Neil D. Granger

501 total citations
13 papers, 385 citations indexed

About

Neil D. Granger is a scholar working on Immunology, Surgery and Molecular Biology. According to data from OpenAlex, Neil D. Granger has authored 13 papers receiving a total of 385 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Immunology, 3 papers in Surgery and 3 papers in Molecular Biology. Recurrent topics in Neil D. Granger's work include Inflammatory Bowel Disease (3 papers), Cell Adhesion Molecules Research (3 papers) and Cardiac Ischemia and Reperfusion (2 papers). Neil D. Granger is often cited by papers focused on Inflammatory Bowel Disease (3 papers), Cell Adhesion Molecules Research (3 papers) and Cardiac Ischemia and Reperfusion (2 papers). Neil D. Granger collaborates with scholars based in United States, Japan and Spain. Neil D. Granger's co-authors include Hideo Yoshida, Georg Singer, John C. McDonald, Gazi B. Zibari, Mark F. Brown, Julián Panés, Wolfgang H. Cerwinka, Feza M. Akgür, Inderjeet Singh and Jacob J. Burke and has published in prestigious journals such as Hepatology, Inflammatory Bowel Diseases and Shock.

In The Last Decade

Neil D. Granger

13 papers receiving 382 citations

Peers

Neil D. Granger

SURGE

IMMUN

EPIDE

GENET

PHYSI

Taisei Nakao Japan

Yekta Tüzün Türkiye

M Maruyama Japan

J C Jensen United States

Séverine Beaudreuil France

Nevin Oruç Türkiye

Eduardo Reyes Spain

Tsutomu Ishizuka Japan

Agnieszka Bińczak‐Kuleta Poland

Mitsue Sogawa Japan

Taisei Nakao Japan

Neil D. Granger

94 ×0.9

SURGE

103 ×1.0

IMMUN

77 ×1.0

EPIDE

57 ×0.8

GENET

75 ×1.5

PHYSI

Citations per year, relative to Neil D. Granger Neil D. Granger (= 1×) peers Taisei Nakao

Countries citing papers authored by Neil D. Granger

Since Specialization

Citations

This map shows the geographic impact of Neil D. Granger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Neil D. Granger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Neil D. Granger more than expected).

Fields of papers citing papers by Neil D. Granger

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Neil D. Granger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Neil D. Granger. The network helps show where Neil D. Granger may publish in the future.

Co-authorship network of co-authors of Neil D. Granger

This figure shows the co-authorship network connecting the top 25 collaborators of Neil D. Granger. A scholar is included among the top collaborators of Neil D. Granger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Neil D. Granger. Neil D. Granger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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13 of 13 papers shown

Yoshida, Hideo, et al.. (2010). Role of tumor necrosis factor-α in the extraintestinal thrombosis associated with colonic inflammation. Inflammatory Bowel Diseases. 17(11). 2217–2223. 44 indexed citations

Yoshida, Hideo & Neil D. Granger. (2009). Inflammatory bowel disease. Inflammatory Bowel Diseases. 15(8). 1245–1255. 101 indexed citations

Singer, Georg & Neil D. Granger. (2007). Inflammatory Responses Underlying the Microvascular Dysfunction Associated with Obesity and Insulin Resistance. Microcirculation. 14(4-5). 375–387. 98 indexed citations

Li, Jie, Wun-Ling Chang, Guang Sun, et al.. (2003). Intercellular Adhesion Molecule 1 Is Important for the Development of Severe Experimental Malaria but Is Not Required for Leukocyte Adhesion in the Brain. Journal of Investigative Medicine. 51(3). 128–140. 6 indexed citations

Cerwinka, Wolfgang H., et al.. (2001). HYPERCHOLESTEROLEMIA ALTERS ENDOTOXIN-INDUCED ENDOTHELIAL CELL ADHESION MOLECULE EXPRESSION. Shock. 16(1). 44–50. 16 indexed citations

Akgür, Feza M., Gazi B. Zibari, John C. McDonald, Neil D. Granger, & Mark F. Brown. (2000). KINETICS OF P-SELECTIN EXPRESSION IN REGIONAL VASCULAR BEDS AFTER RESUSCITATION OF HEMORRHAGIC SHOCK: A CLUE TO THE MECHANISM OF MULTIPLE SYSTEM ORGAN FAILURE. Shock. 13(2). 140–144. 13 indexed citations

Kawachi, Shigeyuki, Zenichi Morise, Stephen R. Jennings, et al.. (2000). Cytokine and Adhesion Molecule Expression in SCID Mice Reconstituted with CD4+ T Cells. Inflammatory Bowel Diseases. 6(3). 171–180. 33 indexed citations

Panés, Julián, Sofía Pérez‐del‐Pulgar, María Casadevall, et al.. (1999). Impaired mesenteric leukocyte recruitment in experimental portal hypertension in the rat. Hepatology. 30(2). 445–453. 20 indexed citations

Burke, Jacob J., Gazi B. Zibari, Mark F. Brown, et al.. (1998). Hepatic ischemia–reperfusion injury causes E-selectin upregulation. Transplantation Proceedings. 30(5). 2321–2323. 23 indexed citations

10.

Zibari, Gazi B., et al.. (1998). Role of P-selectin in the recruitment of leukocytes in mouse liver exposed to ischemia and reperfusion. Transplantation Proceedings. 30(5). 2327–2330. 18 indexed citations

11.

Eppihimer, Michael J. & Neil D. Granger. (1997). REGULATION OF ENDOTHELIAL CELL ADHESION MOLECULES IN THE INTESTINAL VASCULATURE. Shock. 7(Supplement). 48–48. 1 indexed citations

12.

Panés, Julián & Neil D. Granger. (1994). Leukocyte-Endothelial Cell Interactions: Implications for the Pathogenesis and Treatment of Gastrointestinal Disease. Digestive Diseases. 12(4). 232–241. 9 indexed citations

13.

Granger, Neil D., Peter R. Kvietys, Dale A. Parks, & Joseph N. Benoit. (1983). Intestinal Blood Flow: Relations to Function. Digestive Diseases. 1(4). 217–228. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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