Muyao Wu

409 total citations
26 papers, 280 citations indexed

About

Muyao Wu is a scholar working on Molecular Biology, Neurology and Cancer Research. According to data from OpenAlex, Muyao Wu has authored 26 papers receiving a total of 280 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 7 papers in Neurology and 5 papers in Cancer Research. Recurrent topics in Muyao Wu's work include Mitochondrial Function and Pathology (3 papers), Cancer, Lipids, and Metabolism (3 papers) and Neuroinflammation and Neurodegeneration Mechanisms (3 papers). Muyao Wu is often cited by papers focused on Mitochondrial Function and Pathology (3 papers), Cancer, Lipids, and Metabolism (3 papers) and Neuroinflammation and Neurodegeneration Mechanisms (3 papers). Muyao Wu collaborates with scholars based in China and United States. Muyao Wu's co-authors include Baoqi Dang, Gang Chen, Di Li, Lian Xue, Shujun Fu, Fan Gao, Chanjuan Zheng, Xiyun Deng, Jinchao Shen and Jianyu Zhu and has published in prestigious journals such as SHILAP Revista de lepidopterología, Brain Research and Biochemical and Biophysical Research Communications.

In The Last Decade

Muyao Wu

25 papers receiving 278 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Muyao Wu China 11 130 71 55 41 40 26 280
Shiyu Meng China 8 156 1.2× 66 0.9× 70 1.3× 40 1.0× 33 0.8× 15 383
Claudio Derada Troletti Netherlands 7 147 1.1× 88 1.2× 72 1.3× 61 1.5× 31 0.8× 7 333
Lili Xie China 12 168 1.3× 66 0.9× 34 0.6× 47 1.1× 40 1.0× 28 366
Lei Xia China 9 176 1.4× 86 1.2× 68 1.2× 34 0.8× 31 0.8× 15 386
Jiejie Zhuang China 10 177 1.4× 38 0.5× 40 0.7× 30 0.7× 28 0.7× 23 338
Xiaowei Fei China 11 137 1.1× 66 0.9× 53 1.0× 52 1.3× 18 0.5× 28 309
Woosoung Choi South Korea 7 139 1.1× 34 0.5× 42 0.8× 41 1.0× 23 0.6× 13 275
Xianming Zhang China 10 166 1.3× 80 1.1× 44 0.8× 65 1.6× 28 0.7× 16 350
Zhenjie Zhu China 12 144 1.1× 76 1.1× 160 2.9× 48 1.2× 41 1.0× 21 402

Countries citing papers authored by Muyao Wu

Since Specialization
Citations

This map shows the geographic impact of Muyao Wu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Muyao Wu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Muyao Wu more than expected).

Fields of papers citing papers by Muyao Wu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Muyao Wu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Muyao Wu. The network helps show where Muyao Wu may publish in the future.

