Nícolas C. Hoch

1.2k total citations
24 papers, 619 citations indexed

About

Nícolas C. Hoch is a scholar working on Molecular Biology, Oncology and Immunology. According to data from OpenAlex, Nícolas C. Hoch has authored 24 papers receiving a total of 619 indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Molecular Biology, 14 papers in Oncology and 5 papers in Immunology. Recurrent topics in Nícolas C. Hoch's work include DNA Repair Mechanisms (15 papers), PARP inhibition in cancer therapy (12 papers) and Microtubule and mitosis dynamics (4 papers). Nícolas C. Hoch is often cited by papers focused on DNA Repair Mechanisms (15 papers), PARP inhibition in cancer therapy (12 papers) and Microtubule and mitosis dynamics (4 papers). Nícolas C. Hoch collaborates with scholars based in Brazil, United Kingdom and Australia. Nícolas C. Hoch's co-authors include Keith W. Caldecott, Luis Mariano Polo, Stuart L. Rulten, Zhihong Zeng, William H. Gittens, Kevin Staras, Lilian C. Russo, Grace Yoon, Hana Hanzlíková and Martine Tétreault and has published in prestigious journals such as Nature, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Nícolas C. Hoch

24 papers receiving 616 citations

Peers

Nícolas C. Hoch

ONCOL

IMMUN

GENET

Tara L. Davis Canada

Yasuharu Tanaka Japan

Shengchen Lin China

Kheewoong Baek United States

Xinchao Yu United States

Justyna Iwaszkiewicz Switzerland

Sally Shpungin Israel

L. Holmberg-Schiavone Sweden

Mario Leutert Switzerland

Alma Seitova Canada

Tara L. Davis Canada

Nícolas C. Hoch

541 ×1.2

91 ×0.3

ONCOL

90 ×1.2

IMMUN

39 ×0.8

GENET

23 ×0.5

Citations per year, relative to Nícolas C. Hoch Nícolas C. Hoch (= 1×) peers Tara L. Davis

Countries citing papers authored by Nícolas C. Hoch

Since Specialization

Citations

This map shows the geographic impact of Nícolas C. Hoch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nícolas C. Hoch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nícolas C. Hoch more than expected).

Fields of papers citing papers by Nícolas C. Hoch

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nícolas C. Hoch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nícolas C. Hoch. The network helps show where Nícolas C. Hoch may publish in the future.

Co-authorship network of co-authors of Nícolas C. Hoch

This figure shows the co-authorship network connecting the top 25 collaborators of Nícolas C. Hoch. A scholar is included among the top collaborators of Nícolas C. Hoch based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nícolas C. Hoch. Nícolas C. Hoch is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Russo, Lilian C., et al.. (2025). Interferon-induced ADP-ribosylation: technical developments driving ICAB discovery. Bioscience Reports. 45(3). 197–209. 1 indexed citations

Hoch, Nícolas C., et al.. (2024). Molecular mechanisms of cell death by parthanatos: More questions than answers. Genetics and Molecular Biology. 47(suppl 1). e20230357–e20230357. 4 indexed citations

Leandro, Giovana da Silva, André Uchimura Bastos, Natália Cestari Moreno, et al.. (2024). Human translesion DNA polymerases ι and κ mediate tolerance to temozolomide in MGMT-deficient glioblastoma cells.. DNA repair. 141. 103715–103715. 1 indexed citations

Russo, Lilian C., et al.. (2024). PARP14 is regulated by the PARP9/DTX3L complex and promotes interferon γ-induced ADP-ribosylation. The EMBO Journal. 43(14). 2908–2928. 14 indexed citations

Schuller, M., et al.. (2024). Synthesis of Structural ADP-Ribose Analogues as Inhibitors for SARS-CoV-2 Macrodomain 1. Organic Letters. 26(27). 5700–5704. 1 indexed citations

Serrano-Benítez, Almudena, Lilian C. Russo, Mark A. Farrow, et al.. (2023). Unrepaired base excision repair intermediates in template DNA strands trigger replication fork collapse and PARP inhibitor sensitivity. The EMBO Journal. 42(18). e113190–e113190. 22 indexed citations

