Nigel Barrass

1.0k total citations
20 papers, 793 citations indexed

About

Nigel Barrass is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Nigel Barrass has authored 20 papers receiving a total of 793 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 8 papers in Oncology and 6 papers in Cancer Research. Recurrent topics in Nigel Barrass's work include Carcinogens and Genotoxicity Assessment (4 papers), Cancer-related Molecular Pathways (4 papers) and Drug Transport and Resistance Mechanisms (3 papers). Nigel Barrass is often cited by papers focused on Carcinogens and Genotoxicity Assessment (4 papers), Cancer-related Molecular Pathways (4 papers) and Drug Transport and Resistance Mechanisms (3 papers). Nigel Barrass collaborates with scholars based in United Kingdom, France and United States. Nigel Barrass's co-authors include Rose Ann Padua, G A Currie, John R. Foster, Robert W. Wilkinson, Rajesh Odedra, Stephen R. Wedge, Kirsten Mundt, Kevin M. Foote, Nicholas Keen and Andrew Mortlock and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Molecular and Cellular Biology.

In The Last Decade

Nigel Barrass

20 papers receiving 760 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Nigel Barrass United Kingdom	12	505	404	244	144	53	20	793
Jijiang Zhu United States	12	827 1.6×	586 1.5×	299 1.2×	341 2.4×	73 1.4×	14	1.2k
Murray J. Towle United States	17	440 0.9×	509 1.3×	162 0.7×	119 0.8×	95 1.8×	24	1.1k
Richard A. Woo Canada	10	674 1.3×	454 1.1×	165 0.7×	153 1.1×	62 1.2×	11	893
Minshu Yu United States	20	541 1.1×	516 1.3×	82 0.3×	155 1.1×	77 1.5×	37	959
Timothy Ackerson United States	8	1.3k 2.6×	296 0.7×	195 0.8×	224 1.6×	110 2.1×	8	1.4k
Wenqing Qi United States	20	668 1.3×	370 0.9×	224 0.9×	83 0.6×	126 2.4×	42	1.0k
Mike I. Walton United Kingdom	7	630 1.2×	283 0.7×	93 0.4×	118 0.8×	91 1.7×	9	856
Michele Goyette United States	12	715 1.4×	439 1.1×	79 0.3×	212 1.5×	46 0.9×	13	1.3k
N. Barry Elkind Hungary	13	572 1.1×	571 1.4×	129 0.5×	64 0.4×	81 1.5×	14	910
Richard E. Francis United Kingdom	8	752 1.5×	240 0.6×	150 0.6×	105 0.7×	52 1.0×	8	959

Countries citing papers authored by Nigel Barrass

Since Specialization

Citations

This map shows the geographic impact of Nigel Barrass's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nigel Barrass with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nigel Barrass more than expected).

Fields of papers citing papers by Nigel Barrass

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nigel Barrass. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nigel Barrass. The network helps show where Nigel Barrass may publish in the future.

Co-authorship network of co-authors of Nigel Barrass

This figure shows the co-authorship network connecting the top 25 collaborators of Nigel Barrass. A scholar is included among the top collaborators of Nigel Barrass based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nigel Barrass. Nigel Barrass is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Yu, Hong, Nigel Barrass, Eva M. Lenz, et al.. (2014). Metabolism by conjugation appears to confer resistance to paracetamol (acetaminophen) hepatotoxicity in the cynomolgus monkey. Xenobiotica. 45(3). 270–277. 11 indexed citations

Wilkinson, Robert W., Rajesh Odedra, Simon P. Heaton, et al.. (2007). AZD1152, a Selective Inhibitor of Aurora B Kinase, Inhibits Human Tumor Xenograft Growth by Inducing Apoptosis. Clinical Cancer Research. 13(12). 3682–3688. 278 indexed citations

Wilkinson, Robert W., Nicholas Keen, Rajesh Odedra, et al.. (2006). AZD1152: A highly potent and specific aurora kinase inhibitor.. Cancer Research. 66. 1333–1334. 6 indexed citations

