Michaela Bissinger

877 total citations
16 papers, 549 citations indexed

About

Michaela Bissinger is a scholar working on Molecular Biology, Cell Biology and Oncology. According to data from OpenAlex, Michaela Bissinger has authored 16 papers receiving a total of 549 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 8 papers in Cell Biology and 4 papers in Oncology. Recurrent topics in Michaela Bissinger's work include Ubiquitin and proteasome pathways (5 papers), Hippo pathway signaling and YAP/TAZ (4 papers) and Liver physiology and pathology (4 papers). Michaela Bissinger is often cited by papers focused on Ubiquitin and proteasome pathways (5 papers), Hippo pathway signaling and YAP/TAZ (4 papers) and Liver physiology and pathology (4 papers). Michaela Bissinger collaborates with scholars based in Germany, United States and Switzerland. Michaela Bissinger's co-authors include Peter Schirmacher, Kai Breuhahn, Mona Malz, Jana Samarin, Norbert Gretz, Stephan Singer, Volker Ehemann, Thomas Longerich, Achim Weber and Marc‐Oliver Riener and has published in prestigious journals such as Hepatology, Cancer Research and International Journal of Molecular Sciences.

In The Last Decade

Michaela Bissinger

16 papers receiving 542 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Michaela Bissinger Germany	11	387	172	129	101	53	16	549
Tai-On Yau Hong Kong	6	436 1.1×	155 0.9×	114 0.9×	133 1.3×	53 1.0×	6	565
Hui Rao China	10	299 0.8×	125 0.7×	77 0.6×	103 1.0×	37 0.7×	19	439
Ji-Hai Yu China	12	276 0.7×	143 0.8×	55 0.4×	140 1.4×	71 1.3×	19	483
Yibin Ren China	12	365 0.9×	163 0.9×	68 0.5×	149 1.5×	90 1.7×	13	531
Chengdong Qin China	10	358 0.9×	303 1.8×	52 0.4×	105 1.0×	31 0.6×	19	545
Zhenhai Lin China	9	329 0.9×	150 0.9×	70 0.5×	143 1.4×	57 1.1×	15	536
Diana Becker Germany	14	237 0.6×	127 0.7×	59 0.5×	132 1.3×	43 0.8×	22	426
Kai‐Yu Ng Hong Kong	9	402 1.0×	235 1.4×	42 0.3×	130 1.3×	43 0.8×	11	542
Floriane Pez France	5	288 0.7×	120 0.7×	47 0.4×	92 0.9×	29 0.5×	6	431

Countries citing papers authored by Michaela Bissinger

Since Specialization

Citations

This map shows the geographic impact of Michaela Bissinger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michaela Bissinger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michaela Bissinger more than expected).

Fields of papers citing papers by Michaela Bissinger

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michaela Bissinger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michaela Bissinger. The network helps show where Michaela Bissinger may publish in the future.

Co-authorship network of co-authors of Michaela Bissinger

This figure shows the co-authorship network connecting the top 25 collaborators of Michaela Bissinger. A scholar is included among the top collaborators of Michaela Bissinger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michaela Bissinger. Michaela Bissinger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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16 of 16 papers shown

Weiler, Sofia M.E., Michaela Bissinger, Teresa Lutz, et al.. (2024). SEPTIN10-mediated crosstalk between cytoskeletal networks controls mechanotransduction and oncogenic YAP/TAZ signaling. Cancer Letters. 584. 216637–216637. 9 indexed citations

Weiler, Sofia M.E., Michaela Bissinger, Sabine Merker, et al.. (2024). CRKL Enhances YAP Signaling through Binding and JNK/JUN Pathway Activation in Liver Cancer. International Journal of Molecular Sciences. 25(15). 8549–8549. 5 indexed citations

Thomann, Stefan, Sofia M.E. Weiler, Michaela Bissinger, et al.. (2020). Yes-associated protein (YAP) induces a secretome phenotype and transcriptionally regulates plasminogen activator Inhibitor-1 (PAI-1) expression in hepatocarcinogenesis. Cell Communication and Signaling. 18(1). 166–166. 33 indexed citations

