Miloslav Kolaj

965 total citations
36 papers, 798 citations indexed

About

Miloslav Kolaj is a scholar working on Cellular and Molecular Neuroscience, Endocrine and Autonomic Systems and Cognitive Neuroscience. According to data from OpenAlex, Miloslav Kolaj has authored 36 papers receiving a total of 798 indexed citations (citations by other indexed papers that have themselves been cited), including 23 papers in Cellular and Molecular Neuroscience, 21 papers in Endocrine and Autonomic Systems and 12 papers in Cognitive Neuroscience. Recurrent topics in Miloslav Kolaj's work include Neuroscience and Neuropharmacology Research (18 papers), Neuroscience of respiration and sleep (17 papers) and Ion channel regulation and function (11 papers). Miloslav Kolaj is often cited by papers focused on Neuroscience and Neuropharmacology Research (18 papers), Neuroscience of respiration and sleep (17 papers) and Ion channel regulation and function (11 papers). Miloslav Kolaj collaborates with scholars based in Canada, United States and South Korea. Miloslav Kolaj's co-authors include Leo P. Renaud, Mirjana Randić, Li Zhang, Elaine Coderre, Gong Cheng, R. Cerne, Michael Hermes, L. Zhang, P. A. Doroshenko and Trevor Richter and has published in prestigious journals such as Journal of Neuroscience, The Journal of Physiology and Journal of Neurophysiology.

In The Last Decade

Miloslav Kolaj

36 papers receiving 779 citations

Peers

Miloslav Kolaj
Miloslav Kolaj
Citations per year, relative to Miloslav Kolaj Miloslav Kolaj (= 1×) peers Ju-en Zhang

Countries citing papers authored by Miloslav Kolaj

Since Specialization
Citations

This map shows the geographic impact of Miloslav Kolaj's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Miloslav Kolaj with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Miloslav Kolaj more than expected).

Fields of papers citing papers by Miloslav Kolaj

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Miloslav Kolaj. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Miloslav Kolaj. The network helps show where Miloslav Kolaj may publish in the future.

Co-authorship network of co-authors of Miloslav Kolaj

This figure shows the co-authorship network connecting the top 25 collaborators of Miloslav Kolaj. A scholar is included among the top collaborators of Miloslav Kolaj based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Miloslav Kolaj. Miloslav Kolaj is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Kolaj, Miloslav, Li Zhang, & Leo P. Renaud. (2014). Novel coupling between TRPC-like and KNa channels modulates low threshold spike-induced afterpotentials in rat thalamic midline neurons. Neuropharmacology. 86. 88–96. 13 indexed citations
3.
Kolaj, Miloslav, Li Zhang, Michael Hermes, & Leo P. Renaud. (2014). Intrinsic properties and neuropharmacology of midline paraventricular thalamic nucleus neurons. Frontiers in Behavioral Neuroscience. 8. 132–132. 43 indexed citations
4.
Zhang, Li, Miloslav Kolaj, & Leo P. Renaud. (2013). GIRK-like and TRPC-like conductances mediate thyrotropin-releasing hormone-induced increases in excitability in thalamic paraventricular nucleus neurons. Neuropharmacology. 72. 106–115. 17 indexed citations
5.
Hermes, Michael, Miloslav Kolaj, Elaine Coderre, & Leo P. Renaud. (2013). Gastrin‐releasing peptide acts via postsynaptic BB2 receptors to modulate inward rectifier K+ and TRPV1‐like conductances in rat paraventricular thalamic neurons. The Journal of Physiology. 591(7). 1823–1839. 21 indexed citations
6.
Hermes, Michael, Miloslav Kolaj, P. A. Doroshenko, Elaine Coderre, & Leo P. Renaud. (2009). Effects of VPAC2 Receptor Activation on Membrane Excitability and GABAergic Transmission in Subparaventricular Zone Neurons Targeted by Suprachiasmatic Nucleus. Journal of Neurophysiology. 102(3). 1834–1842. 20 indexed citations
7.
Kolaj, Miloslav & Leo P. Renaud. (2009). Metabotropic Glutamate Receptors in Median Preoptic Neurons Modulate Neuronal Excitability and Glutamatergic and GABAergic Inputs From the Subfornical Organ. Journal of Neurophysiology. 103(2). 1104–1113. 14 indexed citations
8.
Kolaj, Miloslav, Elaine Coderre, & Leo P. Renaud. (2008). Orexin peptides enhance median preoptic nucleus neuronal excitability via postsynaptic membrane depolarization and enhancement of glutamatergic afferents. Neuroscience. 155(4). 1212–1220. 17 indexed citations
9.
Gálik, Ján, Dong‐ho Youn, Miloslav Kolaj, & Mirjana Randić. (2008). Involvement of group I metabotropic glutamate receptors and glutamate transporters in the slow excitatory synaptic transmission in the spinal cord dorsal horn. Neuroscience. 154(4). 1372–1387. 15 indexed citations
10.
Kolaj, Miloslav & Leo P. Renaud. (2007). Presynaptic α-adrenoceptors in median preoptic nucleus modulate inhibitory neurotransmission from subfornical organ and organum vasculosum lamina terminalis. American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 292(5). R1907–R1915. 11 indexed citations
13.
Richter, Trevor, Miloslav Kolaj, & Leo P. Renaud. (2005). Low Voltage-Activated Ca2+Channels Are Coupled to Ca2+-Induced Ca2+Release in Rat Thalamic Midline Neurons. Journal of Neuroscience. 25(36). 8267–8271. 33 indexed citations
14.
Zhang, L., et al.. (2005). Vasopressin induces depolarization and state-dependent firing patterns in rat thalamic paraventricular nucleus neurons in vitro. American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 290(5). R1226–R1232. 24 indexed citations
15.
Kolaj, Miloslav, Donglin Bai, & Leo P. Renaud. (2004). GABAB Receptor Modulation of Rapid Inhibitory and Excitatory Neurotransmission From Subfornical Organ and Other Afferents to Median Preoptic Nucleus Neurons. Journal of Neurophysiology. 92(1). 111–122. 37 indexed citations
16.
Kolaj, Miloslav & Leo P. Renaud. (2001). Norepinephrine acts via α2 adrenergic receptors to suppress N-type calcium channels in dissociated rat median preoptic nucleus neurons. Neuropharmacology. 41(4). 472–480. 12 indexed citations
17.
Kolaj, Miloslav, C.R Yang, & Leo P. Renaud. (2000). Presynaptic GABAB receptors modulate organum vasculosum lamina terminalis-evoked postsynaptic currents in rat hypothalamic supraoptic neurons. Neuroscience. 98(1). 129–133. 14 indexed citations
18.
Kolaj, Miloslav & Leo P. Renaud. (1999). Vasopressin acting at V1-type receptors produces membrane depolarization in neonatal rat spinal lateral column neurons. Progress in brain research. 119. 275–284. 7 indexed citations
19.
Kolaj, Miloslav & Leo P. Renaud. (1998). Vasopressin’s Depolarizing Action on Neonatal Rat Spinal Lateral Horn Neurons May Involve Multiple Conductances. Advances in experimental medicine and biology. 449. 201–210. 1 indexed citations
20.
Randić, Mirjana, M. C. Jiang, K.I. Rusin, R. Cerne, & Miloslav Kolaj. (1993). Interactions between excitatory amino acids and tachykinins and long-term changes of synaptic responses in the rat spinal dorsal horn. Regulatory Peptides. 46(1-2). 418–420. 22 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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