Camidge, D. Ross, Dominic Smethurst, Jim Growcott, et al.. (2006). A first-in-man phase I tolerability and pharmacokinetic study of the cyclin-dependent kinase-inhibitor AZD5438 in healthy male volunteers. Cancer Chemotherapy and Pharmacology. 60(3). 391–398. 23 indexed citations

Double, John A., et al.. (2002). Toxicity testing in the development of anticancer drugs. The Lancet Oncology. 3(7). 438–442. 7 indexed citations

Barrass, Nigel, M. Stewart, Sarah Warburton, et al.. (1993). Cell proliferation in the liver and thyroid of C57Bl/10J mice after dietary administration of chlordane.. Environmental Health Perspectives. 101(suppl 5). 219–223. 15 indexed citations

Jones, H. B., et al.. (1993). Phenobarbital-induced hepatocellular proliferation: anti-bromodeoxyuridine and anti-proliferating cell nuclear antigen immunocytochemistry.. Journal of Histochemistry & Cytochemistry. 41(1). 21–27. 38 indexed citations

Barrass, Nigel, Roger J. Price, Brian G. Lake, & T.C. Orton. (1993). Comparison of the acute and chronic mitogenic effects of the peroxisome proliferators methylclofenapate and clofibric acid in rat liver. Carcinogenesis. 14(7). 1451–1456. 52 indexed citations

Barrass, Nigel, Mairi Stewart, D.A. Jackson, et al.. (1993). Cell Proliferation in the Liver and Thyroid of C57B1/10J Mice after Dietary Administration of Chlordane. Environmental Health Perspectives. 101. 219–219. 2 indexed citations

10.

Rumsby, Paul C., et al.. (1991). Analysis of the Ha-ras oncogene in C3H/He mouse liver tumours derived spontaneously or induced with diethylintrosamine or phenobarbitone. Carcinogenesis. 12(12). 2331–2336. 34 indexed citations

11.

Skilleter, David N., Nigel Barrass, & Roger J. Price. (1991). C‐myc expression is maintained during the G₁ phase cell cycle block produced by beryllium. Cell Proliferation. 24(2). 229–237. 2 indexed citations

12.

Volsen, Stephen G., Nigel Barrass, Mary P. Scott, & Klara Miller. (1989). Cellular and molecular effects of di-n-octyltin dichloride on the rat thymus. International Journal of Immunopharmacology. 11(6). 703–715. 8 indexed citations

13.

Barrass, Nigel. (1989). From genes to clones: Introduction to gene technology. Food and Chemical Toxicology. 27(6). 416–416. 2 indexed citations

14.

Sinha, Subrata, C Webber, C. J. Marshall, et al.. (1988). Activation of ras oncogene in aflatoxin-induced rat liver carcinogenesis.. Proceedings of the National Academy of Sciences. 85(11). 3673–3677. 59 indexed citations

15.

Barrass, Nigel. (1987). Oncogenes and growth control. Food and Chemical Toxicology. 25(9). 714–715. 7 indexed citations

16.

Padua, Rose Ann, et al.. (1985). Activation of N-ras in a human melanoma cell line.. Molecular and Cellular Biology. 5(3). 582–585. 44 indexed citations

17.

Padua, Rose Ann, Nigel Barrass, & G A Currie. (1985). Activation of N-ras in a Human Melanoma Cell Line. Molecular and Cellular Biology. 5(3). 582–585. 68 indexed citations

18.

Padua, Rose Ann, Nigel Barrass, & G A Currie. (1984). A novel transforming gene in a human malignant melanoma cell line. Nature. 311(5987). 671–673. 102 indexed citations

19.

Barrass, Nigel, Gail ter Haar, & G. Casey. (1982). The effect of ultrasound and hyperthermia on sister chromatid exchange and division kinetics of BHK 21 C13/A3 cells.. PubMed. 5. 187–91. 13 indexed citations

20.

West, Catharine, Ian J. Stratford, Nigel Barrass, & Elizabeth Smith. (1981). A comparison of adriamycin and mAMSA in vitro: Cell lethality and SCE studies. British Journal of Cancer. 44(6). 798–809. 22 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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