Hoch, Nícolas C.. (2023). Tissue Specificity of DNA Damage and Repair. Physiology. 38(5). 231–241. 9 indexed citations

Bertola, Débora Romeo, et al.. (2023). Rothmund-Thomson syndrome, a disorder far from solved. SHILAP Revista de lepidopterología. 4. 1296409–1296409. 9 indexed citations

Moreno, Natália Cestari, Giovana da Silva Leandro, André Uchimura Bastos, et al.. (2022). DNA polymerase eta protects human cells against DNA damage induced by the tumor chemotherapeutic temozolomide. Mutation Research/Genetic Toxicology and Environmental Mutagenesis. 878. 503498–503498. 9 indexed citations

10.

Russo, Lilian C., Rebeka Tomasin, Sven T. Sowa, et al.. (2021). The SARS-CoV-2 Nsp3 macrodomain reverses PARP9/DTX3L-dependent ADP-ribosylation induced by interferon signaling. Journal of Biological Chemistry. 297(3). 101041–101041. 69 indexed citations

11.

Hoch, Nícolas C.. (2021). Host ADP-ribosylation and the SARS-CoV-2 macrodomain. Biochemical Society Transactions. 49(4). 1711–1721. 15 indexed citations

12.

Breslin, Claire, Rajam S. Mani, Mesfin Fanta, et al.. (2017). The Rev1 interacting region (RIR) motif in the scaffold protein XRCC1 mediates a low-affinity interaction with polynucleotide kinase/phosphatase (PNKP) during DNA single-strand break repair. Journal of Biological Chemistry. 292(39). 16024–16031. 16 indexed citations

13.

Liu, Rui, Nícolas C. Hoch, Catherine Chang, et al.. (2017). ASCIZ/ATMIN is dispensable for ATM signaling in response to replication stress. DNA repair. 57. 29–34. 8 indexed citations

14.

Hoch, Nícolas C., Hana Hanzlíková, Stuart L. Rulten, et al.. (2016). XRCC1 mutation is associated with PARP1 hyperactivation and cerebellar ataxia. Nature. 541(7635). 87–91. 197 indexed citations

15.

Grundy, Gabrielle J., Luis Mariano Polo, Zhihong Zeng, et al.. (2016). PARP3 is a sensor of nicked nucleosomes and monoribosylates histone H2BGlu2. Nature Communications. 7(1). 12404–12404. 71 indexed citations

16.

Hoch, Nícolas C., et al.. (2013). Use of Quantitative Mass Spectrometric Analysis to Elucidate the Mechanisms of Phospho-priming and Auto-activation of the Checkpoint Kinase Rad53 in Vivo. Molecular & Cellular Proteomics. 13(2). 551–565. 14 indexed citations

17.

Hoch, Nícolas C.. (2012). Genomic stability disorders from budding yeast to humans. Frontiers in Bioscience-Scholar. S5(2). 396–411. 2 indexed citations

18.

Jurado, Sabine, Emma K. Baker, Nora Tenis, et al.. (2011). ATM Substrate Chk2-interacting Zn2+ Finger (ASCIZ) Is a Bi-functional Transcriptional Activator and Feedback Sensor in the Regulation of Dynein Light Chain (DYNLL1) Expression. Journal of Biological Chemistry. 287(5). 3156–3164. 49 indexed citations

19.

Hoch, Nícolas C., et al.. (2008). Allelism of Saccharomyces cerevisiae gene PSO10, involved in error-prone repair of psoralen-induced DNA damage, with SUMO ligase-encoding MMS21. Current Genetics. 53(6). 361–371. 4 indexed citations

20.

Rosa, Renato Moreira, Nícolas C. Hoch, Gabriel Vasata Furtado, Jenifer Saffi, & João Antônio Pêgas Henriques. (2007). DNA damage in tissues and organs of mice treated with diphenyl diselenide. Mutation Research/Genetic Toxicology and Environmental Mutagenesis. 633(1). 35–45. 30 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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