Otoya Ueda

4.2k total citations
43 papers, 2.3k citations indexed

About

Otoya Ueda is a scholar working on Molecular Biology, Genetics and Public Health, Environmental and Occupational Health. According to data from OpenAlex, Otoya Ueda has authored 43 papers receiving a total of 2.3k indexed citations (citations by other indexed papers that have themselves been cited), including 28 papers in Molecular Biology, 13 papers in Genetics and 7 papers in Public Health, Environmental and Occupational Health. Recurrent topics in Otoya Ueda's work include Pluripotent Stem Cells Research (10 papers), Reproductive Biology and Fertility (7 papers) and CRISPR and Genetic Engineering (7 papers). Otoya Ueda is often cited by papers focused on Pluripotent Stem Cells Research (10 papers), Reproductive Biology and Fertility (7 papers) and CRISPR and Genetic Engineering (7 papers). Otoya Ueda collaborates with scholars based in Japan, United States and Chile. Otoya Ueda's co-authors include Hiroshi Suzuki, Nobuo Kamada, Kou‐ichi Jishage, Hiroshi Suzuki, Kou-ichi Jishage, Miho Watanabe, Takamitsu Iwata, Toshio Sofuni, Masami Yamada and Masaya Katoh and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Medicine.

In The Last Decade

Otoya Ueda

43 papers receiving 2.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Otoya Ueda Japan 24 1.2k 390 381 363 340 43 2.3k
Shuh Narumiya Japan 18 1.5k 1.2× 659 1.7× 533 1.4× 313 0.9× 270 0.8× 21 3.5k
Yoichi Mizukami Japan 28 1.5k 1.2× 411 1.1× 327 0.9× 223 0.6× 177 0.5× 108 3.0k
Kazuhiro Oka United States 34 1.9k 1.6× 770 2.0× 391 1.0× 272 0.7× 492 1.4× 74 3.8k
Maria Sjöberg Sweden 12 1.1k 0.9× 989 2.5× 144 0.4× 337 0.9× 153 0.5× 14 2.2k
Anna Greka United States 25 2.3k 1.9× 278 0.7× 379 1.0× 215 0.6× 199 0.6× 48 4.3k
Jianxin Sun United States 33 1.8k 1.5× 176 0.5× 486 1.3× 186 0.5× 680 2.0× 75 3.5k
Fukushi Kambe Japan 26 1.1k 0.9× 261 0.7× 245 0.6× 234 0.6× 270 0.8× 75 2.0k
Alice Lin United States 8 2.3k 1.9× 359 0.9× 613 1.6× 271 0.7× 290 0.9× 15 3.6k
Karen M. Neilson United States 17 1.1k 0.9× 814 2.1× 184 0.5× 221 0.6× 342 1.0× 30 2.6k
Hitoshi Matsushime Japan 20 2.8k 2.3× 408 1.0× 323 0.8× 1.9k 5.2× 294 0.9× 27 4.6k

Countries citing papers authored by Otoya Ueda

Since Specialization
Citations

This map shows the geographic impact of Otoya Ueda's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Otoya Ueda with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Otoya Ueda more than expected).

Fields of papers citing papers by Otoya Ueda

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Otoya Ueda. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Otoya Ueda. The network helps show where Otoya Ueda may publish in the future.

