M.J. Bastié

554 total citations
25 papers, 466 citations indexed

About

M.J. Bastié is a scholar working on Surgery, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, M.J. Bastié has authored 25 papers receiving a total of 466 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Surgery, 11 papers in Molecular Biology and 6 papers in Cellular and Molecular Neuroscience. Recurrent topics in M.J. Bastié's work include Pancreatic function and diabetes (10 papers), Ion Transport and Channel Regulation (8 papers) and Pancreatitis Pathology and Treatment (6 papers). M.J. Bastié is often cited by papers focused on Pancreatic function and diabetes (10 papers), Ion Transport and Channel Regulation (8 papers) and Pancreatitis Pathology and Treatment (6 papers). M.J. Bastié collaborates with scholars based in France, United States and Mali. M.J. Bastié's co-authors include Nicole Vaysse, A Ribet, Lucien Pradayrol, Jean‐Pierre Pascal, Viviane Bertrand, J.L. Scemama, Nathalie Viguerie, C. Susini, M. Delvaux and D. Balas and has published in prestigious journals such as Gastroenterology, Gut and FEBS Letters.

In The Last Decade

M.J. Bastié

25 papers receiving 429 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
M.J. Bastié France 12 215 139 115 85 68 25 466
Shigeru Genka Japan 13 90 0.4× 178 1.3× 73 0.6× 106 1.2× 68 1.0× 20 529
F. Clémente France 16 288 1.3× 239 1.7× 105 0.9× 106 1.2× 130 1.9× 32 588
Y.L. Lee United States 8 219 1.0× 100 0.7× 78 0.7× 35 0.4× 68 1.0× 9 533
Numa R. Gottardi-Littell United States 10 236 1.1× 198 1.4× 66 0.6× 25 0.3× 72 1.1× 15 505
Yoshimi Mori Japan 11 250 1.2× 105 0.8× 82 0.7× 35 0.4× 57 0.8× 12 591
Fabrice Jaffré France 11 429 2.0× 87 0.6× 114 1.0× 101 1.2× 82 1.2× 17 831
Sara Welch United States 8 411 1.9× 73 0.5× 68 0.6× 20 0.2× 39 0.6× 8 694
Chikao Iwai Japan 13 201 0.9× 89 0.6× 39 0.3× 72 0.8× 48 0.7× 15 558
Juan José Díaz-Gil Spain 17 170 0.8× 109 0.8× 124 1.1× 94 1.1× 15 0.2× 33 589
Georgina M. Nemecek United States 9 184 0.9× 65 0.5× 64 0.6× 23 0.3× 29 0.4× 12 398

Countries citing papers authored by M.J. Bastié

Since Specialization
Citations

This map shows the geographic impact of M.J. Bastié's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by M.J. Bastié with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites M.J. Bastié more than expected).

Fields of papers citing papers by M.J. Bastié

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by M.J. Bastié. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by M.J. Bastié. The network helps show where M.J. Bastié may publish in the future.

