Miguel Cervantes

1.1k total citations
33 papers, 908 citations indexed

About

Miguel Cervantes is a scholar working on Cellular and Molecular Neuroscience, Neurology and Cognitive Neuroscience. According to data from OpenAlex, Miguel Cervantes has authored 33 papers receiving a total of 908 indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Cellular and Molecular Neuroscience, 12 papers in Neurology and 10 papers in Cognitive Neuroscience. Recurrent topics in Miguel Cervantes's work include Neuroscience and Neuropharmacology Research (16 papers), Neuroinflammation and Neurodegeneration Mechanisms (10 papers) and Stress Responses and Cortisol (7 papers). Miguel Cervantes is often cited by papers focused on Neuroscience and Neuropharmacology Research (16 papers), Neuroinflammation and Neurodegeneration Mechanisms (10 papers) and Stress Responses and Cortisol (7 papers). Miguel Cervantes collaborates with scholars based in Mexico and United States. Miguel Cervantes's co-authors include Gabriela Moralı́, Ignacio González‐Burgos, María Esther Olvera‐Cortés, Alfonso Escobar, Claudia Espinosa‐García, Marisela Hernández‐González, Miguel Ángel Guevara, Dulce A. Velázquez-Zamora, Christina Sobin and Tanner Schaub and has published in prestigious journals such as Brain Research, Neuroscience and Behavioural Brain Research.

In The Last Decade

Miguel Cervantes

32 papers receiving 886 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Miguel Cervantes Mexico 18 306 219 192 157 157 33 908
Sonia Luquı́n Mexico 22 339 1.1× 155 0.7× 283 1.5× 118 0.8× 249 1.6× 56 1.5k
Takashi Fujioka Japan 13 468 1.5× 154 0.7× 118 0.6× 164 1.0× 43 0.3× 34 1.2k
Margaret M. Koletar Canada 15 250 0.8× 157 0.7× 138 0.7× 339 2.2× 107 0.7× 33 1.1k
De‐Lai Qiu China 15 401 1.3× 145 0.7× 194 1.0× 126 0.8× 35 0.2× 79 725
Olusegun J. Ariwodola United States 18 628 2.1× 276 1.3× 110 0.6× 70 0.4× 80 0.5× 22 923
Natalina Salmaso Canada 19 474 1.5× 191 0.9× 223 1.2× 305 1.9× 41 0.3× 40 1.5k
Li‐Chun Lin United States 10 233 0.8× 144 0.7× 121 0.6× 141 0.9× 45 0.3× 17 830
Lisa S. Robison United States 23 323 1.1× 143 0.7× 268 1.4× 104 0.7× 77 0.5× 39 1.2k
Charles Finsterwald Switzerland 10 265 0.9× 107 0.5× 137 0.7× 49 0.3× 41 0.3× 11 799
Zsolt Kis Hungary 22 334 1.1× 103 0.5× 218 1.1× 55 0.4× 40 0.3× 48 1.1k

Countries citing papers authored by Miguel Cervantes

Since Specialization
Citations

This map shows the geographic impact of Miguel Cervantes's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Miguel Cervantes with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Miguel Cervantes more than expected).

Fields of papers citing papers by Miguel Cervantes

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Miguel Cervantes. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Miguel Cervantes. The network helps show where Miguel Cervantes may publish in the future.

Co-authorship network of co-authors of Miguel Cervantes

This figure shows the co-authorship network connecting the top 25 collaborators of Miguel Cervantes. A scholar is included among the top collaborators of Miguel Cervantes based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Miguel Cervantes. Miguel Cervantes is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
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González‐Burgos, Ignacio, et al.. (2015). A Golgi study of the plasticity of dendritic spines in the hypothalamic ventromedial nucleus during the estrous cycle of female rats. Neuroscience. 298. 74–80. 8 indexed citations
5.
Schaub, Tanner, et al.. (2013). Microglial Disruption in Young Mice with Early Chronic Exposure to Lead. Toxicology Letters. 220. 1 indexed citations
6.
Espinosa‐García, Claudia, et al.. (2013). Post-ischemic administration of progesterone reduces caspase-3 activation and DNA fragmentation in the hippocampus following global cerebral ischemia. Neuroscience Letters. 550. 98–103. 12 indexed citations
7.
Sobin, Christina, et al.. (2013). Microglial disruption in young mice with early chronic lead exposure. Toxicology Letters. 220(1). 44–52. 36 indexed citations
8.
Espinosa‐García, Claudia, et al.. (2013). Effects of progesterone on neurite growth inhibitors in the hippocampus following global cerebral ischemia. Brain Research. 1545. 23–34. 23 indexed citations
9.
Velázquez-Zamora, Dulce A., et al.. (2012). Plastic changes in dendritic spines of hippocampal CA1 pyramidal neurons from ovariectomized rats after estradiol treatment. Brain Research. 1470. 1–10. 21 indexed citations
10.
Olvera‐Cortés, María Esther, et al.. (2011). Differential learning-related changes in theta activity during place learning in young and old rats. Behavioural Brain Research. 226(2). 555–562. 24 indexed citations
11.
Cervantes, Miguel, et al.. (2008). Melatonin and ischemia–reperfusion injury of the brain. Journal of Pineal Research. 45(1). 1–7. 82 indexed citations
12.
Moralı́, Gabriela, et al.. (2005). Post-ischemic administration of progesterone in rats exerts neuroprotective effects on the hippocampus. Neuroscience Letters. 382(3). 286–290. 65 indexed citations
13.
González‐Burgos, Ignacio, et al.. (2005). Spine-type densities of hippocampal CA1 neurons vary in proestrus and estrus rats. Neuroscience Letters. 379(1). 52–54. 58 indexed citations
14.
Cervantes, Miguel, et al.. (2002). Neuroprotective Effects of Progesterone on Damage Elicited by Acute Global Cerebral Ischemia in Neurons of the Caudate Nucleus. Archives of Medical Research. 33(1). 6–14. 104 indexed citations
15.
Olvera‐Cortés, María Esther, Miguel Cervantes, & Ignacio González‐Burgos. (2002). Place-learning, but not cue-learning training, modifies the hippocampal theta rhythm in rats. Brain Research Bulletin. 58(3). 261–270. 56 indexed citations
16.
Hernández‐González, Marisela, Miguel Ángel Guevara, Miguel Cervantes, Gabriela Moralı́, & María Corsi‐Cabrera. (1998). Characteristic frequency bands of the cortico-frontal EEG during the sexual interaction of the male rat as a result of factorial analysis. Journal of Physiology-Paris. 92(1). 43–50. 26 indexed citations
17.
Hernández‐González, Marisela, et al.. (1997). Computer Programs to Analyze Brain Electrical Activity During Copulatory Pelvic Thrusting in Male Rats. Physiology & Behavior. 62(4). 701–708. 10 indexed citations
18.
Cervantes, Miguel, et al.. (1995). Effects of propofol on alterations of multineuronal activity of limbic and mesencephalic structures and neurological deficit elicited by acute global cerebral ischemia.. PubMed. 26(4). 385–95. 8 indexed citations
19.
Cervantes, Miguel, Laura de la Torre, & Carlos Beyer. (1975). Analysis of various factors involved in EEG synchronization during milk drinking in the cat. Brain Research. 91(1). 89–98. 10 indexed citations
20.
Salas, M, Miguel Cervantes, & C Guzmán-Flores. (1968). Mechanism of action of quipazine maleate on the central nervous system.. PubMed. 24(1). 191–205. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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