Minghui Qin

400 total citations
11 papers, 298 citations indexed

About

Minghui Qin is a scholar working on Molecular Biology, Immunology and Cell Biology. According to data from OpenAlex, Minghui Qin has authored 11 papers receiving a total of 298 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 4 papers in Immunology and 3 papers in Cell Biology. Recurrent topics in Minghui Qin's work include Immune cells in cancer (3 papers), Cardiac Fibrosis and Remodeling (2 papers) and Cell Adhesion Molecules Research (2 papers). Minghui Qin is often cited by papers focused on Immune cells in cancer (3 papers), Cardiac Fibrosis and Remodeling (2 papers) and Cell Adhesion Molecules Research (2 papers). Minghui Qin collaborates with scholars based in United States and China. Minghui Qin's co-authors include Prediman K. Shah, Behrooz G. Sharifi, Lai Wang, Mingjie Yang, Fuqiang Li, Lei Song, Ada Yukht, Fang Tian, M. Rocı́o Sierra-Honigmann and Sebastian Wachsmann‐Hogiu and has published in prestigious journals such as Journal of the American College of Cardiology, PLoS ONE and International Journal of Molecular Sciences.

In The Last Decade

Minghui Qin

11 papers receiving 291 citations

Peers

Minghui Qin
Shan Wen United States
Johnny E. Croy United States
Alexandra Surcel United States
Minghui Qin
Citations per year, relative to Minghui Qin Minghui Qin (= 1×) peers Akiomi Nagasaka

Countries citing papers authored by Minghui Qin

Since Specialization
Citations

This map shows the geographic impact of Minghui Qin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Minghui Qin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Minghui Qin more than expected).

Fields of papers citing papers by Minghui Qin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Minghui Qin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Minghui Qin. The network helps show where Minghui Qin may publish in the future.

Co-authorship network of co-authors of Minghui Qin

This figure shows the co-authorship network connecting the top 25 collaborators of Minghui Qin. A scholar is included among the top collaborators of Minghui Qin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Minghui Qin. Minghui Qin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
1.
Feng, Jinlin, Minghui Qin, Lixia Yao, et al.. (2022). The N-terminal acetyltransferase Naa50 regulates tapetum degradation and pollen development in Arabidopsis. Plant Science. 316. 111180–111180. 4 indexed citations
2.
Liu, Chengyu, Ningning Shen, Minghui Qin, et al.. (2022). Magnaporthe oryzae Transcription Factor MoBZIP3 Regulates Appressorium Turgor Pressure Formation during Pathogenesis. International Journal of Molecular Sciences. 23(2). 881–881. 9 indexed citations
3.
Qin, Minghui, et al.. (2022). Silencing of a Cotton Actin-Binding Protein GhWLIM1C Decreases Resistance against Verticillium dahliae Infection. Plants. 11(14). 1828–1828. 4 indexed citations
4.
Xu, Rui, Yuanbao Li, Chengyu Liu, et al.. (2021). Twinfilin regulates actin assembly and Hexagonal peroxisome 1 (Hex1) localization in the pathogenesis of rice blast fungus Magnaporthe oryzae. Molecular Plant Pathology. 22(12). 1641–1655. 8 indexed citations
5.
Yang, Mingjie, Lei Song, Lai Wang, et al.. (2018). Deficiency of GATA3-Positive Macrophages Improves Cardiac Function Following Myocardial Infarction or Pressure Overload Hypertrophy. Journal of the American College of Cardiology. 72(8). 885–904. 45 indexed citations
6.
Song, Lei, Lai Wang, Fuqiang Li, et al.. (2017). Bone Marrow-Derived Tenascin-C Attenuates Cardiac Hypertrophy by Controlling Inflammation. Journal of the American College of Cardiology. 70(13). 1601–1615. 35 indexed citations
7.
Qin, Minghui, Lai Wang, Fuqiang Li, et al.. (2017). Oxidized LDL activated eosinophil polarize macrophage phenotype from M2 to M1 through activation of CD36 scavenger receptor. Atherosclerosis. 263. 82–91. 44 indexed citations
8.
Wang, Lai, Mingjie Yang, Lei Song, et al.. (2015). Splenocytes Seed Bone Marrow of Myeloablated Mice: Implication for Atherosclerosis. PLoS ONE. 10(6). e0125961–e0125961. 4 indexed citations
9.
Li, Fuqiang, Mingjie Yang, Lai Wang, et al.. (2012). Autofluorescence contributes to false-positive intracellular Foxp3 staining in macrophages: A lesson learned from flow cytometry. Journal of Immunological Methods. 386(1-2). 101–107. 46 indexed citations
10.
Sharifi, Behrooz G., Zhaohui Zeng, Lai Wang, et al.. (2006). Pleiotrophin Induces Transdifferentiation of Monocytes Into Functional Endothelial Cells. Arteriosclerosis Thrombosis and Vascular Biology. 26(6). 1273–1280. 63 indexed citations
11.
Qin, Minghui, Zhaohui Zeng, Jie Zheng, et al.. (2003). Suppression Subtractive Hybridization Identifies Distinctive Expression Markers for Coronary and Internal Mammary Arteries. Arteriosclerosis Thrombosis and Vascular Biology. 23(3). 425–433. 36 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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