Co-authorship network of co-authors of Muyao Wu

This figure shows the co-authorship network connecting the top 25 collaborators of Muyao Wu. A scholar is included among the top collaborators of Muyao Wu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Muyao Wu. Muyao Wu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wu, Muyao, et al.. (2025). Research developments in the neurovascular unit and the blood‑brain barrier (Review). Biomedical Reports. 22(5). 1–14. 7 indexed citations
2.
3.
Wu, Muyao, et al.. (2024). Enriched environment may improve secondary brain injury after traumatic brain injury by regulating the TLR2/NF-κB signaling pathway. SHILAP Revista de lepidopterología. 16. 2699828800–2699828800. 2 indexed citations
4.
Wang, Chaoyu, et al.. (2024). Tetramethylpyrazine protects mitochondrial function by up-regulation of TFAM and inhibition of neuronal apoptosis in a rat model of surgical brain injury.. SHILAP Revista de lepidopterología. 27(3). 352–359. 2 indexed citations
5.
Wu, Muyao, et al.. (2024). Up-regulation of BMAL1 by epigallocatechin-3-gallate improves neurological damage in SBI rats. Brain Research Bulletin. 215. 111033–111033.
6.
Zhang, Fengyi, Xiangyu Yan, Muyao Wu, et al.. (2023). Modulating Lineage Specification in Stem Cell Differentiation via Bioelectrical Stimulation Intensity Matching. Advanced Materials Interfaces. 11(5). 3 indexed citations
7.
Zhu, Jianyu, Muyao Wu, Lian Xue, et al.. (2023). Amino acids and their roles in tumor immunotherapy of breast cancer. The Journal of Gene Medicine. 26(1). e3647–e3647. 2 indexed citations
8.
Zheng, Chanjuan, Muyao Wu, Lian Xue, et al.. (2023). Neutrophils in triple-negative breast cancer: an underestimated player with increasingly recognized importance. Breast Cancer Research. 25(1). 88–88. 30 indexed citations
9.
Gu, Haiping, et al.. (2023). RGFP966 exerts neuroprotective effect via HDAC3/Nrf2 pathway after surgical brain injury in rats. Heliyon. 9(7). e18160–e18160. 9 indexed citations
10.
Wu, Muyao, Chaoyu Wang, Yaqian Huang, et al.. (2023). Potential mechanism of TMEM2/CD44 in endoplasmic reticulum stress‑induced neuronal apoptosis in a rat model of traumatic brain injury. International Journal of Molecular Medicine. 52(6). 8 indexed citations
11.
Chen, Sisi, Ying Li, Muyao Wu, et al.. (2022). Nucleolar and Coiled-Body Phosphoprotein 1 Is Associated With Stemness and Represents a Potential Therapeutic Target in Triple-Negative Breast Cancer. Frontiers in Oncology. 12. 731528–731528. 8 indexed citations
12.
Xue, Lian, Ying Li, Muyao Wu, et al.. (2022). Hexokinase 2 Is a Pivot for Lovastatin-induced Glycolysis-to-Autophagy Reprogramming in Triple-Negative Breast Cancer Cells. Journal of Cancer. 13(12). 3368–3377. 7 indexed citations
13.
Hu, Zhe-Yu, Chanjuan Zheng, Jianbo Yang, et al.. (2022). Co-Expression and Combined Prognostic Value of CSPG4 and PDL1 in TP53-Aberrant Triple-Negative Breast Cancer. Frontiers in Oncology. 12. 804466–804466. 12 indexed citations
14.
Wu, Muyao, et al.. (2022). Inhibition of LRRK2-Rab10 Pathway Improves Secondary Brain Injury After Surgical Brain Injury in Rats. Frontiers in Surgery. 8. 749310–749310. 4 indexed citations
15.
16.
Wu, Muyao, et al.. (2022). VEGF regulates the blood‑brain barrier through MMP‑9 in a rat model of traumatic brain injury. Experimental and Therapeutic Medicine. 24(6). 728–728. 20 indexed citations
17.
Li, Ying, Jianyu Zhu, Chanjuan Zheng, et al.. (2021). Lovastatin enhances chemosensitivity of paclitaxel-resistant prostate cancer cells through inhibition of CYP2C8. Biochemical and Biophysical Research Communications. 589. 85–91. 16 indexed citations
18.
Wu, Muyao, et al.. (2021). Inhibition of the NKCC1/NF-κB Signaling Pathway Decreases Inflammation and Improves Brain Edema and Nerve Cell Apoptosis in an SBI Rat Model. Frontiers in Molecular Neuroscience. 14. 641993–641993. 22 indexed citations
19.
Wu, Muyao, Rong Gao, Baoqi Dang, & Gang Chen. (2020). The Blood Component Iron Causes Neuronal Apoptosis Following Intracerebral Hemorrhage via the PERK Pathway. Frontiers in Neurology. 11. 588548–588548. 6 indexed citations
20.
Wu, Muyao, Fan Gao, Xiaomei Yang, et al.. (2019). Matrix metalloproteinase-9 regulates the blood brain barrier via the hedgehog pathway in a rat model of traumatic brain injury. Brain Research. 1727. 146553–146553. 33 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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