Naoki Saita

1.7k total citations
41 papers, 1.4k citations indexed

About

Naoki Saita is a scholar working on Pulmonary and Respiratory Medicine, Immunology and Rheumatology. According to data from OpenAlex, Naoki Saita has authored 41 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Pulmonary and Respiratory Medicine, 16 papers in Immunology and 12 papers in Rheumatology. Recurrent topics in Naoki Saita's work include Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis (14 papers), Eosinophilic Disorders and Syndromes (12 papers) and Galectins and Cancer Biology (12 papers). Naoki Saita is often cited by papers focused on Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis (14 papers), Eosinophilic Disorders and Syndromes (12 papers) and Galectins and Cancer Biology (12 papers). Naoki Saita collaborates with scholars based in Japan, United States and Taiwan. Naoki Saita's co-authors include Mitsuomi Hirashima, Akira Yamauchi, Nozomu Nishi, Kazuhiro Iyonaga, Masako Seki, Souichi Oomizu, Tomohiro Arikawa, Toshiro Niki, Ken-mei Sakata and Akihiro Matsukawa and has published in prestigious journals such as Journal of Clinical Oncology, The Journal of Immunology and PLoS ONE.

In The Last Decade

Naoki Saita

40 papers receiving 1.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Naoki Saita Japan 14 1.0k 480 307 243 134 41 1.4k
Pedro Cano United States 19 740 0.7× 134 0.3× 31 0.1× 159 0.7× 127 0.9× 56 1.4k
Kathryn Hobbs United States 13 424 0.4× 209 0.4× 74 0.2× 95 0.4× 39 0.3× 25 988
Osamu Saiki Japan 21 788 0.8× 219 0.5× 113 0.4× 233 1.0× 230 1.7× 52 1.4k
Kazuhiro Usui Japan 19 235 0.2× 392 0.8× 882 2.9× 724 3.0× 117 0.9× 90 1.7k
Yoshitaro Shindo Japan 19 334 0.3× 263 0.5× 191 0.6× 552 2.3× 248 1.9× 94 1.0k
Kayoko Sato Japan 17 650 0.6× 275 0.6× 109 0.4× 163 0.7× 169 1.3× 52 1.1k
AA Fauser Germany 22 490 0.5× 342 0.7× 129 0.4× 370 1.5× 63 0.5× 63 2.1k
Caroline Mary France 16 611 0.6× 199 0.4× 206 0.7× 117 0.5× 141 1.1× 34 1.1k
Jean‐Michel Boiron France 21 336 0.3× 255 0.5× 185 0.6× 313 1.3× 153 1.1× 60 1.7k
Masaharu Kasai Japan 22 501 0.5× 252 0.5× 98 0.3× 375 1.5× 230 1.7× 89 1.6k

Countries citing papers authored by Naoki Saita

Since Specialization
Citations

This map shows the geographic impact of Naoki Saita's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Naoki Saita with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Naoki Saita more than expected).

Fields of papers citing papers by Naoki Saita

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Naoki Saita. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Naoki Saita. The network helps show where Naoki Saita may publish in the future.

