Miguel Weil

5.9k total citations · 1 hit paper
54 papers, 4.8k citations indexed

About

Miguel Weil is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cell Biology. According to data from OpenAlex, Miguel Weil has authored 54 papers receiving a total of 4.8k indexed citations (citations by other indexed papers that have themselves been cited), including 39 papers in Molecular Biology, 15 papers in Cellular and Molecular Neuroscience and 13 papers in Cell Biology. Recurrent topics in Miguel Weil's work include Hereditary Neurological Disorders (9 papers), Cellular Mechanics and Interactions (6 papers) and Amyotrophic Lateral Sclerosis Research (6 papers). Miguel Weil is often cited by papers focused on Hereditary Neurological Disorders (9 papers), Cellular Mechanics and Interactions (6 papers) and Amyotrophic Lateral Sclerosis Research (6 papers). Miguel Weil collaborates with scholars based in Israel, United Kingdom and United States. Miguel Weil's co-authors include Martin Raff, Michael D. Jacobson, Manoel Jacobsen, Vania Braga, T. J. Davies, Harriet Coles, R.L. Gardner, Bodo Christ, Helge Amthor and Adit Ben‐Baruch and has published in prestigious journals such as Cell, Nature Communications and Journal of Neuroscience.

In The Last Decade

Miguel Weil

54 papers receiving 4.8k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Programmed Cell Death in Animal Development

1997 2.2k citations Michael D. Jacobson, Miguel Weil et al. Cell profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Miguel Weil Israel	24	3.1k	892	645	588	530	54	4.8k
Bjørn Steen Skålhegg Norway	36	2.6k 0.8×	888 1.0×	578 0.9×	432 0.7×	294 0.6×	125	4.3k
Yih‐Cherng Liou Singapore	39	3.5k 1.1×	1.0k 1.1×	1.1k 1.7×	562 1.0×	739 1.4×	89	5.3k
H. Robert Horvitz United States	14	2.7k 0.9×	642 0.7×	369 0.6×	455 0.8×	486 0.9×	16	4.3k
Friedrich Buck Germany	41	3.0k 0.9×	562 0.6×	459 0.7×	397 0.7×	412 0.8×	120	4.6k
Dapeng Zhang China	45	3.5k 1.1×	729 0.8×	421 0.7×	385 0.7×	394 0.7×	178	6.4k
Michael Nehls Germany	29	2.9k 0.9×	1.1k 1.3×	530 0.8×	440 0.7×	501 0.9×	53	5.1k
Tim Thomas Australia	46	4.4k 1.4×	634 0.7×	424 0.7×	523 0.9×	432 0.8×	138	6.3k
Frank N. van Leeuwen Netherlands	45	3.3k 1.0×	792 0.9×	593 0.9×	1.4k 2.3×	373 0.7×	112	6.7k
Christoph Rahner United States	28	2.4k 0.8×	977 1.1×	666 1.0×	487 0.8×	223 0.4×	38	4.8k

Countries citing papers authored by Miguel Weil

Since Specialization

Citations

This map shows the geographic impact of Miguel Weil's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Miguel Weil with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Miguel Weil more than expected).

Fields of papers citing papers by Miguel Weil

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Miguel Weil. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Miguel Weil. The network helps show where Miguel Weil may publish in the future.

Co-authorship network of co-authors of Miguel Weil

This figure shows the co-authorship network connecting the top 25 collaborators of Miguel Weil. A scholar is included among the top collaborators of Miguel Weil based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Miguel Weil. Miguel Weil is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Wolf, Lior, et al.. (2024). Aberrant migration features in primary skin fibroblasts of Huntington's disease patients hold potential for unraveling disease progression using an image based machine learning tool. Computers in Biology and Medicine. 180. 108970–108970. 2 indexed citations

Weil, Miguel, et al.. (2024). Characterization of fission and fusion mitochondrial dynamics in HD fibroblasts according to patient's severity status. Neurobiology of Disease. 201. 106667–106667. 3 indexed citations

Sweetat, Sahar, Saja Baraghithy, Hava Glickstein, et al.. (2024). The Autophagic Activator GHF-201 Can Alleviate Pathology in a Mouse Model and in Patient Fibroblasts of Type III Glycogenosis. Biomolecules. 14(8). 893–893. 1 indexed citations

