Morten Løbner

424 total citations
9 papers, 347 citations indexed

About

Morten Løbner is a scholar working on Immunology, Neurology and Molecular Biology. According to data from OpenAlex, Morten Løbner has authored 9 papers receiving a total of 347 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Immunology, 3 papers in Neurology and 2 papers in Molecular Biology. Recurrent topics in Morten Løbner's work include Immune Response and Inflammation (4 papers), Neuroinflammation and Neurodegeneration Mechanisms (3 papers) and Immune cells in cancer (2 papers). Morten Løbner is often cited by papers focused on Immune Response and Inflammation (4 papers), Neuroinflammation and Neurodegeneration Mechanisms (3 papers) and Immune cells in cancer (2 papers). Morten Løbner collaborates with scholars based in Denmark, Austria and Canada. Morten Løbner's co-authors include Trevor Owens, Oriane Cédile, Agnieszka Włodarczyk, Claus Henrik Nielsen, Reza Khorooshi, Klaus Bendtzen, Henrik Toft‐Hansen, Rune Larsen, Jason M. Millward and Rachel D. Wheeler and has published in prestigious journals such as The Journal of Immunology, Journal of Periodontology and Brain Behavior and Immunity.

In The Last Decade

Morten Løbner

9 papers receiving 338 citations

Peers

Morten Løbner

IMMUN

NEURO

MB

PHYSI

PFM

Minseo Lee South Korea

Xiaoxiao Fu China

Carolina Manganeli Polonio Brazil

Chelsea King United States

Barbara Gisevius Germany

Phillip K. West Australia

Thuong Manh Le Japan

C.J. Fish United States

Maria Laura Fois Italy

Mikhail Melnikov Russia

Minseo Lee South Korea

Morten Løbner

113 ×0.7

IMMUN

263 ×1.9

NEURO

173 ×2.0

MB

45 ×1.4

PHYSI

25 ×0.9

PFM

Citations per year, relative to Morten Løbner Morten Løbner (= 1×) peers Minseo Lee

Countries citing papers authored by Morten Løbner

Since Specialization

Citations

This map shows the geographic impact of Morten Løbner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Morten Løbner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Morten Løbner more than expected).

Fields of papers citing papers by Morten Løbner

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Morten Løbner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Morten Løbner. The network helps show where Morten Løbner may publish in the future.

Co-authorship network of co-authors of Morten Løbner

This figure shows the co-authorship network connecting the top 25 collaborators of Morten Løbner. A scholar is included among the top collaborators of Morten Løbner based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Morten Løbner. Morten Løbner is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

1.

Cédile, Oriane, Morten Løbner, Henrik Toft‐Hansen, et al.. (2014). Thymic CCL2 influences induction of T-cell tolerance. Journal of Autoimmunity. 55. 73–85. 21 indexed citations

2.

Włodarczyk, Agnieszka, Morten Løbner, Oriane Cédile, & Trevor Owens. (2014). Comparison of microglia and infiltrating CD11c+ cells as antigen presenting cells for T cell proliferation and cytokine response. Journal of Neuroinflammation. 11(1). 57–57. 116 indexed citations

3.

Khorooshi, Reza, Francesca Ruffini, Morten Løbner, et al.. (2012). Lentiviral-mediated administration of IL-25 in the CNS induces alternative activation of microglia. Gene Therapy. 20(5). 487–496. 20 indexed citations

4.

Salem, Mohammad, et al.. (2011). Interferon regulatory factor-7 modulates experimental autoimmune encephalomyelitis in mice. Journal of Neuroinflammation. 8(1). 181–181. 33 indexed citations

5.

Holm, Thomas Hellesøe, et al.. (2010). Angiotensin II Type 1 receptor (AT1) signaling in astrocytes regulates synaptic degeneration-induced leukocyte entry to the central nervous system. Brain Behavior and Immunity. 25(5). 897–904. 46 indexed citations

6.

Millward, Jason M., Morten Løbner, Rachel D. Wheeler, & Trevor Owens. (2010). Inflammation in the Central Nervous System and Th17 Responses Are Inhibited by IFN-γ–Induced IL-18 Binding Protein. The Journal of Immunology. 185(4). 2458–2466. 38 indexed citations

7.

Løbner, Morten, et al.. (2009). Decreased Interleukin‐2 Responses to Fusobacterium nucleatum and Porphyromonas gingivalis in Generalized Aggressive Periodontitis. Journal of Periodontology. 80(5). 800–807. 14 indexed citations

8.

Løbner, Morten, R. G. Q. Leslie, Wolfgang M. Prodinger, & Claus Henrik Nielsen. (2009). Spontaneous complement activation on human B cells results in localized membrane depolarization and the clustering of complement receptor type 2 and C3 fragments. Immunology. 128(1pt2). 8 indexed citations

9.

Løbner, Morten, et al.. (2008). Enhancement of Human Adaptive Immune Responses by Administration of a High-Molecular-Weight Polysaccharide Extract from the Cyanobacterium Arthrospira platensis. Journal of Medicinal Food. 11(2). 313–322. 51 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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