Nadia Akawi

2.4k total citations
41 papers, 637 citations indexed

About

Nadia Akawi is a scholar working on Molecular Biology, Genetics and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Nadia Akawi has authored 41 papers receiving a total of 637 indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Molecular Biology, 11 papers in Genetics and 8 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Nadia Akawi's work include RNA modifications and cancer (5 papers), Genetic Syndromes and Imprinting (4 papers) and Cardiovascular Disease and Adiposity (4 papers). Nadia Akawi is often cited by papers focused on RNA modifications and cancer (5 papers), Genetic Syndromes and Imprinting (4 papers) and Cardiovascular Disease and Adiposity (4 papers). Nadia Akawi collaborates with scholars based in United Arab Emirates, United Kingdom and United States. Nadia Akawi's co-authors include Bassam R. Ali, Lihadh Al‐Gazali, Anne John, Charalambos Antoniades, Ioannis Akoumianakis, Ruth Langer, Noushad Karuvantevida, Huifang Xu, Birgit Leitinger and Ganeshwaran H. Mochida and has published in prestigious journals such as Circulation, Journal of the American College of Cardiology and PLoS ONE.

In The Last Decade

Nadia Akawi

35 papers receiving 634 citations

Peers

Nadia Akawi
Raúl Tonda Spain
Vreni Schneider Switzerland
Emily J. Welch United States
Jeanette Hamlington United States
Barbara D. Summers United States
Peisong Ma United States
Ilkka Jaakkola Finland
Xiangrong Dai China
Shogo Kasuda Japan
Raúl Tonda Spain
Nadia Akawi
Citations per year, relative to Nadia Akawi Nadia Akawi (= 1×) peers Raúl Tonda

Countries citing papers authored by Nadia Akawi

Since Specialization
Citations

This map shows the geographic impact of Nadia Akawi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nadia Akawi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nadia Akawi more than expected).

Fields of papers citing papers by Nadia Akawi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nadia Akawi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nadia Akawi. The network helps show where Nadia Akawi may publish in the future.

