Michelle M. Lissner

890 total citations
8 papers, 635 citations indexed

About

Michelle M. Lissner is a scholar working on Public Health, Environmental and Occupational Health, Immunology and Molecular Biology. According to data from OpenAlex, Michelle M. Lissner has authored 8 papers receiving a total of 635 indexed citations (citations by other indexed papers that have themselves been cited), including 4 papers in Public Health, Environmental and Occupational Health, 3 papers in Immunology and 2 papers in Molecular Biology. Recurrent topics in Michelle M. Lissner's work include Mosquito-borne diseases and control (4 papers), Malaria Research and Control (3 papers) and interferon and immune responses (2 papers). Michelle M. Lissner is often cited by papers focused on Mosquito-borne diseases and control (4 papers), Malaria Research and Control (3 papers) and interferon and immune responses (2 papers). Michelle M. Lissner collaborates with scholars based in United States, Canada and Italy. Michelle M. Lissner's co-authors include Stephen T. Smale, Dev Bhatt, Gioacchino Natoli, Amy Pandya‐Jones, Douglas L. Black, Iros Barozzi, Brandon J. Thomas, David S. Schneider, Grant D. Barish and Xin Liu and has published in prestigious journals such as Cell, Nature Genetics and PLoS ONE.

In The Last Decade

Michelle M. Lissner

8 papers receiving 632 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Michelle M. Lissner United States	8	417	251	145	48	41	8	635
Sneha Rath United States	11	609 1.5×	243 1.0×	95 0.7×	17 0.4×	35 0.9×	14	751
Sara R. Heras Spain	17	682 1.6×	88 0.4×	94 0.6×	36 0.8×	88 2.1×	22	841
Indira Paddibhatla United States	10	353 0.8×	165 0.7×	299 2.1×	25 0.5×	19 0.5×	19	594
Didier Scherrer France	14	201 0.5×	124 0.5×	81 0.6×	17 0.4×	97 2.4×	32	533
Sang-Ho Yoon South Korea	8	205 0.5×	215 0.9×	165 1.1×	50 1.0×	6 0.1×	14	457
Hans-Rudolf Hotz Switzerland	15	555 1.3×	51 0.2×	98 0.7×	93 1.9×	45 1.1×	21	713
Baskar Bakthavachalu India	10	402 1.0×	76 0.3×	171 1.2×	14 0.3×	29 0.7×	16	544
Amit Frishberg Israel	7	195 0.5×	133 0.5×	50 0.3×	11 0.2×	19 0.5×	12	332
Cecil Han South Korea	13	292 0.7×	100 0.4×	61 0.4×	102 2.1×	113 2.8×	22	519

Countries citing papers authored by Michelle M. Lissner

Since Specialization

Citations

This map shows the geographic impact of Michelle M. Lissner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michelle M. Lissner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michelle M. Lissner more than expected).

Fields of papers citing papers by Michelle M. Lissner

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michelle M. Lissner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michelle M. Lissner. The network helps show where Michelle M. Lissner may publish in the future.

Co-authorship network of co-authors of Michelle M. Lissner

This figure shows the co-authorship network connecting the top 25 collaborators of Michelle M. Lissner. A scholar is included among the top collaborators of Michelle M. Lissner based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michelle M. Lissner. Michelle M. Lissner is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Mouri, Kousuke, Michael H. Guo, Carl G. de Boer, et al.. (2022). Prioritization of autoimmune disease-associated genetic variants that perturb regulatory element activity in T cells. Nature Genetics. 54(5). 603–612. 27 indexed citations

2.

Davis, Nicole M., et al.. (2021). Metabolomic Analysis of Diverse Mice Reveals Hepatic Arginase-1 as Source of Plasma Arginase in Plasmodium chabaudi Infection. mBio. 12(5). e0242421–e0242421. 7 indexed citations

3.

Lissner, Michelle M., Katherine Cumnock, Nicole M. Davis, et al.. (2020). Metabolic profiling during malaria reveals the role of the aryl hydrocarbon receptor in regulating kidney injury. eLife. 9. 9 indexed citations

4.

Cumnock, Katherine, et al.. (2018). Host Energy Source Is Important for Disease Tolerance to Malaria. Current Biology. 28(10). 1635–1642.e3. 53 indexed citations

5.

Lissner, Michelle M. & David S. Schneider. (2018). The physiological basis of disease tolerance in insects. Current Opinion in Insect Science. 29. 133–136. 14 indexed citations

6.

Liu, Xin, Brandon J. Thomas, Michelle M. Lissner, et al.. (2016). A Stringent Systems Approach Uncovers Gene-Specific Mechanisms Regulating Inflammation. Cell. 165(1). 165–179. 136 indexed citations

7.

Lissner, Michelle M., Brandon J. Thomas, Kathleen Wee, et al.. (2015). Age-Related Gene Expression Differences in Monocytes from Human Neonates, Young Adults, and Older Adults. PLoS ONE. 10(7). e0132061–e0132061. 38 indexed citations

8.

Bhatt, Dev, Amy Pandya‐Jones, Iros Barozzi, et al.. (2012). Transcript Dynamics of Proinflammatory Genes Revealed by Sequence Analysis of Subcellular RNA Fractions. Cell. 150(2). 279–290. 351 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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