Co-authorship network of co-authors of Otoya Ueda

This figure shows the co-authorship network connecting the top 25 collaborators of Otoya Ueda. A scholar is included among the top collaborators of Otoya Ueda based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Otoya Ueda. Otoya Ueda is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
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Ohtomo, Shuichi, et al.. (2022). TNF-α induces Claudin-1 expression in renal tubules in Alport mice. PLoS ONE. 17(3). e0265081–e0265081. 10 indexed citations
3.
Tominaga, Tatsuya, Hideharu Abe, Naoshi Fukushima, et al.. (2018). An adjustment in BMP4 function represents a treatment for diabetic nephropathy and podocyte injury. Scientific Reports. 8(1). 13011–13011. 16 indexed citations
4.
Fukuzawa, Taku, Masanori Fukazawa, Otoya Ueda, et al.. (2013). SGLT5 Reabsorbs Fructose in the Kidney but Its Deficiency Paradoxically Exacerbates Hepatic Steatosis Induced by Fructose. PLoS ONE. 8(2). e56681–e56681. 45 indexed citations
5.
Tominaga, Tatsuya, Hideharu Abe, Otoya Ueda, et al.. (2011). Activation of Bone Morphogenetic Protein 4 Signaling Leads to Glomerulosclerosis That Mimics Diabetic Nephropathy. Journal of Biological Chemistry. 286(22). 20109–20116. 46 indexed citations
6.
Kishi, Seiji, Hideharu Abe, Haruhiko Akiyama, et al.. (2011). SOX9 Protein Induces a Chondrogenic Phenotype of Mesangial Cells and Contributes to Advanced Diabetic Nephropathy. Journal of Biological Chemistry. 286(37). 32162–32169. 28 indexed citations
7.
Sohara, Eisei, Otoya Ueda, Takanori Tachibe, et al.. (2006). Morphologic and functional analysis of sperm and testes in Aquaporin 7 knockout mice. Fertility and Sterility. 87(3). 671–676. 50 indexed citations
8.
Tachibe, Takanori, et al.. (2006). Fertility of mice receiving vitrified adult mouse ovaries. Reproduction. 131(4). 681–687. 22 indexed citations
9.
Katsume, Asao, Hiroyuki Saito, Yoshiki Yamada, et al.. (2002). ANTI-INTERLEUKIN 6 (IL-6) RECEPTOR ANTIBODY SUPPRESSES CASTLEMAN'S DISEASE LIKE SYMPTOMS EMERGED IN IL-6 TRANSGENIC MICE. Cytokine. 20(6). 304–311. 112 indexed citations
10.
11.
Jishage, Kou-ichi, Makoto Arita, Takamitsu Iwata, et al.. (2001). α-Tocopherol Transfer Protein Is Important for the Normal Development of Placental Labyrinthine Trophoblasts in Mice. Journal of Biological Chemistry. 276(3). 1669–1672. 150 indexed citations
12.
Masumura, Kenichi, M. Matsui, Masaya Katoh, et al.. (1999). Spectra ofgpt mutations in ethylnitrosourea-treated and untreated transgenic mice. Environmental and Molecular Mutagenesis. 34(1). 1–8. 63 indexed citations
13.
Chui, Dehua, Hiroshi Tanahashi, Kazuharu Ozawa, et al.. (1999). Transgenic mice with Alzheimer presenilin 1 mutations show accelerated neurodegeneration without amyloid plaque formation. Nature Medicine. 5(5). 560–564. 313 indexed citations
14.
Nohmi, T., Makoto Suzuki, Kenichi Masumura, et al.. (1999). Spi? selection: An efficient method to detect ?-ray-induced deletions in transgenic mice. Environmental and Molecular Mutagenesis. 34(1). 9–15. 55 indexed citations
15.
Okada, Naoki, Hajime MIYAMOTO, Tatsunobu Yoshioka, et al.. (1997). Cytomedical therapy for IgG1 plasmacytosis in human interleukin-6 transgenic mice using hybridoma cells microencapsulated in alginate-poly(l) lysine-alginate membrane. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1360(1). 53–63. 23 indexed citations
16.
Nakagata, Naomi, Masanori Okamoto, Otoya Ueda, & Hiroshi Suzuki. (1997). Positive Effect of Partial Zona-Pellucida Dissection on the in Vitro Fertilizing Capacity of Cryopreserved C57BL/6J Transgenic Mouse Spermatozoa of Low Motility. Biology of Reproduction. 57(5). 1050–1055. 90 indexed citations
17.
Yoshioka, Tatsunobu, Naoki Okada, Hajime MIYAMOTO, et al.. (1997). Development of novel drug delivery system of bioactive molecules from "cytomedicine" using hybridoma cells entrapped in alginate-poly(L) lysine-alginate microcapsules.. Drug Delivery System. 12(2). 107–114. 1 indexed citations
18.
Nohmi, T., Masaya Katoh, Hiroshi Suzuki, et al.. (1996). Other transgenic mutation assays: A new transgenic mouse mutagenesis test system using Spi− and 6-thioguanine selections. Environmental and Molecular Mutagenesis. 28(4). 465–470. 183 indexed citations
19.
Terauchi, Yasuo, Hiroshi Sakura, Kazuki Yasuda, et al.. (1995). Pancreatic β-Cell-specific Targeted Disruption of Glucokinase Gene. Journal of Biological Chemistry. 270(51). 30253–30256. 180 indexed citations
20.
Suzuki, Hiroshi, et al.. (1994). Improved Embryo Transfer into the Oviduct by Local Application of a Vasoconstrictor in Mice. Journal of Mammalian Ova Research. 11(1). 49–53. 13 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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