Michel Démarchez
- Molecular Biology
- Cell Biology top 5%
- Dermatology top 2%
- Immunology
- Rehabilitation top 2%
- Co-authors
- M PruniérasDaniel HartmannJanusz CzernielewskiPhilippe SengelDaniel AsselineauD. HerbageAnnick MaugerFrançoise Bernerd
- Topics
- Skin and Cellular Biology Research (13 papers)Wound Healing and Treatments (6 papers)melanin and skin pigmentation (5 papers)
- Partner nations
- FranceUnited KingdomBelgium
In The Last Decade
Michel Démarchez
34 papers receiving 912 citations
Peers
Comparison fields: 5 of 81
- Molecular Biology 306
- Cell Biology 306
- Dermatology 194
- Immunology 186
- Rehabilitation 180
Countries citing papers authored by Michel Démarchez
This map shows the geographic impact of Michel Démarchez's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michel Démarchez with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michel Démarchez more than expected).
Fields of papers citing papers by Michel Démarchez
This network shows the impact of papers produced by Michel Démarchez. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michel Démarchez. The network helps show where Michel Démarchez may publish in the future.
Co-authorship network of co-authors of Michel Démarchez
This figure shows the co-authorship network connecting the top 25 collaborators of Michel Démarchez. A scholar is included among the top collaborators of Michel Démarchez based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michel Démarchez. Michel Démarchez is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 29 | |
| 2 | 61 | |
| 3 | 40 | |
| 4 | 4 | |
| 5 | 99 | |
| 6 | 35 | |
| 7 | 1 | |
| 8 | 13 | |
| 9 | 1 | |
| 10 | 12 | |
| 11 | 10 | |
| 12 | 56 | |
| 13 | 1 | |
| 14 | 18 | |
| 15 | 39 | |
| 16 | 12 | |
| 17 | 26 | |
| 18 | Role of extracellular matrix and of dermal-epidermal junction architecture in skin development. | 10 |
| 19 | 25 | |
| 20 | The dermal-epidermal junction during the development of skin and cutaneous appendages in the chick embryo. | 8 |
About Michel Démarchez
Michel Démarchez is a scholar working on Cell Biology, Rehabilitation and Transplantation, having authored 38 papers that have together received 950 indexed citations. Recurring topics across this work include Skin and Cellular Biology Research (13 papers), Wound Healing and Treatments (6 papers) and melanin and skin pigmentation (5 papers). The work is most often cited by research in Rehabilitation (180 citations), Dermatology (194 citations) and Cell Biology (306 citations). Michel Démarchez has collaborated with scholars based in France, United Kingdom and Belgium. Frequent co-authors include M Pruniéras, Daniel Hartmann, Janusz Czernielewski, Philippe Sengel, Daniel Asselineau, D. Herbage, Annick Mauger, Françoise Bernerd, Annie Ladoux and Serge Michel. Their work appears in journals such as Journal of Biological Chemistry, Developmental Biology and Journal of Investigative Dermatology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.