Abashidze, Anastasia, et al.. (2023). Perturbed actin cap as a new personalized biomarker in primary fibroblasts of Huntington’s disease patients. Frontiers in Cell and Developmental Biology. 11. 1013721–1013721. 8 indexed citations

Morelli, Giovanni, Sophie Laguesse, Nathalie Krusy, et al.. (2021). ATP-citrate lyase promotes axonal transport across species. Nature Communications. 12(1). 5878–5878. 16 indexed citations

Peled, A., Liat Samuelov, Ofer Sarig, et al.. (2021). 078 Treatment of hereditary hypotrichosis simplex of the scalp with topical gentamicin. Journal of Investigative Dermatology. 141(10). S161–S161. 1 indexed citations

Morelli, Giovanni, Loïc Broix, Chiara Scaramuzzino, et al.. (2019). ATAT1-enriched vesicles promote microtubule acetylation via axonal transport. Science Advances. 5(12). eaax2705–eaax2705. 44 indexed citations

Pichinuk, Edward, et al.. (2017). A differential autophagy dependent response to DNA-double strand brakes in bone marrow mesenchymal stem cells from sporadic ALS patients. Disease Models & Mechanisms. 10(5). 645–654. 4 indexed citations

Cohen, Malkiel A., David Cheishvili, Tikva Turetsky, et al.. (2015). Familial Dysautonomia (FD) Human Embryonic Stem Cell Derived PNS Neurons Reveal that Synaptic Vesicular and Neuronal Transport Genes Are Directly or Indirectly Affected by IKBKAP Downregulation. PLoS ONE. 10(10). e0138807–e0138807. 20 indexed citations

10.

Bulvik, Shlomo, et al.. (2012). Two Potential Biomarkers Identified in Mesenchymal Stem Cells and Leukocytes of Patients with Sporadic Amyotrophic lateral Sclerosis. Disease Markers. 32(4). 211–220. 13 indexed citations

11.

Cheishvili, David, et al.. (2011). IKAP/Elp1 involvement in cytoskeleton regulation and implication for familial dysautonomia. Human Molecular Genetics. 20(8). 1585–1594. 49 indexed citations

12.

Cohen-Kupiec, Rachel, et al.. (2010). IKAP/hELP1 down-regulation in neuroblastoma cells causes enhanced cell adhesion mediated by contactin overexpression. Cell Adhesion & Migration. 4(4). 541–550. 4 indexed citations

13.

Solmesky, Leonardo J., et al.. (2009). Bone Morphogenetic Protein Signaling Is Involved in Human Mesenchymal Stem Cell Survival in Serum-Free Medium. Stem Cells and Development. 18(9). 1283–1292. 16 indexed citations

14.

Seksenyan, Akop, Noga Ron‐Harel, David Azoulay, et al.. (2009). Thymic involution, a co‐morbidity factor in amyotrophic lateral sclerosis. Journal of Cellular and Molecular Medicine. 14(10). 2470–2482. 32 indexed citations

15.

Weil, Miguel, et al.. (2005). Neural tube closure depends on nitric oxide synthase activity. Journal of Neurochemistry. 96(1). 247–253. 24 indexed citations

16.

Traister, Alexandra, et al.. (2002). Evidence that nitric oxide regulates cell‐cycle progression in the developing chick neuroepithelium. Developmental Dynamics. 225(3). 271–276. 25 indexed citations

17.

Weil, Miguel, Martin Raff, & Vania Braga. (1999). Caspase activation in the terminal differentiation of human epidermal keratinocytes. Current Biology. 9(7). 361–365. 175 indexed citations

18.

Amthor, Helge, et al.. (1998). The importance of timing differentiation during limb muscle development. Current Biology. 8(11). 642–652. 142 indexed citations

19.

Jacobson, Michael D., Miguel Weil, & Martin Raff. (1997). Programmed Cell Death in Animal Development. Cell. 88(3). 347–354. 2247 indexed citations breakdown →

20.

Jacobsen, Manoel, Miguel Weil, & Martin Raff. (1996). Role of Ced-3/ICE-family proteases in staurosporine-induced programmed cell death.. The Journal of Cell Biology. 133(5). 1041–1051. 338 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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