Co-authorship network of co-authors of M.J. Bastié

This figure shows the co-authorship network connecting the top 25 collaborators of M.J. Bastié. A scholar is included among the top collaborators of M.J. Bastié based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with M.J. Bastié. M.J. Bastié is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Pyronnet, Stéphane, et al.. (1998). Arachidonic-acid-selective cytosolic phospholipase A2 is involved in gastrin-induced AR4-2J-cell proliferation. International Journal of Cancer. 75(2). 239–245. 8 indexed citations
2.
Travo, Pierre, et al.. (1996). Loss of differences in mesangial cell phenotype between diabetic and normal rats: Role of culture passages. Biology of the Cell. 86(2-3). 127–133. 9 indexed citations
3.
Bertrand, Vincent, M.J. Bastié, M. Bouisson, Nicole Vaysse, & Lucien Pradayrol. (1996). AR4-2J cell line coexpresses dihydropyridine and ω-conotoxin sensitive Ca2+ channels. Cell Calcium. 19(6). 495–500. 2 indexed citations
4.
Bertrand, Viviane, M.J. Bastié, N. Vaysse, & Lucien Pradayrol. (1995). Different Implications of Ca2+i and Ca2+e in CCK/Gastrin-Induced Amylase Secretion in AR4-2J Cells. Pancreas. 11(3). 230–235. 1 indexed citations
5.
Bertrand, Viviane, M.J. Bastié, Nicole Vaysse, & Lucien Pradayrol. (1994). Inhibition of gastrin‐induced proliferation of AR4–2j cells by calcium channel antagonists. International Journal of Cancer. 56(3). 427–432. 35 indexed citations
6.
Bertrand, Viviane, et al.. (1994). Pharmacological study of gastrin-mediated amylase release in pancreatic acinar cells (AR4-2J). Regulatory Peptides. 54(2-3). 513–525. 9 indexed citations
7.
Bascands, Jean‐Loup, Christiane Pécher, M.J. Bastié, et al.. (1993). Evidence for existence of two distinct bradykinin receptors on rat mesangial cells. American Journal of Physiology-Renal Physiology. 264(3). F548–F556. 76 indexed citations
8.
Delvaux, M., et al.. (1990). Na<sup>+</sup>/H<sup>+</sup> Exchange in AR4–2J Cells: Stimulation by Growth Factors and Pancreatic Secretagogues. Digestion. 46(2). 156–161. 6 indexed citations
9.
Delvaux, M., et al.. (1990). Amiloride and analogues inhibit Na(+)-H+ exchange and cell proliferation in AR42J pancreatic cell line. American Journal of Physiology-Gastrointestinal and Liver Physiology. 259(5). G842–G849. 31 indexed citations
10.
Bastié, M.J. & John A. Williams. (1990). Gastrointestinal peptides activate Na(+)-H+ exchanger in AR42J cells by increasing its affinity for intracellular H+. American Journal of Physiology-Gastrointestinal and Liver Physiology. 258(6). G958–G966. 10 indexed citations
11.
12.
Bastié, M.J., Marlène Dufresne, M. Delvaux, Nicole Vaysse, & A Ribet. (1987). CCK-related peptides stimulation of the Na+/H+ antiport in pancreatic acinar cells leads to cytoplasmic alkalinisation.. Gut. 28(Suppl). 85–88. 3 indexed citations
13.
Dufresne, Marlène, et al.. (1985). The amiloride sensitive Na+/H+ antiport in guinea pig pancreatic acini. FEBS Letters. 187(1). 126–130. 17 indexed citations
14.
Bastié, M.J., et al.. (1985). Comparative Study of Histological and Kinetic Variations of the Digestive Mucosa and Pancreatic Parenchyma after Hypophysectomy in the Rat. Cells Tissues Organs. 124(3-4). 133–144. 3 indexed citations
15.
Bastié, M.J., D. Balas, Jacques Laval, et al.. (1982). Histological Variations of Jejunal and Ileal Mucosa on Days 8 and 15 after Hypophysectomy in Rat: Morphometrical Analysis in Light and Electron Microscopy. Cells Tissues Organs. 112(4). 321–337. 20 indexed citations
16.
Senegas‐Balas, F., et al.. (1981). Trophicité des muqueuses digestives. annales de biologie animale biochimie biophysique. 21(5B). 783–803. 1 indexed citations
17.
Ribet, A, D. Balas, M.J. Bastié, et al.. (1981). Long term effects of H2-receptor antagonists (cimetidine and ranitidine) on the human gastric and duodenal mucosa.. PubMed. 70. 155–74. 8 indexed citations
18.
Bastié, M.J., D. Balas, F. Senegas‐Balas, et al.. (1979). A Cyto-physiological Study of the G-cell Secretory Cycle in the Antrum Mucosa of the Hamster and of the Rat. Scandinavian Journal of Gastroenterology. 14(1). 35–48. 9 indexed citations
19.
Bastié, M.J., et al.. (1977). Effects of catecholamines and their inhibitors on the isolated canine pancreas. II. Dopamine.. PubMed. 72(4 Pt 1). 719–23. 21 indexed citations
20.
Bastié, M.J., et al.. (1977). Effects of Catecholamines and Their Inhibitors on the Isolated Canine Pancreas. Gastroenterology. 72(4). 719–723. 18 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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