Mohammed H. Al-Wadei

599 total citations
17 papers, 518 citations indexed

About

Mohammed H. Al-Wadei is a scholar working on Psychiatry and Mental health, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Mohammed H. Al-Wadei has authored 17 papers receiving a total of 518 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Psychiatry and Mental health, 10 papers in Molecular Biology and 6 papers in Cellular and Molecular Neuroscience. Recurrent topics in Mohammed H. Al-Wadei's work include Cancer, Stress, Anesthesia, and Immune Response (16 papers), Nicotinic Acetylcholine Receptors Study (10 papers) and Neuropeptides and Animal Physiology (6 papers). Mohammed H. Al-Wadei is often cited by papers focused on Cancer, Stress, Anesthesia, and Immune Response (16 papers), Nicotinic Acetylcholine Receptors Study (10 papers) and Neuropeptides and Animal Physiology (6 papers). Mohammed H. Al-Wadei collaborates with scholars based in United States and Yemen. Mohammed H. Al-Wadei's co-authors include Hussein A.N. Al-Wadei, Hildegard M. Schuller, Mohammad Fahad Ullah, Jheelam Banerjee, Thomas Masi, Jiangang Chen and Paul Terry and has published in prestigious journals such as PLoS ONE, Cancer Research and European Journal of Cancer.

In The Last Decade

Mohammed H. Al-Wadei

17 papers receiving 505 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mohammed H. Al-Wadei United States 13 296 240 125 111 43 17 518
Juli Schlichtiger Germany 8 230 0.8× 138 0.6× 172 1.4× 234 2.1× 121 2.8× 9 577
Amalia Merelli Argentina 15 72 0.2× 201 0.8× 78 0.6× 78 0.7× 97 2.3× 19 579
Anchen Guo China 12 47 0.2× 218 0.9× 84 0.7× 61 0.5× 87 2.0× 22 485
Leonardo Lisbôa da Motta Brazil 13 47 0.2× 248 1.0× 58 0.5× 38 0.3× 19 0.4× 21 481
Megan K. Herbert Netherlands 14 42 0.1× 303 1.3× 122 1.0× 68 0.6× 86 2.0× 22 775
Débora G. Flores Brazil 10 30 0.1× 120 0.5× 89 0.7× 55 0.5× 18 0.4× 11 372
Emma L. B. Soldner United States 6 75 0.3× 103 0.4× 75 0.6× 102 0.9× 181 4.2× 10 479
Bjoern Bauer United States 10 49 0.2× 181 0.8× 58 0.5× 194 1.7× 145 3.4× 16 532
Deok-Soo Son United States 16 25 0.1× 252 1.1× 186 1.5× 65 0.6× 82 1.9× 24 800
Fabien Dutheil France 7 40 0.1× 154 0.6× 67 0.5× 258 2.3× 95 2.2× 8 653

Countries citing papers authored by Mohammed H. Al-Wadei

Since Specialization
Citations

This map shows the geographic impact of Mohammed H. Al-Wadei's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mohammed H. Al-Wadei with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mohammed H. Al-Wadei more than expected).

Fields of papers citing papers by Mohammed H. Al-Wadei

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mohammed H. Al-Wadei. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mohammed H. Al-Wadei. The network helps show where Mohammed H. Al-Wadei may publish in the future.

