papers), Cancer-related molecular mechanisms research (6 papers) and Inflammatory mediators and NSAID effects (4 papers). Pengcheng Dou is often cited by papers focused on Osteoarthritis Treatment and Mechanisms (7 papers), Cancer-related molecular mechanisms research (6 papers) and Inflammatory mediators and NSAID effects (4 papers). Pengcheng Dou collaborates with scholars based in China and United States. Pengcheng Dou's co-authors include Tang Liu, Zhihong Li, Juan Duan, Wanchun Wang, Lian‐Jun Bao, Eddy Y. Zeng, Lei Mai, Zhengxiao Ouyang, Xiaoning Guo and Tingting Tan and has published in prestigious journals such as Biomaterials, Marine Pollution Bulletin and BioMed Research International.

In The Last Decade

Pengcheng Dou

17 papers receiving 443 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Pengcheng Dou China 13 241 192 97 48 41 18 446
Hyeongseok Yun South Korea 10 316 1.3× 70 0.4× 75 0.8× 30 0.6× 8 0.2× 20 586
Zhengang Zha China 11 228 0.9× 71 0.4× 49 0.5× 21 0.4× 14 0.3× 21 381
Rolando André Rios Villacis Brazil 14 190 0.8× 101 0.5× 29 0.3× 33 0.7× 27 0.7× 30 458
Jinzhong Ma China 11 274 1.1× 88 0.5× 172 1.8× 126 2.6× 6 0.1× 34 568
Guo Qiang Chen China 11 272 1.1× 81 0.4× 26 0.3× 28 0.6× 43 1.0× 29 511
Mingjie Tang China 13 279 1.2× 227 1.2× 16 0.2× 72 1.5× 64 1.6× 23 518
Yixin He Hong Kong 10 253 1.0× 40 0.2× 38 0.4× 106 2.2× 24 0.6× 19 520
Yamei Wu China 13 309 1.3× 119 0.6× 15 0.2× 26 0.5× 129 3.1× 43 701
Ria Margiana Indonesia 9 128 0.5× 50 0.3× 17 0.2× 61 1.3× 45 1.1× 47 448
Shuting Zhang China 7 140 0.6× 40 0.2× 68 0.7× 12 0.3× 25 0.6× 19 281

Countries citing papers authored by Pengcheng Dou

Since Specialization
Citations

This map shows the geographic impact of Pengcheng Dou's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Pengcheng Dou with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Pengcheng Dou more than expected).

Fields of papers citing papers by Pengcheng Dou

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Pengcheng Dou. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Pengcheng Dou. The network helps show where Pengcheng Dou may publish in the future.