Weiler, Sofia M.E., Teresa Lutz, Michaela Bissinger, et al.. (2020). TAZ target gene ITGAV regulates invasion and feeds back positively on YAP and TAZ in liver cancer cells. Cancer Letters. 473. 164–175. 41 indexed citations

Pinna, Federico, Michaela Bissinger, Nicolas Huber, et al.. (2017). A20/TNFAIP3 Discriminates Tumor Necrosis Factor (TNF)-Induced NF-κB from JNK Pathway Activation in Hepatocytes. Frontiers in Physiology. 8. 610–610. 16 indexed citations

Schildberg, Frank A., Federico Pinna, Ute Albrecht, et al.. (2017). Quantitative and integrative analysis of paracrine hepatocyte activation by nonparenchymal cells upon lipopolysaccharide induction. FEBS Journal. 284(5). 796–813. 1 indexed citations

Samarin, Jana, Vibor Laketa, Mona Malz, et al.. (2015). PI3K/AKT/mTOR‐dependent stabilization of oncogenic far‐upstream element binding proteins in hepatocellular carcinoma cells. Hepatology. 63(3). 813–826. 55 indexed citations

Malz, Mona, Jana Samarin, Carsten Sticht, et al.. (2014). Overexpression of far upstream element (FUSE) binding protein (FBP)-interacting repressor (FIR) supports growth of hepatocellular carcinoma. Hepatology. 60(4). 1241–1250. 37 indexed citations

Pinna, Federico, Sven Sahle, Michaela Bissinger, et al.. (2013). Abstract 5235: A model for TNFα-mediated NFκB signalling: A systems biology study on hepatocytes and liver cancer cells.. Cancer Research. 73(8_Supplement). 5235–5235. 2 indexed citations

10.

Malz, Mona, Jana Samarin, Michaela Bissinger, et al.. (2012). Nuclear accumulation of seven in absentia homologue‐2 supports motility and proliferation of liver cancer cells. International Journal of Cancer. 131(9). 2016–2026. 23 indexed citations

11.

Pinna, Federico, Sven Sahle, Michaela Bissinger, et al.. (2012). A Systems Biology Study on NFκB Signaling in Primary Mouse Hepatocytes. Frontiers in Physiology. 3. 466–466. 5 indexed citations

12.

Malz, Mona, Darjus F. Tschaharganeh, Nisar P. Malek, et al.. (2011). Nuclear expression of the ubiquitin ligase seven in absentia homolog (SIAH)-1 induces proliferation and migration of liver cancer cells. Journal of Hepatology. 55(5). 1049–1057. 40 indexed citations

13.

Singer, Stephan, Mona Malz, Esther Herpel, et al.. (2009). Coordinated Expression of Stathmin Family Members by Far Upstream Sequence Element-Binding Protein-1 Increases Motility in Non–Small Cell Lung Cancer. Cancer Research. 69(6). 2234–2243. 83 indexed citations

14.

Malz, Mona, Achim Weber, Stephan Singer, et al.. (2009). Overexpression of Far Upstream Element Binding Proteins. Hepatology. 50(4). 1130–1139. 92 indexed citations

15.

Bissinger, Michaela, Roland Eils, Peter Angel, et al.. (2008). AP-1-Controlled Hepatocyte Growth Factor Activation Promotes Keratinocyte Migration via CEACAM1 and Urokinase Plasminogen Activator/Urokinase Plasminogen Receptor. Journal of Investigative Dermatology. 129(5). 1140–1148. 17 indexed citations

16.

Samarin, Jana, Volker Ehemann, Michaela Bissinger, et al.. (2008). Autocrine insulin‐like growth factor‐II stimulation of tumor cell migration is a progression step in human hepatocarcinogenesis†‡. Hepatology. 48(1). 146–156. 90 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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