Pascale Bomont

5.1k total citations
26 papers, 1.3k citations indexed

About

Pascale Bomont is a scholar working on Cell Biology, Cellular and Molecular Neuroscience and Molecular Biology. According to data from OpenAlex, Pascale Bomont has authored 26 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Cell Biology, 14 papers in Cellular and Molecular Neuroscience and 11 papers in Molecular Biology. Recurrent topics in Pascale Bomont's work include Skin and Cellular Biology Research (12 papers), Hereditary Neurological Disorders (11 papers) and Neurological diseases and metabolism (7 papers). Pascale Bomont is often cited by papers focused on Skin and Cellular Biology Research (12 papers), Hereditary Neurological Disorders (11 papers) and Neurological diseases and metabolism (7 papers). Pascale Bomont collaborates with scholars based in France, United States and Japan. Pascale Bomont's co-authors include Don W. Cleveland, Rudolf Korinthenberg, Mériem Tazir, Haluk Topaloğlu, P. Landrieu, Samir Belal, L Cavalier, Fayçal Hentati, Ercan Demir and Beyhan Tüysüz and has published in prestigious journals such as Journal of Clinical Investigation, Nature Communications and Nature Genetics.

In The Last Decade

Pascale Bomont

26 papers receiving 1.2k citations

Peers

Pascale Bomont

CMN

NEURO

Hong Wei Yang Japan

Jennifer A. Barrie United Kingdom

Jens Reimann Germany

Maire Leyne United States

Nicholas A. DiProspero United States

Éléonore Eymard-Pierre France

Sofia A. Oliveira Portugal

Alice B. Schindler United States

Hossein Darvish Iran

V. M.‐Y. Lee United States

Hong Wei Yang Japan

Pascale Bomont

446 ×0.7

329 ×0.6

331 ×0.7

CMN

214 ×0.9

NEURO

155 ×0.7

NEURO

Citations per year, relative to Pascale Bomont Pascale Bomont (= 1×) peers Hong Wei Yang

Countries citing papers authored by Pascale Bomont

Since Specialization

Citations

This map shows the geographic impact of Pascale Bomont's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Pascale Bomont with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Pascale Bomont more than expected).

Fields of papers citing papers by Pascale Bomont

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Pascale Bomont. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Pascale Bomont. The network helps show where Pascale Bomont may publish in the future.

Co-authorship network of co-authors of Pascale Bomont

This figure shows the co-authorship network connecting the top 25 collaborators of Pascale Bomont. A scholar is included among the top collaborators of Pascale Bomont based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Pascale Bomont. Pascale Bomont is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Bomont, Pascale, et al.. (2024). Neuronal Autophagy: Regulations and Implications in Health and Disease. Cells. 13(1). 103–103. 21 indexed citations

Asperen, Jessy V. van, et al.. (2024). Neurofilaments: Novel findings and future challenges. Current Opinion in Cell Biology. 87. 102326–102326. 7 indexed citations

Baudot, Anaı̈s, et al.. (2023). A multilevel screening pipeline in zebrafish identifies therapeutic drugs for GAN. EMBO Molecular Medicine. 15(7). e16267–e16267. 3 indexed citations

Bomont, Pascale, et al.. (2023). Neurofilaments in health and Charcot-Marie-Tooth disease. Frontiers in Cell and Developmental Biology. 11. 1275155–1275155. 11 indexed citations

Bomont, Pascale, et al.. (2022). Development of a high-throughput tailored imaging method in zebrafish to understand and treat neuromuscular diseases. Frontiers in Molecular Neuroscience. 15. 956582–956582. 4 indexed citations

Bomont, Pascale. (2021). The dazzling rise of neurofilaments: Physiological functions and roles as biomarkers. Current Opinion in Cell Biology. 68. 181–191. 48 indexed citations

Bomont, Pascale, et al.. (2020). E3 Ubiquitin Ligases in Neurological Diseases: Focus on Gigaxonin and Autophagy. Frontiers in Physiology. 11. 1022–1022. 23 indexed citations

Scrivo, Aurora, Patrice Codogno, & Pascale Bomont. (2019). Gigaxonin E3 ligase governs ATG16L1 turnover to control autophagosome production. Nature Communications. 10(1). 780–780. 51 indexed citations

Yoshida, Sayaka, Yuya Araki, Takayasu Mori, et al.. (2018). Decreased KLHL3 expression is involved in the pathogenesis of pseudohypoaldosteronism type II caused by cullin 3 mutation in vivo. Clinical and Experimental Nephrology. 22(6). 1251–1257. 19 indexed citations

10.

Bomont, Pascale. (2015). Degradation of the Intermediate Filament Family by Gigaxonin. Methods in enzymology on CD-ROM/Methods in enzymology. 569. 215–231. 22 indexed citations

11.

Kuhlenbäumer, Gregor, Vincent Timmerman, & Pascale Bomont. (2014). Giant Axonal Neuropathy. 5 indexed citations

12.

Morrogh, Deborah, Nancy L. Kuntz, C. Halbert, et al.. (2014). The instability of the BTB-KELCH protein Gigaxonin causes Giant Axonal Neuropathy and constitutes a new penetrant and specific diagnostic test. Acta Neuropathologica Communications. 2(1). 47–47. 22 indexed citations

13.

Mahammad, Saleemulla, S. N. Prasanna Murthy, Alessandro Didonna, et al.. (2013). Giant axonal neuropathy–associated gigaxonin mutations impair intermediate filament protein degradation. Journal of Clinical Investigation. 123(5). 1964–1975. 94 indexed citations

14.

Chauvin, J P, et al.. (2011). Sensory-motor deficits and neurofilament disorganization in gigaxonin-null mice. Molecular Neurodegeneration. 6(1). 25–25. 37 indexed citations

15.

Cleveland, Don W., Koji Yamanaka, & Pascale Bomont. (2009). Gigaxonin controls vimentin organization through a tubulin chaperone-independent pathway. Human Molecular Genetics. 18(8). 1384–1394. 40 indexed citations

16.

Bomont, Pascale, Paul S. Maddox, Jagesh V. Shah, Arshad Desai, & Don W. Cleveland. (2005). Unstable microtubule capture at kinetochores depleted of the centromere‐associated protein CENP‐F. The EMBO Journal. 24(22). 3927–3939. 98 indexed citations

17.

Bomont, Pascale, Christine Ioos, Cengiz Yalçınkaya, et al.. (2003). Identification of seven novel mutations in theGAN gene. Human Mutation. 21(4). 446–446. 42 indexed citations

18.

Bomont, Pascale. (2003). Intermediate filament aggregation in fibroblasts of giant axonal neuropathy patients is aggravated in non dividing cells and by microtubule destabilization. Human Molecular Genetics. 12(8). 813–822. 41 indexed citations

19.

Barbot, Clara, Nobutada Tachi, Pascale Bomont, et al.. (2002). Autosomal recessive ataxias: a new gene - aprataxin - responsible for ataxia-ocular apraxia 1, and a new locus on 9q34. European Journal of Human Genetics. 10. 272–273. 3 indexed citations

20.

Tazir, Mériem, Jean-Michel Vallat, Pascale Bomont, et al.. (2002). Genetic heterogeneity in giant axonal neuropathy: an Algerian family not linked to chromosome 16q24.1. Neuromuscular Disorders. 12(9). 849–852. 21 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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