Moaz Ahmad

440 total citations
21 papers, 299 citations indexed

About

Moaz Ahmad is a scholar working on Molecular Biology, Public Health, Environmental and Occupational Health and Infectious Diseases. According to data from OpenAlex, Moaz Ahmad has authored 21 papers receiving a total of 299 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 5 papers in Public Health, Environmental and Occupational Health and 4 papers in Infectious Diseases. Recurrent topics in Moaz Ahmad's work include RNA and protein synthesis mechanisms (6 papers), DNA Repair Mechanisms (5 papers) and Malaria Research and Control (4 papers). Moaz Ahmad is often cited by papers focused on RNA and protein synthesis mechanisms (6 papers), DNA Repair Mechanisms (5 papers) and Malaria Research and Control (4 papers). Moaz Ahmad collaborates with scholars based in India, United States and Italy. Moaz Ahmad's co-authors include Renu Tuteja, Mohammed Tarique, Farhat Afrin, Shailja Singh, Indu S. Ambudkar, Hwei Ling Ong, Narendra Tuteja, Mst. Sufara Akhter Banu, Javier Manzella‐Lapeira and Joseph Brzostowski and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of the American College of Cardiology and PLoS ONE.

In The Last Decade

Moaz Ahmad

20 papers receiving 297 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Moaz Ahmad India 13 201 114 45 33 27 21 299
Elizabeth K. K. Glennon United States 12 125 0.6× 172 1.5× 22 0.5× 44 1.3× 6 0.2× 19 387
N. Parmar India 12 113 0.6× 191 1.7× 34 0.8× 33 1.0× 13 0.5× 22 506
Antonino Missineo Italy 10 180 0.9× 81 0.7× 53 1.2× 11 0.3× 4 0.1× 13 316
Shuling Lin China 11 237 1.2× 22 0.2× 49 1.1× 20 0.6× 7 0.3× 15 411
Donald D. Lorimer United States 12 361 1.8× 22 0.2× 42 0.9× 60 1.8× 3 0.1× 40 500
Mary Kate Alexander United States 10 367 1.8× 19 0.2× 53 1.2× 16 0.5× 6 0.2× 13 474
Sunanda Bhattacharyya India 13 252 1.3× 101 0.9× 44 1.0× 17 0.5× 28 338
Eva Hernández France 8 127 0.6× 74 0.6× 32 0.7× 39 1.2× 10 368
Xiaoshan Jiang United States 13 227 1.1× 87 0.8× 176 3.9× 52 1.6× 1 0.0× 18 495
Abhinav Kaushik United States 11 111 0.6× 63 0.6× 24 0.5× 9 0.3× 2 0.1× 31 289

Countries citing papers authored by Moaz Ahmad

Since Specialization
Citations

This map shows the geographic impact of Moaz Ahmad's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Moaz Ahmad with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Moaz Ahmad more than expected).

Fields of papers citing papers by Moaz Ahmad

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Moaz Ahmad. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Moaz Ahmad. The network helps show where Moaz Ahmad may publish in the future.

