Paulo Correia‐de‐Sá

3.5k total citations
126 papers, 2.8k citations indexed

About

Paulo Correia‐de‐Sá is a scholar working on Physiology, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Paulo Correia‐de‐Sá has authored 126 papers receiving a total of 2.8k indexed citations (citations by other indexed papers that have themselves been cited), including 63 papers in Physiology, 48 papers in Molecular Biology and 36 papers in Cellular and Molecular Neuroscience. Recurrent topics in Paulo Correia‐de‐Sá's work include Adenosine and Purinergic Signaling (62 papers), Ion channel regulation and function (29 papers) and Neuroscience and Neuropharmacology Research (21 papers). Paulo Correia‐de‐Sá is often cited by papers focused on Adenosine and Purinergic Signaling (62 papers), Ion channel regulation and function (29 papers) and Neuroscience and Neuropharmacology Research (21 papers). Paulo Correia‐de‐Sá collaborates with scholars based in Portugal, Brazil and Canada. Paulo Correia‐de‐Sá's co-authors include Joaquim A. Ribeiro, M. Alexandrina Timóteo, Fátima Ferreirinha, M.T. Magalhães‐Cardoso, Laura Oliveira, Ana M. Sebastião, Isabel Silva, José Bernardo Noronha‐Matos, Rodrigo A. Cunha and Maria Adelina Costa and has published in prestigious journals such as Journal of Biological Chemistry, PLoS ONE and The Journal of Physiology.

In The Last Decade

Paulo Correia‐de‐Sá

123 papers receiving 2.8k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Paulo Correia‐de‐Sá Portugal 31 1.3k 1.1k 858 363 326 126 2.8k
Gillian E. Knight United Kingdom 22 2.7k 2.0× 1.1k 1.1× 556 0.6× 935 2.6× 286 0.9× 62 3.9k
Philip M. Dunn United Kingdom 27 1.7k 1.3× 1.5k 1.4× 912 1.1× 1.1k 2.9× 465 1.4× 58 3.5k
Philippe Bodin United Kingdom 22 1.4k 1.1× 781 0.7× 356 0.4× 721 2.0× 318 1.0× 39 2.8k
Francesco Caciagli Italy 35 1.5k 1.2× 1.3k 1.2× 1.5k 1.7× 224 0.6× 52 0.2× 101 3.7k
Peter Sneddon United Kingdom 26 1.2k 0.9× 1.2k 1.1× 878 1.0× 587 1.6× 148 0.5× 40 2.5k
Debra A. Cockayne United States 29 2.1k 1.6× 1.2k 1.1× 870 1.0× 1.5k 4.0× 1.3k 4.1× 45 5.4k
Jonathan M. Beckel United States 22 372 0.3× 633 0.6× 208 0.2× 385 1.1× 723 2.2× 65 2.0k
Geoffrey Burnstock United Kingdom 13 1.7k 1.3× 680 0.6× 377 0.4× 602 1.7× 76 0.2× 21 2.5k
G Burnstock United Kingdom 19 1.2k 0.9× 624 0.6× 447 0.5× 608 1.7× 53 0.2× 40 2.0k
Ping K. Yip United Kingdom 34 345 0.3× 1.1k 1.0× 1.7k 2.0× 290 0.8× 112 0.3× 78 4.1k

Countries citing papers authored by Paulo Correia‐de‐Sá

Since Specialization
Citations

This map shows the geographic impact of Paulo Correia‐de‐Sá's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Paulo Correia‐de‐Sá with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Paulo Correia‐de‐Sá more than expected).

Fields of papers citing papers by Paulo Correia‐de‐Sá

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Paulo Correia‐de‐Sá. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Paulo Correia‐de‐Sá. The network helps show where Paulo Correia‐de‐Sá may publish in the future.

