Mo‐Jun Lin

1.6k total citations
35 papers, 1.3k citations indexed

About

Mo‐Jun Lin is a scholar working on Molecular Biology, Pulmonary and Respiratory Medicine and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Mo‐Jun Lin has authored 35 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 17 papers in Pulmonary and Respiratory Medicine and 10 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Mo‐Jun Lin's work include Pulmonary Hypertension Research and Treatments (14 papers), Ion Channels and Receptors (10 papers) and Ion channel regulation and function (9 papers). Mo‐Jun Lin is often cited by papers focused on Pulmonary Hypertension Research and Treatments (14 papers), Ion Channels and Receptors (10 papers) and Ion channel regulation and function (9 papers). Mo‐Jun Lin collaborates with scholars based in China and United States. Mo‐Jun Lin's co-authors include James S.K. Sham, Xiao-Ru Yang, Kay‐Pong Yip, Wei-Min Zhang, Gph Leung, Chung‐Ming Tse, Long‐Xin Gui, Zhi‐Juan Wu, Ruixing Wang and Larissa A. Shimoda and has published in prestigious journals such as Circulation Research, Hypertension and Journal of Pharmacology and Experimental Therapeutics.

In The Last Decade

Mo‐Jun Lin

34 papers receiving 1.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mo‐Jun Lin China 18 611 503 432 319 239 35 1.3k
Xiao-Ru Yang United States 11 476 0.8× 514 1.0× 362 0.8× 284 0.9× 179 0.7× 11 1.1k
Jessica Sabourin France 18 432 0.7× 391 0.8× 221 0.5× 105 0.3× 287 1.2× 30 927
Helga Vitzthum Germany 16 723 1.2× 280 0.6× 180 0.4× 135 0.4× 190 0.8× 25 1.8k
Jonathan M. Bisaillon United States 7 492 0.8× 808 1.6× 91 0.2× 171 0.5× 123 0.5× 10 1.2k
Florian Thilo Germany 16 283 0.5× 457 0.9× 48 0.1× 200 0.6× 90 0.4× 24 986
Willm‐Thomas Heyken Germany 7 399 0.7× 359 0.7× 60 0.1× 332 1.0× 180 0.8× 8 839
Marie Demion France 17 734 1.2× 681 1.4× 51 0.1× 128 0.4× 359 1.5× 32 1.4k
Søren Tullin Denmark 11 313 0.5× 196 0.4× 50 0.1× 457 1.4× 124 0.5× 11 1.2k
Birgit Salanova Germany 8 330 0.5× 484 1.0× 67 0.2× 126 0.4× 79 0.3× 8 826
Valério Farfariello Italy 22 548 0.9× 530 1.1× 82 0.2× 184 0.6× 22 0.1× 37 1.3k

Countries citing papers authored by Mo‐Jun Lin

Since Specialization
Citations

This map shows the geographic impact of Mo‐Jun Lin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mo‐Jun Lin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mo‐Jun Lin more than expected).

Fields of papers citing papers by Mo‐Jun Lin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mo‐Jun Lin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mo‐Jun Lin. The network helps show where Mo‐Jun Lin may publish in the future.

