Nadia Al‐Fakhri
- Molecular Biology
- Clinical Biochemistry top 1%
- Immunology top 10%
- Immunology and Allergy top 2%
- Hematology top 5%
- Co-authors
- Klaus T. PreissnerTriantafyllos ChavakisPeter P. NawrothMichael SchoppetJohn MorserThomas LinnMariko NagashimaSteffen Witte
- Topics
- Cell Adhesion Molecules Research (10 papers)Protease and Inhibitor Mechanisms (5 papers)Bone Metabolism and Diseases (5 papers)
- Journals
- The Journal of Experimental MedicineThe Journal of ImmunologyThe Journal of Clinical Endocrinology & Metabolism
- Partner nations
- GermanyUnited StatesAustralia
In The Last Decade
Nadia Al‐Fakhri
26 papers receiving 1.4k citations
Peers
Comparison fields: 5 of 87
- Molecular Biology 480
- Clinical Biochemistry 349
- Immunology 344
- Immunology and Allergy 212
- Hematology 181
Countries citing papers authored by Nadia Al‐Fakhri
This map shows the geographic impact of Nadia Al‐Fakhri's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nadia Al‐Fakhri with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nadia Al‐Fakhri more than expected).
Fields of papers citing papers by Nadia Al‐Fakhri
This network shows the impact of papers produced by Nadia Al‐Fakhri. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nadia Al‐Fakhri. The network helps show where Nadia Al‐Fakhri may publish in the future.
Co-authorship network of co-authors of Nadia Al‐Fakhri
This figure shows the co-authorship network connecting the top 25 collaborators of Nadia Al‐Fakhri. A scholar is included among the top collaborators of Nadia Al‐Fakhri based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nadia Al‐Fakhri. Nadia Al‐Fakhri is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 11 | |
| 2 | 7 | |
| 3 | 5 | |
| 4 | 13 | |
| 5 | 2 | |
| 6 | 13 | |
| 7 | 119 | |
| 8 | 32 | |
| 9 | 23 | |
| 10 | 5 | |
| 11 | 72 | |
| 12 | 50 | |
| 13 | 155 | |
| 14 | 489 | |
| 15 | 50 | |
| 16 | 7 | |
| 17 | 49 | |
| 18 | 45 | |
| 19 | 31 | |
| 20 | 2 |
About Nadia Al‐Fakhri
Nadia Al‐Fakhri is a scholar working on Immunology and Allergy, Hematology and Cancer Research, having authored 26 papers that have together received 1.4k indexed citations. Recurring topics across this work include Cell Adhesion Molecules Research (10 papers), Protease and Inhibitor Mechanisms (5 papers) and Bone Metabolism and Diseases (5 papers). The work is most often cited by research in Clinical Biochemistry (349 citations), Immunology and Allergy (212 citations) and Immunology (344 citations). Nadia Al‐Fakhri has collaborated with scholars based in Germany, United States and Australia. Frequent co-authors include Klaus T. Preissner, Triantafyllos Chavakis, Peter P. Nawroth, Michael Schoppet, John Morser, Thomas Linn, Mariko Nagashima, Steffen Witte, Darius Schneider and Bernd Arnold. Their work appears in journals such as The Journal of Experimental Medicine, The Journal of Immunology and The Journal of Clinical Endocrinology & Metabolism.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.