Oliver Hölsken

529 total citations
8 papers, 142 citations indexed

About

Oliver Hölsken is a scholar working on Immunology, Oncology and Surgery. According to data from OpenAlex, Oliver Hölsken has authored 8 papers receiving a total of 142 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Immunology, 3 papers in Oncology and 1 paper in Surgery. Recurrent topics in Oliver Hölsken's work include Immune Cell Function and Interaction (5 papers), CAR-T cell therapy research (3 papers) and IL-33, ST2, and ILC Pathways (2 papers). Oliver Hölsken is often cited by papers focused on Immune Cell Function and Interaction (5 papers), CAR-T cell therapy research (3 papers) and IL-33, ST2, and ILC Pathways (2 papers). Oliver Hölsken collaborates with scholars based in Germany, Russia and United States. Oliver Hölsken's co-authors include Matthias Miller, Adelheid Cerwenka, Chiara Romagnani, Jing Ni, Quirin Hammer, Merlin V. Luetke-Eversloh, Andreas Diefenbach, Caroline Tizian, Frederik Heinrich and Mir‐Farzin Mashreghi and has published in prestigious journals such as European Journal of Immunology, Cell Reports and eLife.

In The Last Decade

Oliver Hölsken

8 papers receiving 140 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Oliver Hölsken Germany	5	116	47	33	16	9	8	142
Anna Bremser Germany	5	83 0.7×	10 0.2×	20 0.6×	40 2.5×	3 0.3×	6	123
Anthony M. Joudi United States	4	47 0.4×	16 0.3×	25 0.8×	16 1.0×		9	104
Leila Godfrey United Kingdom	5	63 0.5×	22 0.5×	7 0.2×	23 1.4×	2 0.2×	7	107
Kirstin Hoff Germany	3	100 0.9×	23 0.5×	10 0.3×	36 2.3×	1 0.1×	4	141
Mara Cenerenti Switzerland	2	67 0.6×	44 0.9×	6 0.2×	22 1.4×	1 0.1×	2	103
Margaux Saillard Switzerland	4	77 0.7×	58 1.2×	6 0.2×	32 2.0×	1 0.1×	4	124
Ruddy Montandon France	5	85 0.7×	13 0.3×	5 0.2×	16 1.0×	2 0.2×	7	112
Michael Chait United States	3	52 0.4×	27 0.6×	13 0.4×	7 0.4×		3	86
Ramez Wahib Germany	2	71 0.6×	20 0.4×	12 0.4×	26 1.6×		2	92

Countries citing papers authored by Oliver Hölsken

Since Specialization

Citations

This map shows the geographic impact of Oliver Hölsken's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Oliver Hölsken with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Oliver Hölsken more than expected).

Fields of papers citing papers by Oliver Hölsken

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Oliver Hölsken. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Oliver Hölsken. The network helps show where Oliver Hölsken may publish in the future.

Co-authorship network of co-authors of Oliver Hölsken

This figure shows the co-authorship network connecting the top 25 collaborators of Oliver Hölsken. A scholar is included among the top collaborators of Oliver Hölsken based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Oliver Hölsken. Oliver Hölsken is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Hölsken, Oliver, Jens Martens‐Lobenhoffer, Stefanie M. Bode‐Böger, et al.. (2024). First Clinical Application of Aztreonam–Avibactam in Treating Carbapenem-Resistant Enterobacterales: Insights from Therapeutic Drug Monitoring and Pharmacokinetic Simulations. Journal of Personalized Medicine. 14(12). 1135–1135. 1 indexed citations

2.

Hölsken, Oliver, Claudia U. Duerr, Nora Branzk, et al.. (2023). Antibiotic use during pregnancy is linked to offspring gut microbial dysbiosis, barrier disruption, and altered immunity along the gut–lung axis. European Journal of Immunology. 53(10). e2350394–e2350394. 6 indexed citations

3.

Steffen, Johannes, Ute Bank, Aindrila Biswas, et al.. (2022). Type 1 innate lymphoid cells regulate the onset of Toxoplasma gondii-induced neuroinflammation. Cell Reports. 38(13). 110564–110564. 22 indexed citations

4.

Tizian, Caroline, Annette Lahmann, Oliver Hölsken, et al.. (2020). c-Maf restrains T-bet-driven programming of CCR6-negative group 3 innate lymphoid cells. eLife. 9. 28 indexed citations

5.

Tizian, Caroline, Annette Lahmann, Oliver Hölsken, et al.. (2020). Correction: c-Maf restrains T-bet-driven programming of CCR6-negative group 3 innate lymphoid cells. eLife. 9. 3 indexed citations

6.

Ni, Jing, Oliver Hölsken, Matthias Miller, et al.. (2016). Adoptively transferred natural killer cells maintain long-term antitumor activity by epigenetic imprinting and CD4⁺T cell help. OncoImmunology. 5(9). e1219009–e1219009. 59 indexed citations

7.

Hölsken, Oliver, Matthias Miller, & Adelheid Cerwenka. (2015). Die Nutzung natürlicher Killerzellen für die Therapie des Melanoms. JDDG Journal der Deutschen Dermatologischen Gesellschaft. 13(1). 23–29. 4 indexed citations

8.

Hölsken, Oliver, Matthias Miller, & Adelheid Cerwenka. (2015). Exploiting natural killer cells for therapy of melanoma. JDDG Journal der Deutschen Dermatologischen Gesellschaft. 13(1). 23–28. 19 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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