Nathalie Rousselet

594 total citations
15 papers, 467 citations indexed

About

Nathalie Rousselet is a scholar working on Pulmonary and Respiratory Medicine, Molecular Biology and Cancer Research. According to data from OpenAlex, Nathalie Rousselet has authored 15 papers receiving a total of 467 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Pulmonary and Respiratory Medicine, 7 papers in Molecular Biology and 6 papers in Cancer Research. Recurrent topics in Nathalie Rousselet's work include Cystic Fibrosis Research Advances (6 papers), Neonatal Respiratory Health Research (6 papers) and Protease and Inhibitor Mechanisms (3 papers). Nathalie Rousselet is often cited by papers focused on Cystic Fibrosis Research Advances (6 papers), Neonatal Respiratory Health Research (6 papers) and Protease and Inhibitor Mechanisms (3 papers). Nathalie Rousselet collaborates with scholars based in France, United States and United Kingdom. Nathalie Rousselet's co-authors include Raymond Frade, Didier Jean, Harriet Corvol, Olivier Tabary, Philippe Le Rouzic, Bernárd P. Roques, Marie‐Claude Fournié‐Zaluski, Carmen S. Tellez, Manon Ruffin and Menashe Bar‐Eli and has published in prestigious journals such as Journal of Biological Chemistry, Nature Communications and Cancer Research.

In The Last Decade

Nathalie Rousselet

14 papers receiving 459 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Nathalie Rousselet France 12 273 166 116 100 42 15 467
Mark E. Hatley United States 11 441 1.6× 173 1.0× 94 0.8× 264 2.6× 20 0.5× 18 635
Rene Pagila United States 5 236 0.9× 95 0.6× 80 0.7× 58 0.6× 8 0.2× 6 557
Antonia Richter Germany 9 155 0.6× 66 0.4× 209 1.8× 45 0.5× 22 0.5× 15 443
Eun‐Kyoung Breuer United States 13 309 1.1× 152 0.9× 181 1.6× 65 0.7× 9 0.2× 17 475
N Gutman France 14 167 0.6× 71 0.4× 68 0.6× 70 0.7× 21 0.5× 30 483
Stanislav Zelivianski United States 12 366 1.3× 104 0.6× 190 1.6× 234 2.3× 32 0.8× 16 605
Shekhar Saha United States 13 339 1.2× 240 1.4× 69 0.6× 47 0.5× 6 0.1× 34 529
Giuseppina Claps United States 9 322 1.2× 142 0.9× 107 0.9× 101 1.0× 8 0.2× 13 498
Maria Nyåkern Italy 7 426 1.6× 62 0.4× 82 0.7× 19 0.2× 17 0.4× 11 585
Tracy Warr United Kingdom 13 268 1.0× 112 0.7× 94 0.8× 51 0.5× 12 0.3× 27 492

Countries citing papers authored by Nathalie Rousselet

Since Specialization
Citations

This map shows the geographic impact of Nathalie Rousselet's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nathalie Rousselet with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nathalie Rousselet more than expected).

Fields of papers citing papers by Nathalie Rousselet

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nathalie Rousselet. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nathalie Rousselet. The network helps show where Nathalie Rousselet may publish in the future.

