Noritaka Koga

413 total citations
10 papers, 353 citations indexed

About

Noritaka Koga is a scholar working on Immunology, Molecular Biology and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Noritaka Koga has authored 10 papers receiving a total of 353 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Immunology, 3 papers in Molecular Biology and 3 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Noritaka Koga's work include T-cell and B-cell Immunology (4 papers), Viral Infections and Immunology Research (3 papers) and Peroxisome Proliferator-Activated Receptors (3 papers). Noritaka Koga is often cited by papers focused on T-cell and B-cell Immunology (4 papers), Viral Infections and Immunology Research (3 papers) and Peroxisome Proliferator-Activated Receptors (3 papers). Noritaka Koga collaborates with scholars based in Japan. Noritaka Koga's co-authors include Hisanori Kosuge, Mitsuaki Isobe, Jun‐ichi Suzuki, Yasuhiro Maejima, Go Haraguchi, Hideki Futamatsu, Toshimitsu Uede, Manabu Inobe, Kenzo Hirao and Hiroshi Ito and has published in prestigious journals such as Circulation, Circulation Research and Arteriosclerosis Thrombosis and Vascular Biology.

In The Last Decade

Noritaka Koga

10 papers receiving 348 citations

Peers

Noritaka Koga
Nathan S. Ihrcke United States
Moh Moh Lian United States
Jacob Amersfoort Netherlands
Noritaka Koga
Citations per year, relative to Noritaka Koga Noritaka Koga (= 1×) peers Abdelouahab Aı̈touche

Countries citing papers authored by Noritaka Koga

Since Specialization
Citations

This map shows the geographic impact of Noritaka Koga's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Noritaka Koga with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Noritaka Koga more than expected).

Fields of papers citing papers by Noritaka Koga

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Noritaka Koga. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Noritaka Koga. The network helps show where Noritaka Koga may publish in the future.

Co-authorship network of co-authors of Noritaka Koga

This figure shows the co-authorship network connecting the top 25 collaborators of Noritaka Koga. A scholar is included among the top collaborators of Noritaka Koga based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Noritaka Koga. Noritaka Koga is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

10 of 10 papers shown
1.
Suzuki, Jun‐ichi, Noritaka Koga, Hisanori Kosuge, et al.. (2007). Pitavastatin Suppresses Acute and Chronic Rejection in Murine Cardiac Allografts. Transplantation. 83(8). 1093–1097. 13 indexed citations
2.
Futamatsu, Hideki, Jun‐ichi Suzuki, Noritaka Koga, et al.. (2006). A CCR1 antagonist prevents the development of experimental autoimmune myocarditis in association with T cell inactivation. Journal of Molecular and Cellular Cardiology. 40(6). 853–861. 17 indexed citations
3.
Haraguchi, Go, Jun‐ichi Suzuki, Hisanori Kosuge, et al.. (2006). A new RXR agonist, HX630, suppresses intimal hyperplasia in a mouse blood flow cessation model. Journal of Molecular and Cellular Cardiology. 41(5). 885–892. 11 indexed citations
4.
Kosuge, Hisanori, Jun‐ichi Suzuki, Go Haraguchi, et al.. (2006). Critical Role of Inducible Costimulator Signaling in the Development of Arteriosclerosis. Arteriosclerosis Thrombosis and Vascular Biology. 26(12). 2660–2665. 15 indexed citations
5.
Kosuge, Hisanori, Go Haraguchi, Noritaka Koga, et al.. (2006). Pioglitazone Prevents Acute and Chronic Cardiac Allograft Rejection. Circulation. 113(22). 2613–2622. 46 indexed citations
6.
Ogawa, Masahito, Jun‐ichi Suzuki, Noritaka Koga, Hisanori Kosuge, & Mitsuaki Isobe. (2005). A specific COX-2 inhibitor attenuates cell infiltration but does not prolong graft survival in murine cardiac transplantation. Transplantation Proceedings. 37(1). 121–122. 2 indexed citations
7.
Futamatsu, Hideki, Jun‐ichi Suzuki, Shinya Mizuno, et al.. (2005). Hepatocyte Growth Factor Ameliorates the Progression of Experimental Autoimmune Myocarditis. Circulation Research. 96(8). 823–830. 75 indexed citations
8.
Kosuge, Hisanori, Jun‐ichi Suzuki, Tsunekazu Kakuta, et al.. (2004). Attenuation of Graft Arterial Disease by Manipulation of the LIGHT Pathway. Arteriosclerosis Thrombosis and Vascular Biology. 24(8). 1409–1415. 26 indexed citations
9.
Koga, Noritaka, Jun‐ichi Suzuki, Hisanori Kosuge, et al.. (2004). Blockade of the Interaction Between PD-1 and PD-L1 Accelerates Graft Arterial Disease in Cardiac Allografts. Arteriosclerosis Thrombosis and Vascular Biology. 24(11). 2057–2062. 79 indexed citations
10.
Kosuge, Hisanori, Jun‐ichi Suzuki, Noritaka Koga, et al.. (2003). Induction of immunologic tolerance to cardiac allograft by simultaneous blockade of inducible co-stimulator and cytotoxic T-lymphocyte antigen 4 pathway1. Transplantation. 75(8). 1374–1380. 69 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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