Co-authorship network of co-authors of Paulo Correia‐de‐Sá

This figure shows the co-authorship network connecting the top 25 collaborators of Paulo Correia‐de‐Sá. A scholar is included among the top collaborators of Paulo Correia‐de‐Sá based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Paulo Correia‐de‐Sá. Paulo Correia‐de‐Sá is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Pereira, Susana P., et al.. (2025). Anti-Hyperalgesic Effect of Isopulegol Involves GABA and NMDA Receptors in a Paclitaxel-Induced Neuropathic Pain Model. Pharmaceuticals. 18(2). 256–256. 1 indexed citations
2.
Oliveira, António, et al.. (2025). HIF-1α Stabilization Hampers the Chondrogenic Differentiation of Aged Bone Marrow Mesenchymal Stem Cells by Adenosine A 2A /A 2B Receptors Imbalance. ACS Pharmacology & Translational Science. 8(7). 2075–2092. 1 indexed citations
3.
Mocumbi, Ana Olga, Viviane Tiemi Hotta, Gene Bukhman, et al.. (2025). Endomyocardial fibrosis: recent advances and future therapeutic targets. Nature Reviews Cardiology. 22(8). 564–576.
4.
Rodrigues, Tânia Maria Barreto, et al.. (2023). Blockage of the adenosine A2B receptor prevents cardiac fibroblasts overgrowth in rats with pulmonary arterial hypertension. Purinergic Signalling. 20(2). 163–179. 3 indexed citations
5.
Lepiarczyk, Ewa, Łukasz Paukszto, Elżbieta Łopieńska–Biernat, et al.. (2023). The Carcinogenic Potential of Bisphenol A in the Liver Based on Transcriptomic Studies. Cancers. 15(20). 5014–5014. 5 indexed citations
6.
Rei, Nádia, et al.. (2023). Adenosinergic System and BDNF Signaling Changes as a Cross-Sectional Feature of RTT: Characterization of Mecp2 Heterozygous Mouse Females. International Journal of Molecular Sciences. 24(22). 16249–16249. 2 indexed citations
7.
Bessa, Cláudia, Joana B. Loureiro, Vera M. S. Isca, et al.. (2023). Counteracting Colon Cancer by Inhibiting Mitochondrial Respiration and Glycolysis with a Selective PKCδ Activator. International Journal of Molecular Sciences. 24(6). 5710–5710. 6 indexed citations
8.
Correia‐de‐Sá, Paulo, et al.. (2020). VIPergic neuroprotection in epileptogenesis: challenges and opportunities. Pharmacological Research. 164. 105356–105356. 10 indexed citations
9.
Pinheiro-Costa, João, Paulo Correia‐de‐Sá, Luís Torrão, et al.. (2020). Increased choroidal thickness is not a disease progression marker in keratoconus. Scientific Reports. 10(1). 19938–19938. 16 indexed citations
10.
Silva, Isabel, Ana Filipa Costa, Sílvia Moreira, et al.. (2017). Inhibition of cholinergic neurotransmission by β 3 -adrenoceptors depends on adenosine release and A 1 -receptor activation in human and rat urinary bladders. American Journal of Physiology-Renal Physiology. 313(2). F388–F403. 33 indexed citations
11.
Piovesan, Angela Regina, Dênis Reis de Assis, Lúcia Vinadé, et al.. (2017). Methylprednisolone as a memory enhancer in rats: Effects on aversive memory, long-term potentiation and calcium influx. Brain Research. 1670. 44–51. 6 indexed citations
12.
Noronha‐Matos, José Bernardo & Paulo Correia‐de‐Sá. (2016). Mesenchymal Stem Cells Ageing: Targeting the “Purinome” to Promote Osteogenic Differentiation and Bone Repair. Journal of Cellular Physiology. 231(9). 1852–1861. 41 indexed citations
13.
Oliveira, Laura, Alexandra Correia, Ana Costa, et al.. (2015). Deficits in Endogenous Adenosine Formation by Ecto‐5′‐Nucleotidase/CD73 Impair Neuromuscular Transmission and Immune Competence in Experimental Autoimmune Myasthenia Gravis. Mediators of Inflammation. 2015(1). 460610–460610. 21 indexed citations
14.
Silva‐Ramos, Miguel, et al.. (2015). Impairment of ATP hydrolysis decreases adenosine A1 receptor tonus favoring cholinergic nerve hyperactivity in the obstructed human urinary bladder. Purinergic Signalling. 11(4). 595–606. 19 indexed citations
15.
Silva‐Ramos, Miguel, Isabel Silva, M. Alexandrina Timóteo, et al.. (2012). UDP-SENSITIVE P2Y6 RECEPTORS PLAY A DUAL ROLE IN THE HUMAN URINARY BLADDER INDIRECTLY VIA THE RELEASE OF ATP FROM UROTHELIUM. Neurourology and Urodynamics. 4 indexed citations
16.
Noronha‐Matos, José Bernardo, Tiago Morais, Diogo Trigo, et al.. (2011). Tetanic failure due to decreased endogenous adenosine A2A tonus operating neuronal Cav1 (L-type) influx in Myasthenia gravis. Journal of Neurochemistry. 117(5). 797–811. 30 indexed citations
17.
Correia‐de‐Sá, Paulo, et al.. (2011). Presynaptic muscarinic and adenosine receptors are involved in 2 Hz-induced train-of-four fade caused by antinicotinic neuromuscular relaxants in the rat. Clinical and Experimental Pharmacology and Physiology. 38(11). 764–770. 9 indexed citations
18.
Correia‐de‐Sá, Paulo, et al.. (2009). Apamin reduces neuromuscular transmission by activating inhibitory muscarinic M2 receptors on motor nerve terminals. European Journal of Pharmacology. 626(2-3). 239–243. 7 indexed citations
19.
Charrua, Ana, Carlos Reguenga, Miguel Cordeiro, et al.. (2009). Functional Transient Receptor Potential Vanilloid 1 is Expressed in Human Urothelial Cells. The Journal of Urology. 182(6). 2944–2950. 52 indexed citations
20.
Oliveira, Laura, M. Alexandrina Timóteo, & Paulo Correia‐de‐Sá. (2002). Modulation by adenosine of both muscarinic M1‐facilitation and M2‐inhibition of [3H]‐acetylcholine release from the rat motor nerve terminals. European Journal of Neuroscience. 15(11). 1728–1736. 61 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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