Co-authorship network of co-authors of Nadia Akawi

This figure shows the co-authorship network connecting the top 25 collaborators of Nadia Akawi. A scholar is included among the top collaborators of Nadia Akawi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nadia Akawi. Nadia Akawi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Al‐Saffar, Muna, et al.. (2025). Metabolic perturbations in cardiomyopathies: implications for early diagnosis and targeted interventions. Frontiers in Cardiovascular Medicine. 12. 1616677–1616677.
2.
Yasin, Javed, et al.. (2024). Circulating ceramide levels and ratios in Emirati youth under 18 years: associations with cardiometabolic risk factors. Lipids in Health and Disease. 23(1). 93–93. 1 indexed citations
3.
Mahmeed, Wael Al, Tarek Ansari, Kornelia Zaręba, et al.. (2023). Introducing and Implementing Genetic Assessment in Cardio-Obstetrics Clinical Practice: Clinical and Genetic Workup of Patients with Cardiomyopathy. International Journal of Molecular Sciences. 24(11). 9119–9119.
4.
Carena, Maria Cristina, Ileana Badi, Murray Polkinghorne, et al.. (2023). Role of Human Epicardial Adipose Tissue–Derived miR-92a-3p in Myocardial Redox State. Journal of the American College of Cardiology. 82(4). 317–332. 14 indexed citations
5.
Akawi, Nadia, Aisha Al‐Shamsi, Amanat Ali, et al.. (2022). Bi‐allelic null variant in matrix metalloproteinase‐15, causes congenital cardiac defect, cholestasis jaundice, and failure to thrive. Clinical Genetics. 101(4). 403–410. 4 indexed citations
6.
Akawi, Nadia, et al.. (2022). Endoglin Wild Type and Variants Associated With Hereditary Hemorrhagic Telangiectasia Type 1 Undergo Distinct Cellular Degradation Pathways. Frontiers in Molecular Biosciences. 9. 828199–828199. 7 indexed citations
7.
Ali, Bassam R., et al.. (2022). Role of Ceramides in the Molecular Pathogenesis and Potential Therapeutic Strategies of Cardiometabolic Diseases: What we Know so Far. Frontiers in Cell and Developmental Biology. 9. 816301–816301. 19 indexed citations
8.
Kondo, Hidekazu, Ioannis Akoumianakis, Nadia Akawi, et al.. (2019). P6267Novel direct effects of SGLT2 inhibitor, Canagliflozin, on myocardial redox state in humans. European Heart Journal. 40(Supplement_1). 1 indexed citations
9.
Rayner, Jennifer J., Inès Abdesselam, Mark A. Peterzan, et al.. (2018). Very low calorie diets are associated with transient ventricular impairment before reversal of diastolic dysfunction in obesity. International Journal of Obesity. 43(12). 2536–2544. 14 indexed citations
10.
Ben‐Salem, Salma, Nara L. M. Sobreira, Angeline M. Lyon, et al.. (2017). Defect in phosphoinositide signalling through a homozygous variant in PLCB3 causes a new form of spondylometaphyseal dysplasia with corneal dystrophy. Journal of Medical Genetics. 55(2). 122–130. 7 indexed citations
11.
Moselhy, Hamdy F., Valsamma Eapen, Nadia Akawi, et al.. (2015). Secondary association of PDLIM5 with paranoid schizophrenia in Emirati patients. Meta Gene. 5. 135–139. 5 indexed citations
12.
Akawi, Nadia, Fatma Al‐Jasmi, Aisha Al‐Shamsi, Bassam R. Ali, & Lihadh Al‐Gazali. (2013). LINS, a modulator of the WNT signaling pathway, is involved in human cognition. Orphanet Journal of Rare Diseases. 8(1). 87–87. 17 indexed citations
13.
Akawi, Nadia, Bassam R. Ali, & Lihadh Al‐Gazali. (2012). A novel mutation in PRG4 gene underlying camptodactyly‐arthropathy‐coxa vara‐pericarditis syndrome with the possible expansion of the phenotype to include congenital cataract. Birth Defects Research Part A Clinical and Molecular Teratology. 94(7). 553–556. 15 indexed citations
14.
Hume, Alistair N., et al.. (2012). Retention in the endoplasmic reticulum is the underlying mechanism of some hereditary haemorrhagic telangiectasia type 2 ALK1 missense mutations. Molecular and Cellular Biochemistry. 373(1-2). 247–257. 22 indexed citations
15.
Ali, Bassam R., et al.. (2011). Identification of New Alleles and the Determination of Alleles and Genotypes Frequencies at the CYP2D6 Gene in Emiratis. PLoS ONE. 6(12). e28943–e28943. 30 indexed citations
16.
Sonnevend, Ágnes, et al.. (2011). Lack of correlation between the 257C-to-T mutation in thegyrAgene and clinical severity of Campylobacter jejuni infection in a region of high incidence of ciprofloxacin resistance. Scandinavian Journal of Infectious Diseases. 43(11-12). 905–911. 5 indexed citations
17.
18.
Akawi, Nadia, et al.. (2010). Highly Sensitive Multiplex RT-PCR System for the Detection of all Common BCR-ABL Transcripts Associated with Different Leukemias. Jordan Medical Journal. 40(2).
19.
Mochida, Ganeshwaran H., Vijay Ganesh, Jillian M. Felie, et al.. (2010). A Homozygous Mutation in the Tight-Junction Protein JAM3 Causes Hemorrhagic Destruction of the Brain, Subependymal Calcification, and Congenital Cataracts. The American Journal of Human Genetics. 87(6). 882–889. 64 indexed citations
20.
Ali, Bassam R., et al.. (2010). Molecular and clinical analysis of Ellis-van Creveld syndrome in the United Arab Emirates. BMC Medical Genetics. 11(1). 33–33. 19 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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