Co-authorship network of co-authors of Nathalie Rousselet

This figure shows the co-authorship network connecting the top 25 collaborators of Nathalie Rousselet. A scholar is included among the top collaborators of Nathalie Rousselet based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nathalie Rousselet. Nathalie Rousselet is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
1.
Rousselet, Nathalie, et al.. (2025). Restoring chloride efflux in cystic fibrosis with TMEM16a antisense oligonucleotides. Molecular Therapy. 33(12). 6463–6478.
2.
Philippart, François, Emmanuelle Sacco, Stéphane Legriel, et al.. (2024). Multicentric investigations of the role in the disease severity of accelerated phospholipid changes in COVID-19 patient airway. Microbes and Infection. 26(5-6). 105354–105354. 2 indexed citations
3.
Rousselet, Nathalie, et al.. (2019). miR-636: A Newly-Identified Actor for the Regulation of Pulmonary Inflammation in Cystic Fibrosis. Frontiers in Immunology. 10. 2643–2643. 15 indexed citations
4.
Taytard, Jessica, Nathalie Rousselet, Dominique Debray, et al.. (2018). Opposite Expression of Hepatic and Pulmonary Corticosteroid-Binding Globulin in Cystic Fibrosis Patients. Frontiers in Pharmacology. 9. 545–545. 2 indexed citations
5.
Blouquit‐Laye, Sabine, et al.. (2018). Small RNA and transcriptome sequencing reveal the role of miR‐199a‐3p in inflammatory processes in cystic fibrosis airways. The Journal of Pathology. 245(4). 410–420. 34 indexed citations
6.
Ruffin, Manon, Christelle Coraux, Nathalie Rousselet, et al.. (2017). MicroRNA-9 downregulates the ANO1 chloride channel and contributes to cystic fibrosis lung pathology. Nature Communications. 8(1). 710–710. 55 indexed citations
7.
Corvol, Harriet, Nathalie Rousselet, Kristin Thompson, et al.. (2017). FAM13A is a modifier gene of cystic fibrosis lung phenotype regulating rhoa activity, actin cytoskeleton dynamics and epithelial-mesenchymal transition. Journal of Cystic Fibrosis. 17(2). 190–203. 35 indexed citations
8.
Ruffin, Manon, Loïc Guillot, Nathalie Rousselet, et al.. (2015). New Insights about miRNAs in Cystic Fibrosis. American Journal Of Pathology. 185(4). 897–908. 36 indexed citations
9.
Ndong, Jean De La Croix, Didier Jean, Nathalie Rousselet, & Raymond Frade. (2009). Down‐regulation of the expression of RB18A/MED1, a cofactor of transcription, triggers strong tumorigenic phenotype of human melanoma cells. International Journal of Cancer. 124(11). 2597–2606. 24 indexed citations
10.
Frade, Raymond, Nathalie Rousselet, & Didier Jean. (2008). Intratumoral gene delivery of anti-cathepsin L single-chain variable fragment by lentiviral vector inhibits tumor progression induced by human melanoma cells. Cancer Gene Therapy. 15(9). 591–604. 17 indexed citations
11.
Jean, Didier, Nathalie Rousselet, & Raymond Frade. (2008). Cathepsin L expression is up-regulated by hypoxia in human melanoma cells: role of its 5′-untranslated region. Biochemical Journal. 413(1). 125–134. 21 indexed citations
12.
Jean, Didier, Nathalie Rousselet, & Raymond Frade. (2005). Expression of cathepsin L in human tumor cells is under the control of distinct regulatory mechanisms. Oncogene. 25(10). 1474–1484. 25 indexed citations
13.
Rousselet, Nathalie, Lisa Mills, Didier Jean, et al.. (2004). Inhibition of Tumorigenicity and Metastasis of Human Melanoma Cells by Anti-Cathepsin L Single Chain Variable Fragment. Cancer Research. 64(1). 146–151. 74 indexed citations
14.
Beaumont, Ann, Michael O’Donohue, Nathalie Rousselet, et al.. (1995). The Role of Histidine 231 in Thermolysin-like Enzymes.. Journal of Biological Chemistry. 270(28). 16803–16808. 60 indexed citations
15.
Fournié‐Zaluski, Marie‐Claude, Pascale Coric, Serge Turcaud, et al.. (1994). New Dual Inhibitors of Neutral Endopeptidase and Angiotensin-Converting Enzyme: Rational Design, Bioavailability, and Pharmacological Responses in Experimental Hypertension. Journal of Medicinal Chemistry. 37(8). 1070–1083. 67 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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