Co-authorship network of co-authors of Mo‐Jun Lin

This figure shows the co-authorship network connecting the top 25 collaborators of Mo‐Jun Lin. A scholar is included among the top collaborators of Mo‐Jun Lin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mo‐Jun Lin. Mo‐Jun Lin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Lin, Mo‐Jun, et al.. (2025). Fraxinus mandshurica galacturonosyltransferase 1 and 12 play negative roles in cadmium tolerance via cell wall remodeling. International Journal of Biological Macromolecules. 306(Pt 2). 141510–141510. 2 indexed citations
2.
Xu, Lixia, Wen-Jing Yi, Junwei Zhang, et al.. (2025). Ginsenoside Rb1 ameliorates post-doxorubicin treatment myocardial hypertrophy via CaN/NFATc4/GATA4. Journal of Ginseng Research. 49(5). 585–593. 1 indexed citations
3.
Xu, Lixia, Ruixing Wang, G. F. Yi, et al.. (2023). TRPC6 promotes daunorubicin-induced mitochondrial fission and cell death in rat cardiomyocytes with the involvement of ERK1/2-DRP1 activation. Toxicology and Applied Pharmacology. 470. 116547–116547. 7 indexed citations
5.
Jiang, Yanan, et al.. (2022). Attenuating effect of magnesium on pulmonary arterial calcification in rodent models of pulmonary hypertension. Journal of Hypertension. 40(10). 1979–1993. 3 indexed citations
6.
Li, Li, et al.. (2021). TRPC3 promotes tumorigenesis of gastric cancer via the CNB2/GSK3β/NFATc2 signaling pathway. Cancer Letters. 519. 211–225. 27 indexed citations
7.
Yan, Furong, et al.. (2019). Increased caveolin‐1 expression enhances the receptor‐operated Ca2+ entry in the aorta of two‐kidney, one‐clip hypertensive rats. Experimental Physiology. 104(6). 932–945. 5 indexed citations
8.
Zhang, Li, Shaokun Chen, Xixi Zeng, et al.. (2019). Revealing the pathogenic changes of PAH based on multiomics characteristics. Journal of Translational Medicine. 17(1). 231–231. 22 indexed citations
10.
Sun, Hui, et al.. (2016). Conformation of ryanodine receptor-2 gates store-operated calcium entry in rat pulmonary arterial myocytes. Cardiovascular Research. 111(1). 94–104. 15 indexed citations
11.
Yan, Furong, et al.. (2016). Alterations in Caveolin-1 Expression and Receptor-Operated Ca2+ Entry in the Aortas of Rats with Pulmonary Hypertension. Cellular Physiology and Biochemistry. 39(2). 438–452. 12 indexed citations
13.
Liu, Qing, et al.. (2013). Down-Regulation of TRPM8 in Pulmonary Arteries of Pulmonary Hypertensive Rats. Cellular Physiology and Biochemistry. 31(6). 892–904. 24 indexed citations
14.
Wu, Zhi‐Juan, et al.. (2013). Sodium Ferulate Protects Against Daunorubicin-induced Cardiotoxicity by Inhibition of Mitochondrial Apoptosis in Juvenile Rats. Journal of Cardiovascular Pharmacology. 63(4). 360–368. 9 indexed citations
15.
Yang, Xiao-Ru, Mo‐Jun Lin, & James S.K. Sham. (2009). Physiological Functions of Transient Receptor Potential Channels in Pulmonary Arterial Smooth Muscle Cells. Advances in experimental medicine and biology. 661. 109–122. 31 indexed citations
16.
Lin, Mo‐Jun, et al.. (2007). Hydrogen peroxide-induced Ca2+mobilization in pulmonary arterial smooth muscle cells. American Journal of Physiology-Lung Cellular and Molecular Physiology. 292(6). L1598–L1608. 71 indexed citations
17.
Yang, Xiao-Ru, et al.. (2006). Functional expression of transient receptor potential melastatin- and vanilloid-related channels in pulmonary arterial and aortic smooth muscle. American Journal of Physiology-Lung Cellular and Molecular Physiology. 290(6). L1267–L1276. 230 indexed citations
18.
Zhang, Wei-Min, Mo‐Jun Lin, & James S.K. Sham. (2004). Endothelin-1 and IP3 Induced Ca2+ Sparks in Pulmonary Arterial Smooth Muscle Cells. Journal of Cardiovascular Pharmacology. 44(Supplement 1). S121–S124. 15 indexed citations
19.
Zhang, Wei-Min, et al.. (2003). ET-1 activates Ca2+sparks in PASMC: local Ca2+signaling between inositol trisphosphate and ryanodine receptors. American Journal of Physiology-Lung Cellular and Molecular Physiology. 285(3). L680–L690. 75 indexed citations
20.
Lin, Mo‐Jun, Angus C. Nairn, & Sandra E. Guggino. (1992). cGMP-dependent protein kinase regulation of a chloride channel in T84 cells. American Journal of Physiology-Cell Physiology. 262(5). C1304–C1312. 38 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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