Co-authorship network of co-authors of Mohammed H. Al-Wadei

This figure shows the co-authorship network connecting the top 25 collaborators of Mohammed H. Al-Wadei. A scholar is included among the top collaborators of Mohammed H. Al-Wadei based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mohammed H. Al-Wadei. Mohammed H. Al-Wadei is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Banerjee, Jheelam, et al.. (2016). Prevention of pancreatic cancer in a hamster model by cAMP decrease. Oncotarget. 7(28). 44430–44441. 10 indexed citations
2.
Banerjee, Jheelam, et al.. (2016). Abstract 823: Prevention of pancreatic cancer by cAMP control. Cancer Research. 76(14_Supplement). 823–823. 1 indexed citations
3.
Al-Wadei, Mohammed H., Jheelam Banerjee, Hussein A.N. Al-Wadei, & Hildegard M. Schuller. (2015). Nicotine induces self-renewal of pancreatic cancer stem cells via neurotransmitter-driven activation of sonic hedgehog signalling. European Journal of Cancer. 52. 188–196. 34 indexed citations
4.
Chen, Jiangang, et al.. (2014). Hydraulic Fracturing: Paving the Way for a Sustainable Future?. Journal of Environmental and Public Health. 2014. 1–10. 19 indexed citations
5.
6.
Banerjee, Jheelam, Mohammed H. Al-Wadei, Hussein A.N. Al-Wadei, & Hildegard M. Schuller. (2013). Abstract 2191: GABA but not baclofen prevents gemcitabine resistance induced by low dose nicotine in pancreatic cancer xenografts.. Cancer Research. 73(8_Supplement). 2191–2191. 1 indexed citations
7.
Al-Wadei, Mohammed H., Hussein A.N. Al-Wadei, & Hildegard M. Schuller. (2012). Effects of chronic nicotine on the autocrine regulation of pancreatic cancer cells and pancreatic duct epithelial cells by stimulatory and inhibitory neurotransmitters. Carcinogenesis. 33(9). 1745–1753. 30 indexed citations
8.
Al-Wadei, Hussein A.N., Mohammad Fahad Ullah, & Mohammed H. Al-Wadei. (2012). Intercepting neoplastic progression in lung malignancies via the beta adrenergic (β-AR) pathway: Implications for anti-cancer drug targets. Pharmacological Research. 66(1). 33–40. 18 indexed citations
9.
Al-Wadei, Hussein A.N., Mohammed H. Al-Wadei, & Hildegard M. Schuller. (2012). Cooperative Regulation of Non-Small Cell Lung Carcinoma by Nicotinic and Beta-Adrenergic Receptors: A Novel Target for Intervention. PLoS ONE. 7(1). e29915–e29915. 69 indexed citations
10.
Al-Wadei, Hussein A.N., Mohammed H. Al-Wadei, Mohammad Fahad Ullah, & Hildegard M. Schuller. (2012). Gamma-Amino Butyric Acid Inhibits the Nicotine-Imposed Stimulatory Challenge in Xenograft Models of Non-Small Cell Lung Carcinoma. Current Cancer Drug Targets. 12(2). 97–106. 20 indexed citations
11.
Al-Wadei, Hussein A.N., Mohammed H. Al-Wadei, Mohammad Fahad Ullah, & Hildegard M. Schuller. (2012). Celecoxib and GABA Cooperatively Prevent the Progression of Pancreatic Cancer In Vitro and in Xenograft Models of Stress-Free and Stress-Exposed Mice. PLoS ONE. 7(8). e43376–e43376. 40 indexed citations
12.
Al-Wadei, Mohammed H., Hussein A.N. Al-Wadei, & Hildegard M. Schuller. (2012). Gamma-amino Butyric Acid (GABA) Prevents the Induction of Nicotinic Receptor–Regulated Signaling by Chronic Ethanol in Pancreatic Cancer Cells and Normal Duct Epithelia. Cancer Prevention Research. 6(2). 139–148. 12 indexed citations
13.
Al-Wadei, Mohammed H., Hussein A.N. Al-Wadei, & Hildegard M. Schuller. (2011). Pancreatic Cancer Cells and Normal Pancreatic Duct Epithelial Cells Express an Autocrine Catecholamine Loop that Is Activated by Nicotinic Acetylcholine Receptors α3, α5, and α7. Molecular Cancer Research. 10(2). 239–249. 77 indexed citations
14.
Al-Wadei, Hussein A.N., Mohammad Fahad Ullah, & Mohammed H. Al-Wadei. (2011). GABA (γ‐aminobutyric acid), a non‐protein amino acid counters the β‐adrenergic cascade‐activated oncogenic signaling in pancreatic cancer: A review of experimental evidence. Molecular Nutrition & Food Research. 55(12). 1745–1758. 33 indexed citations
15.
16.
Al-Wadei, Hussein A.N., Mohammed H. Al-Wadei, Thomas Masi, & Hildegard M. Schuller. (2009). Chronic exposure to estrogen and the tobacco carcinogen NNK cooperatively modulates nicotinic receptors in small airway epithelial cells. Lung Cancer. 69(1). 33–39. 36 indexed citations
17.
Al-Wadei, Hussein A.N., Mohammed H. Al-Wadei, & Hildegard M. Schuller. (2009). Prevention of pancreatic cancer by the beta-blocker propranolol. Anti-Cancer Drugs. 20(6). 477–482. 100 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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