Nathan Snyder

458 total citations · 1 hit paper
11 papers, 289 citations indexed

About

Nathan Snyder is a scholar working on Molecular Biology, Cell Biology and Pathology and Forensic Medicine. According to data from OpenAlex, Nathan Snyder has authored 11 papers receiving a total of 289 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 5 papers in Cell Biology and 3 papers in Pathology and Forensic Medicine. Recurrent topics in Nathan Snyder's work include Endoplasmic Reticulum Stress and Disease (5 papers), Ubiquitin and proteasome pathways (4 papers) and Genetic factors in colorectal cancer (3 papers). Nathan Snyder is often cited by papers focused on Endoplasmic Reticulum Stress and Disease (5 papers), Ubiquitin and proteasome pathways (4 papers) and Genetic factors in colorectal cancer (3 papers). Nathan Snyder collaborates with scholars based in United States, Switzerland and China. Nathan Snyder's co-authors include Gustavo M. Silva, Kyle W. Cunningham, Linghuo Jiang, Adam Kim, Junjun Wang, Mitchell V. Palmer, Timothy A. Reinhardt, Christopher P. Stefan, Carlos Evangelista and Ye Zhou and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Oncology and Cell Reports.

In The Last Decade

Nathan Snyder

8 papers receiving 288 citations

Hit Papers

Deubiquitinating enzymes (DUBs): Regulation, homeostasis,... 2021 2026 2022 2024 2021 50 100 150

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Nathan Snyder United States 6 226 56 48 41 30 11 289
Xingrun Zhang China 6 280 1.2× 37 0.7× 53 1.1× 33 0.8× 41 1.4× 7 360
Emmanuelle Soleilhac France 12 252 1.1× 41 0.7× 72 1.5× 40 1.0× 24 0.8× 22 383
Pille Pata Estonia 9 183 0.8× 33 0.6× 23 0.5× 30 0.7× 17 0.6× 15 310
Tatsuya Yano Japan 11 210 0.9× 51 0.9× 25 0.5× 26 0.6× 41 1.4× 27 388
Shahlo Тurdikulova Uzbekistan 8 155 0.7× 35 0.6× 39 0.8× 20 0.5× 37 1.2× 16 251
Ruobing Ren China 11 215 1.0× 32 0.6× 25 0.5× 21 0.5× 21 0.7× 27 365
Christina Seebode Germany 9 154 0.7× 42 0.8× 67 1.4× 57 1.4× 39 1.3× 17 299
Anant Vasudevan United States 3 269 1.2× 50 0.9× 120 2.5× 31 0.8× 15 0.5× 5 387
Kathrin Schmich Germany 7 214 0.9× 38 0.7× 30 0.6× 58 1.4× 13 0.4× 7 348
Helene Klug Austria 6 319 1.4× 114 2.0× 71 1.5× 39 1.0× 32 1.1× 7 382

Countries citing papers authored by Nathan Snyder

Since Specialization
Citations

This map shows the geographic impact of Nathan Snyder's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nathan Snyder with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nathan Snyder more than expected).

Fields of papers citing papers by Nathan Snyder

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nathan Snyder. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nathan Snyder. The network helps show where Nathan Snyder may publish in the future.

Co-authorship network of co-authors of Nathan Snyder

This figure shows the co-authorship network connecting the top 25 collaborators of Nathan Snyder. A scholar is included among the top collaborators of Nathan Snyder based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nathan Snyder. Nathan Snyder is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
1.
Xu, Jianfeng, Nathan Snyder, Jun Wei, et al.. (2025). Established Cancer Predisposition Genes in Single and Multiple Cancer Diagnoses. JAMA Oncology. 11(10). 1222–1222.
2.
Chen, Chia‐Yu, et al.. (2024). Redox control of the deubiquitinating enzyme Ubp2 regulates translation during stress. Journal of Biological Chemistry. 300(11). 107870–107870. 3 indexed citations
3.
Snyder, Nathan, Ann G. Schwartz, Jun Wei, et al.. (2024). The contribution of rare and common genetic variants to risk of prostate cancer and second primary cancer after prostate in the UK Biobank.. Journal of Clinical Oncology. 42(16_suppl). 10604–10604.
4.
Feng, Bing, Nathan Snyder, Zhuqing Shi, et al.. (2024). Using gene and gene-set association tests to identify lethal prostate cancer genes. Prostate Cancer and Prostatic Diseases. 28(2). 490–495.
5.
Xu, Jianfeng, Nathan Snyder, Zhuqing Shi, et al.. (2024). Association between rare pathogenic variants in established cancer risk genes and the diagnosis of single and multiple common cancers: A UK Biobank study.. Journal of Clinical Oncology. 42(16_suppl). 10505–10505. 1 indexed citations
6.
Zhou, Ye, Nathan Snyder, Jonathan Bouvette, et al.. (2022). Redox-sensitive E2 Rad6 controls cellular response to oxidative stress via K63-linked ubiquitination of ribosomes. Cell Reports. 39(8). 110860–110860. 23 indexed citations
7.
Snyder, Nathan & Gustavo M. Silva. (2021). Deubiquitinating enzymes (DUBs): Regulation, homeostasis, and oxidative stress response. Journal of Biological Chemistry. 297(3). 101077–101077. 186 indexed citations breakdown →
8.
Snyder, Nathan, Mitchell V. Palmer, Timothy A. Reinhardt, & Kyle W. Cunningham. (2019). Milk biosynthesis requires the Golgi cation exchanger TMEM165. Journal of Biological Chemistry. 294(9). 3181–3191. 16 indexed citations
9.
Snyder, Nathan, Adam Kim, Andrew N. Gale, et al.. (2019). Auxin-Inducible Depletion of the Essentialome Suggests Inhibition of TORC1 by Auxins and Inhibition of Vrg4 by SDZ 90-215, a Natural Antifungal Cyclopeptide. G3 Genes Genomes Genetics. 9(3). 829–840. 17 indexed citations
10.
Jiang, Linghuo, et al.. (2018). CaGdt1 plays a compensatory role for the calcium pump CaPmr1 in the regulation of calcium signaling and cell wall integrity signaling in Candida albicans. Cell Communication and Signaling. 16(1). 33–33. 23 indexed citations
11.
Snyder, Nathan, et al.. (2017). H+ and Pi Byproducts of Glycosylation Affect Ca2+ Homeostasis and Are Retrieved from the Golgi Complex by Homologs of TMEM165 and XPR1. G3 Genes Genomes Genetics. 7(12). 3913–3924. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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