Co-authorship network of co-authors of Pengcheng Dou

This figure shows the co-authorship network connecting the top 25 collaborators of Pengcheng Dou. A scholar is included among the top collaborators of Pengcheng Dou based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Pengcheng Dou. Pengcheng Dou is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Wu, Ziyi, Xin Luo, Pengcheng Dou, Tang Liu, & Jian Zhou. (2025). Asthma, social isolation and loneliness, and risk of incident osteoarthritis. Arthritis Research & Therapy. 27(1). 29–29.
2.
Zeng, Jing, Xianghong Zhang, Zhengjun Lin, et al.. (2024). Harnessing ferroptosis for enhanced sarcoma treatment: mechanisms, progress and prospects. Experimental Hematology and Oncology. 13(1). 31–31. 6 indexed citations
3.
Long, Ze, et al.. (2023). MiR-181a-5p promotes osteogenesis by targeting BMP3. Aging. 15(3). 734–747. 20 indexed citations
4.
Cao, Ziqin, Yajia Li, Fuqiang Gao, et al.. (2021). Mesenchymal Stem Cells: A New Choice for Nonsurgical Treatment of OA? Results from a Bayesian Network Meta‐Analysis. BioMed Research International. 2021(1). 6663003–6663003. 5 indexed citations
5.
Duan, Juan, et al.. (2021). Cimifugin Suppresses NF-κB Signaling to Prevent Osteoclastogenesis and Periprosthetic Osteolysis. Frontiers in Pharmacology. 12. 724256–724256. 12 indexed citations
6.
Dou, Pengcheng, et al.. (2021). MicroRNA-9 facilitates hypoxia-induced injury and apoptosis in H9c2 cells via targeting CDK8. Journal of Biosciences. 46(1). 4 indexed citations
7.
Dou, Pengcheng, Lei Mai, Lian‐Jun Bao, & Eddy Y. Zeng. (2021). Microplastics on beaches and mangrove sediments along the coast of South China. Marine Pollution Bulletin. 172. 112806–112806. 42 indexed citations
8.
Cao, Ziqin, Yajia Li, Wanchun Wang, et al.. (2020). Is Lutikizumab, an Anti–Interleukin‐1 α / β Dual Variable Domain Immunoglobulin, efficacious for Osteoarthritis? Results from a bayesian network meta‐analysis. BioMed Research International. 2020(1). 9013283–9013283. 8 indexed citations
9.
Yin, Ziqing, Xia Chen, Guangyao Jiang, et al.. (2020). Tussilagone Inhibits Osteoclastogenesis and Periprosthetic Osteolysis by Suppressing the NF-κB and P38 MAPK Signaling Pathways. Frontiers in Pharmacology. 11. 385–385. 17 indexed citations
10.
Dou, Pengcheng, Yu He, Bo Yu, & Juan Duan. (2020). Downregulation of microRNA-29b by DNMT3B decelerates chondrocyte apoptosis and the progression of osteoarthritis via PTHLH/CDK4/RUNX2 axis. Aging. 13(5). 7676–7690. 20 indexed citations
11.
Ouyang, Zhengxiao, Tingting Tan, Chun‐Feng Liu, et al.. (2019). Targeted delivery of hesperetin to cartilage attenuates osteoarthritis by bimodal imaging with Gd2(CO3)3@PDA nanoparticles via TLR-2/NF-κB/Akt signaling. Biomaterials. 205. 50–63. 81 indexed citations
12.
Zhang, Haibin, Yinghong Cui, Yuqing Li, et al.. (2019). lnc-SAMD14-4 can regulate expression of the COL1A1 and COL1A2 in human chondrocytes. PeerJ. 7. e7491–e7491. 13 indexed citations
14.
Li, Ding, et al.. (2018). A Novel Peptide from <i>Vespa ducalis</i> Induces Apoptosis in Osteosarcoma Cells by Activating the p38 MAPK and JNK Signaling Pathways. Biological and Pharmaceutical Bulletin. 41(4). 458–464. 17 indexed citations
15.
Li, Zhihong, et al.. (2017). Application of Long Noncoding RNAs in Osteosarcoma: Biomarkers and Therapeutic Targets. Cellular Physiology and Biochemistry. 42(4). 1407–1419. 127 indexed citations
16.
Dou, Pengcheng, Weihong Zhu, Qi Tang, et al.. (2017). Long non-coding RNA HOTAIR promotes expression of ADAMTS-5 in human osteoarthritic articular chondrocytes.. PubMed. 72(2). 113–117. 24 indexed citations
17.
Liu, Qian, Wanchun Wang, Andrew R. Thoreson, et al.. (2016). Finite element prediction of contact pressures in cam-type femoroacetabular impingement with varied alpha angles. Computer Methods in Biomechanics & Biomedical Engineering. 20(3). 294–301. 13 indexed citations
18.
Duan, Juan, Yu Yang, Hong Liu, Pengcheng Dou, & Shengyu Tan. (2015). Osthole ameliorates acute myocardial infarction in rats by decreasing the expression of inflammatory-related cytokines, diminishing MMP-2 expression and activating p-ERK. International Journal of Molecular Medicine. 37(1). 207–216. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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