Co-authorship network of co-authors of Naoki Saita

This figure shows the co-authorship network connecting the top 25 collaborators of Naoki Saita. A scholar is included among the top collaborators of Naoki Saita based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Naoki Saita. Naoki Saita is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
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Kojima, Keisuke, Tomohiro Arikawa, Naoki Saita, et al.. (2011). Galectin-9 Attenuates Acute Lung Injury by Expanding CD14– Plasmacytoid Dendritic Cell–like Macrophages. American Journal of Respiratory and Critical Care Medicine. 184(3). 328–339. 36 indexed citations
4.
Arikawa, Tomohiro, Naoki Saita, Souichi Oomizu, et al.. (2009). Galectin‐9 expands immunosuppressive macrophages to ameliorate T‐cell‐mediated lung inflammation. European Journal of Immunology. 40(2). 548–558. 55 indexed citations
5.
Arikawa, Tomohiro, Kota Watanabe, Masako Seki, et al.. (2009). Galectin-9 ameliorates immune complex-induced arthritis by regulating FcγR expression on macrophages. Clinical Immunology. 133(3). 382–392. 51 indexed citations
6.
Seki, Masako, Souichi Oomizu, Ken-mei Sakata, et al.. (2008). Galectin-9 suppresses the generation of Th17, promotes the induction of regulatory T cells, and regulates experimental autoimmune arthritis. Clinical Immunology. 127(1). 78–88. 395 indexed citations
7.
Seki, Masako, Ken-mei Sakata, Souichi Oomizu, et al.. (2007). Beneficial effect of galectin 9 on rheumatoid arthritis by induction of apoptosis of synovial fibroblasts. Arthritis & Rheumatism. 56(12). 3968–3976. 95 indexed citations
8.
Asakura, Hirofumi, Yumiko Kashio, Kazuhiro Nakamura, et al.. (2002). Selective Eosinophil Adhesion to Fibroblast Via IFN-γ-Induced Galectin-9. The Journal of Immunology. 169(10). 5912–5918. 126 indexed citations
9.
Saita, Naoki, Taro Yamamoto, Isamu Cho, et al.. (2002). Association of Galectin-9 with Eosinophil Apoptosis. International Archives of Allergy and Immunology. 128(1). 42–50. 58 indexed citations
10.
Saita, Naoki, et al.. (2001). Apoptotic response of eosinophils in chronic eosinophilic pneumonia. European Respiratory Journal. 17(2). 190–194. 11 indexed citations
11.
Yamanaka, Tohru, Naoki Saita, Osamu Kawano, et al.. (2000). Isolation of a Lactose-Binding Protein with Monocyte/Macrophage Chemotactic Activity. International Archives of Allergy and Immunology. 122(1). 66–75. 3 indexed citations
12.
Suga, Motomu, Kazuhiro Iyonaga, Hidenori Ichiyasu, et al.. (1999). Clinical significance of MCP‐1 levels in BALF and serum in patients with interstitial lung diseases. European Respiratory Journal. 14(2). 376–376. 150 indexed citations
13.
Saita, Naoki, Tohru Yamanaka, Hirotsugu Kohrogi, et al.. (1997). Expression of Apoptosis-Related Antigen on Eosinophils in Chronic Eosinophilic Pneumonia. International Archives of Allergy and Immunology. 114(1). 64–66. 6 indexed citations
14.
Saita, Naoki, et al.. (1995). Chemotactic Heterogeneity of Eosinophils in Idiopathic Pulmonary Eosinophilia. International Archives of Allergy and Immunology. 108(1). 25–27. 3 indexed citations
15.
Sakamoto, Osamu, et al.. (1994). Pulmonary Granulomatosis Caused by Aspirated Green Tea. CHEST Journal. 106(1). 308–309. 7 indexed citations
16.
Iyonaga, Kazuhiro, Motohiro Takeya, Naoki Saita, et al.. (1994). Monocyte chemoattractant protein-1 in idiopathic pulmonary fibrosis and other interstitial lung diseases. Human Pathology. 25(5). 455–463. 86 indexed citations
17.
Saita, Naoki, Ken-mei Sakata, Mitsuhiro Matsumoto, et al.. (1994). Production of fibroblast proliferative cytokines from T lymphocytes stimulated by a B cell lymphoma line and their functional heterogeneity. Immunology Letters. 41(2-3). 279–286. 4 indexed citations
18.
Hirashima, Mitsuomi, et al.. (1993). Induction of an eosinophil chemotactic factor production from T lymphocytes by a B cell lymphoma line. Immunology Letters. 36(3). 273–281. 6 indexed citations
19.
Saita, Naoki, Masayuki Ando, Shukuro Araki, & Masaru Yoshinaga. (1991). Fibroblast Proliferation Factors in Pulmonary Granuloma Induced by <i>Trichosporon cutaneum</i> in Rabbits: Presence of a Lymphocyte-Derived Fibroblast Proliferation Factor and Its Functional Specificity. International Archives of Allergy and Immunology. 95(4). 294–302. 3 indexed citations
20.
Ohkawara, Susumu, et al.. (1989). Interleukin-1 Production by Polymorphonuclear Leukocytes during the Course of Acute Inflammation in Rabbits. Dermatology. 179(Suppl. 1). 84–90. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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