Naomi Goldfinger

7.6k total citations
93 papers, 6.2k citations indexed

About

Naomi Goldfinger is a scholar working on Oncology, Molecular Biology and Biotechnology. According to data from OpenAlex, Naomi Goldfinger has authored 93 papers receiving a total of 6.2k indexed citations (citations by other indexed papers that have themselves been cited), including 67 papers in Oncology, 59 papers in Molecular Biology and 26 papers in Biotechnology. Recurrent topics in Naomi Goldfinger's work include Cancer-related Molecular Pathways (61 papers), Cancer Research and Treatments (26 papers) and Epigenetics and DNA Methylation (15 papers). Naomi Goldfinger is often cited by papers focused on Cancer-related Molecular Pathways (61 papers), Cancer Research and Treatments (26 papers) and Epigenetics and DNA Methylation (15 papers). Naomi Goldfinger collaborates with scholars based in Israel, United States and Germany. Naomi Goldfinger's co-authors include Varda Rotter, Gad Shaulsky, Dov Schwartz, Michael Milyavsky, Shalom Madar, Igor Shats, Alina Molchadsky, Ran Brosh, Ido Goldstein and Moshe Oren and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Nature Genetics.

In The Last Decade

Naomi Goldfinger

93 papers receiving 6.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Naomi Goldfinger Israel 50 4.3k 3.2k 1.5k 840 478 93 6.2k
Takashi Tokino Japan 10 6.0k 1.4× 5.4k 1.7× 1.3k 0.9× 1.2k 1.4× 565 1.2× 14 8.1k
Lee Ann Remington United States 13 3.5k 0.8× 3.5k 1.1× 981 0.7× 800 1.0× 457 1.0× 15 5.8k
Takehiko Kamijo Japan 37 5.1k 1.2× 3.2k 1.0× 1.0k 0.7× 455 0.5× 462 1.0× 114 7.1k
Christine E. Canman United States 34 6.0k 1.4× 4.1k 1.3× 1.6k 1.1× 535 0.6× 368 0.8× 49 7.2k
Margaret Ashcroft United Kingdom 34 3.2k 0.7× 1.7k 0.5× 1.7k 1.2× 389 0.5× 361 0.8× 58 4.6k
Meredith S. Irwin Canada 43 4.2k 1.0× 3.1k 1.0× 1.5k 1.0× 825 1.0× 381 0.8× 124 6.7k
Ada Sacchi Italy 55 5.8k 1.3× 3.7k 1.2× 1.5k 1.0× 868 1.0× 612 1.3× 162 8.4k
B Vogelstein United States 23 5.4k 1.3× 4.2k 1.3× 2.1k 1.4× 646 0.8× 1000 2.1× 23 8.5k
Daniel B. Levy United States 10 6.2k 1.4× 5.8k 1.8× 1.6k 1.1× 1.1k 1.4× 882 1.8× 13 9.1k
Sheau-Yann Shieh Taiwan 28 6.2k 1.4× 4.6k 1.5× 1.3k 0.9× 774 0.9× 530 1.1× 39 7.4k

Countries citing papers authored by Naomi Goldfinger

Since Specialization
Citations

This map shows the geographic impact of Naomi Goldfinger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Naomi Goldfinger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Naomi Goldfinger more than expected).

Fields of papers citing papers by Naomi Goldfinger

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Naomi Goldfinger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Naomi Goldfinger. The network helps show where Naomi Goldfinger may publish in the future.

