Olga Antsiferova

680 total citations
8 papers, 532 citations indexed

About

Olga Antsiferova is a scholar working on Immunology, Oncology and Pathology and Forensic Medicine. According to data from OpenAlex, Olga Antsiferova has authored 8 papers receiving a total of 532 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Immunology, 5 papers in Oncology and 3 papers in Pathology and Forensic Medicine. Recurrent topics in Olga Antsiferova's work include Immune Cell Function and Interaction (6 papers), Viral-associated cancers and disorders (4 papers) and Lymphoma Diagnosis and Treatment (3 papers). Olga Antsiferova is often cited by papers focused on Immune Cell Function and Interaction (6 papers), Viral-associated cancers and disorders (4 papers) and Lymphoma Diagnosis and Treatment (3 papers). Olga Antsiferova collaborates with scholars based in Switzerland, Germany and Italy. Olga Antsiferova's co-authors include Christian Münz, David Aebischer, Maximilian Nitschké, Maria Iolyeva, Cornelia Halin, Benjamin Vigl, Obinna Chijioke, Henri‐Jacques Delecluse, Vanessa Landtwing and Carol S. Leung and has published in prestigious journals such as Blood, The Journal of Immunology and European Journal of Immunology.

In The Last Decade

Olga Antsiferova

7 papers receiving 522 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Olga Antsiferova Switzerland 6 352 312 127 96 70 8 532
Ana Raykova Switzerland 11 350 1.0× 266 0.9× 161 1.3× 166 1.7× 51 0.7× 12 524
Laura L. Quinn United Kingdom 9 351 1.0× 274 0.9× 169 1.3× 95 1.0× 57 0.8× 11 466
Chiharu Kanegane Japan 9 375 1.1× 316 1.0× 51 0.4× 76 0.8× 100 1.4× 14 559
Katharina Reinhard Germany 10 164 0.5× 651 2.1× 102 0.8× 100 1.0× 91 1.3× 12 852
Manfred Uerlich Germany 13 111 0.3× 277 0.9× 120 0.9× 71 0.7× 60 0.9× 33 544
Laëtitia Le Texier Australia 14 133 0.4× 335 1.1× 75 0.6× 28 0.3× 95 1.4× 30 586
Maria Teresa Rodia Italy 9 158 0.4× 242 0.8× 69 0.5× 31 0.3× 103 1.5× 12 451
Magdalena Hagn Australia 11 182 0.5× 560 1.8× 57 0.4× 46 0.5× 94 1.3× 13 697
Jorge Martínez‐Escribano Spain 15 160 0.5× 190 0.6× 41 0.3× 36 0.4× 53 0.8× 45 502
Yasmin Hazrat United States 3 376 1.1× 331 1.1× 79 0.6× 186 1.9× 39 0.6× 6 553

Countries citing papers authored by Olga Antsiferova

Since Specialization
Citations

This map shows the geographic impact of Olga Antsiferova's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Olga Antsiferova with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Olga Antsiferova more than expected).

Fields of papers citing papers by Olga Antsiferova

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Olga Antsiferova. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Olga Antsiferova. The network helps show where Olga Antsiferova may publish in the future.

Co-authorship network of co-authors of Olga Antsiferova

This figure shows the co-authorship network connecting the top 25 collaborators of Olga Antsiferova. A scholar is included among the top collaborators of Olga Antsiferova based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Olga Antsiferova. Olga Antsiferova is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
2.
Chatterjee, Bithi, Yun Deng, Angelika Holler, et al.. (2019). CD8+ T cells retain protective functions despite sustained inhibitory receptor expression during Epstein-Barr virus infection in vivo. PLoS Pathogens. 15(5). e1007748–e1007748. 56 indexed citations
3.
Chijioke, Obinna, Anne Müller, Regina Feederle, et al.. (2015). Human Natural Killer Cells Prevent Infectious Mononucleosis Features by Targeting Lytic Epstein-Barr Virus Infection. Cell Reports. 12(5). 901–901.
4.
Antsiferova, Olga, Anne Müller, Patrick C. Rämer, et al.. (2014). Adoptive Transfer of EBV Specific CD8+ T Cell Clones Can Transiently Control EBV Infection in Humanized Mice. PLoS Pathogens. 10(8). e1004333–e1004333. 55 indexed citations
5.
Chijioke, Obinna, Anne Müller, Regina Feederle, et al.. (2013). Human Natural Killer Cells Prevent Infectious Mononucleosis Features by Targeting Lytic Epstein-Barr Virus Infection. Cell Reports. 5(6). 1489–1498. 183 indexed citations
6.
Bonaccorsi, Irene, Barbara Morandi, Olga Antsiferova, et al.. (2013). Membrane Transfer from Tumor Cells Overcomes Deficient Phagocytic Ability of Plasmacytoid Dendritic Cells for the Acquisition and Presentation of Tumor Antigens. The Journal of Immunology. 192(2). 824–832. 34 indexed citations
7.
Leung, Carol S., Obinna Chijioke, Cornelia Gujer, et al.. (2013). Infectious diseases in humanized mice. European Journal of Immunology. 43(9). 2246–2254. 38 indexed citations
8.
Vigl, Benjamin, David Aebischer, Maximilian Nitschké, et al.. (2011). Tissue inflammation modulates gene expression of lymphatic endothelial cells and dendritic cell migration in a stimulus-dependent manner. Blood. 118(1). 205–215. 164 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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