Co-authorship network of co-authors of Moaz Ahmad

This figure shows the co-authorship network connecting the top 25 collaborators of Moaz Ahmad. A scholar is included among the top collaborators of Moaz Ahmad based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Moaz Ahmad. Moaz Ahmad is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Ahmad, Moaz, et al.. (2022). Functional communication between IP 3 R and STIM2 at subthreshold stimuli is a critical checkpoint for initiation of SOCE. Proceedings of the National Academy of Sciences. 119(3). 29 indexed citations
3.
Mirza, Hasan, Weiyi Xia, Sundeep Kumar, et al.. (2022). INPATIENT HOSPITAL MORTALITY IN SPONTANEOUS CORONARY ARTERY DISSECTION (SCAD). Journal of the American College of Cardiology. 79(9). 1169–1169.
4.
Ahmad, Moaz, et al.. (2022). STIM Proteins and Regulation of SOCE in ER-PM Junctions. Biomolecules. 12(8). 1152–1152. 11 indexed citations
5.
Kidwai, Fahad, Konstantinia Almpani, Priyam Jani, et al.. (2021). Quantitative Craniofacial Analysis and Generation of Human Induced Pluripotent Stem Cells for Muenke Syndrome: A Case Report. Journal of Developmental Biology. 9(4). 39–39. 4 indexed citations
6.
7.
Tarique, Mohammed, et al.. (2017). Genome Wide In silico Analysis of the Mismatch Repair Components of Plasmodium falciparum and Their Comparison with Human Host. Frontiers in Microbiology. 8. 130–130. 15 indexed citations
8.
Tarique, Mohammed, et al.. (2015). Plasmodium falciparum Werner homologue is a nuclear protein and its biochemical activities reside in the N-terminal region. PROTOPLASMA. 253(1). 45–60. 12 indexed citations
9.
Ahmad, Moaz & Renu Tuteja. (2014). Emerging importance of mismatch repair components including UvrD helicase and their cross-talk with the development of drug resistance in malaria parasite. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 770. 54–60. 8 indexed citations
10.
Ahmad, Moaz, Mohammed Tarique, Farhat Afrin, Narendra Tuteja, & Renu Tuteja. (2014). Identification of inhibitors of Plasmodium falciparum RuvB1 helicase using biochemical assays. PROTOPLASMA. 252(1). 117–125. 4 indexed citations
11.
Tuteja, Narendra, Mohammed Tarique, Mst. Sufara Akhter Banu, Moaz Ahmad, & Renu Tuteja. (2014). Pisum sativum p68 DEAD-box protein is ATP-dependent RNA helicase and unique bipolar DNA helicase. Plant Molecular Biology. 85(6). 639–651. 21 indexed citations
12.
Tarique, Mohammed, et al.. (2014). Plasmodium falciparum UvrD activities are downregulated by DNA-interacting compounds and its dsRNA inhibits malaria parasite growth. BMC Biochemistry. 15(1). 9–9. 15 indexed citations
13.
Tarique, Mohammed, et al.. (2013). Plasmodium falciparum DOZI, an RNA helicase interacts with eIF4E. Gene. 522(1). 46–59. 28 indexed citations
14.
Ahmad, Moaz & Renu Tuteja. (2013). Plasmodium falciparum RuvB2 translocates in 5′–3′ direction, relocalizes during schizont stage and its enzymatic activities are up regulated by RuvB3 of the same complex. Biochimica et Biophysica Acta (BBA) - Proteins and Proteomics. 1834(12). 2795–2811. 11 indexed citations
15.
Ahmad, Moaz, Farhat Afrin, & Renu Tuteja. (2013). Identification of R2TP complex ofLeishmania donovaniandPlasmodium falciparumusing genome wide in-silico analysis. Communicative & Integrative Biology. 6(6). e26005–e26005. 12 indexed citations
16.
Ahmad, Moaz, Shailja Singh, Farhat Afrin, & Renu Tuteja. (2012). Novel RuvB nuclear ATPase is specific to intraerythrocytic mitosis during schizogony of Plasmodium falciparum. Molecular and Biochemical Parasitology. 185(1). 58–65. 16 indexed citations
17.
Ahmad, Moaz & Renu Tuteja. (2012). Plasmodium falciparumRuvB proteins. Communicative & Integrative Biology. 5(4). 350–361. 16 indexed citations
18.
Ahmad, Moaz & Renu Tuteja. (2012). Plasmodium falciparum RuvB1 is an active DNA helicase and translocates in the 5′–3′ direction. Gene. 515(1). 99–109. 12 indexed citations
19.
20.
Tarique, Mohammed, et al.. (2011). Plasmodium falciparum MLH is schizont stage specific endonuclease. Molecular and Biochemical Parasitology. 181(2). 153–161. 14 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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