Co-authorship network of co-authors of Naomi Goldfinger

This figure shows the co-authorship network connecting the top 25 collaborators of Naomi Goldfinger. A scholar is included among the top collaborators of Naomi Goldfinger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Naomi Goldfinger. Naomi Goldfinger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Koifman, Gabriela, Yoav Shetzer, Hilla Solomon, et al.. (2018). A Mutant p53-Dependent Embryonic Stem Cell Gene Signature Is Associated with Augmented Tumorigenesis of Stem Cells. Cancer Research. 78(20). 5833–5847. 17 indexed citations
2.
Charni‐Natan, Meital, et al.. (2018). Various stress stimuli rewire the profile of liver secretome in a p53-dependent manner. Cell Death and Disease. 9(6). 647–647. 9 indexed citations
3.
Koifman, Gabriela, Alon Silberman, Hilla Solomon, et al.. (2018). Mutant p53-dependent mitochondrial metabolic alterations in a mesenchymal stem cell-based model of progressive malignancy. Cell Death and Differentiation. 26(9). 1566–1581. 29 indexed citations
4.
Madar, Shalom, Ido Goldstein, Yan Stein, et al.. (2013). Mutant p53 Attenuates the Anti-Tumorigenic Activity of Fibroblasts-Secreted Interferon Beta. PLoS ONE. 8(4). e61353–e61353. 36 indexed citations
5.
Bornstein, Chamutal, Ran Brosh, Alina Molchadsky, et al.. (2011). SPATA18, a Spermatogenesis-Associated Gene, Is a Novel Transcriptional Target of p53 and p63. Molecular and Cellular Biology. 31(8). 1679–1689. 33 indexed citations
6.
Buganim, Yosef, Hilla Solomon, Yoach Rais, et al.. (2010). p53 Regulates the Ras Circuit to Inhibit the Expression of a Cancer-Related Gene Signature by Various Molecular Pathways. Cancer Research. 70(6). 2274–2284. 64 indexed citations
7.
Paland, Nicole, Iris Kamer, Ira Kogan-Sakin, et al.. (2009). Differential Influence of Normal and Cancer-Associated Fibroblasts on the Growth of Human Epithelial Cells in an In vitro Cocultivation Model of Prostate Cancer. Molecular Cancer Research. 7(8). 1212–1223. 56 indexed citations
8.
Ke, Xisong, Yi Qu, Kari Rostad, et al.. (2009). Genome-Wide Profiling of Histone H3 Lysine 4 and Lysine 27 Trimethylation Reveals an Epigenetic Signature in Prostate Carcinogenesis. PLoS ONE. 4(3). e4687–e4687. 115 indexed citations
9.
Ke, Xisong, Yi Qu, Naomi Goldfinger, et al.. (2008). Epithelial to Mesenchymal Transition of a Primary Prostate Cell Line with Switches of Cell Adhesion Modules but without Malignant Transformation. PLoS ONE. 3(10). e3368–e3368. 49 indexed citations
10.
Efrati, Shai, Sylvia Berman, Naomi Goldfinger, et al.. (2007). Enhanced angiotensin II production by renal mesangium is responsible for apoptosis/proliferation of endothelial and epithelial cells in a model of malignant hypertension. Journal of Hypertension. 25(5). 1041–1052. 19 indexed citations
11.
Milyavsky, Michael, Igor Shats, Ran Brosh, et al.. (2007). Inactivation of Myocardin and p16 during Malignant Transformation Contributes to a Differentiation Defect. Cancer Cell. 11(2). 133–146. 61 indexed citations
12.
Kogan, Ira, Naomi Goldfinger, Michael Milyavsky, et al.. (2006). hTERT-Immortalized Prostate Epithelial and Stromal-Derived Cells: an Authentic In vitro Model for Differentiation and Carcinogenesis. Cancer Research. 66(7). 3531–3540. 86 indexed citations
13.
Buganim, Yosef, Eyal Kalo, Ran Brosh, et al.. (2006). Mutant p53 Protects Cells from 12- O -Tetradecanoylphorbol-13-Acetate–Induced Death by Attenuating Activating Transcription Factor 3 Induction. Cancer Research. 66(22). 10750–10759. 31 indexed citations
14.
Milyavsky, Michael, Yuval Tabach, Igor Shats, et al.. (2005). Transcriptional Programs following Genetic Alterations in p53 , INK4A , and H-Ras Genes along Defined Stages of Malignant Transformation. Cancer Research. 65(11). 4530–4543. 51 indexed citations
15.
Sigal, Alex, Devorah Matas, Nava Almog, Naomi Goldfinger, & Varda Rotter. (2001). The C-terminus of mutant p53 is necessary for its ability to interfere with growth arrest or apoptosis. Oncogene. 20(35). 4891–4898. 15 indexed citations
16.
Almog, Nava, Runzhao Li, Amnon Peled, et al.. (1997). The Murine C′-Terminally Alternatively Spliced Form of p53 Induces Attenuated Apoptosis in Myeloid Cells. Molecular and Cellular Biology. 17(2). 713–722. 24 indexed citations
17.
Sthoeger, Zev, Ella Evron, Sorel Goland, et al.. (1997). Anti‐p53 Autoantibodies in Colon Cancer Patients. Annals of the New York Academy of Sciences. 815(1). 496–498. 7 indexed citations
18.
Ronen, D, et al.. (1996). Induction of HL-60 cells to undergo apoptosis is determined by high levels of wild-type p53 protein whereas differentiation of the cells is mediated by lower p53 levels.. PubMed. 7(1). 21–30. 61 indexed citations
19.
Shaulsky, Gad, et al.. (1991). Nuclear localization is essential for the activity of p53 protein.. PubMed. 6(11). 2055–65. 166 indexed citations
20.
Prokocimer, Miron, et al.. (1986). Expression of p53 in human leukemia and lymphoma. Blood. 68(1). 113–118. 85 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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