Michael R. O’Donovan

2.3k total citations
35 papers, 911 citations indexed

About

Michael R. O’Donovan is a scholar working on Cancer Research, Molecular Biology and Health, Toxicology and Mutagenesis. According to data from OpenAlex, Michael R. O’Donovan has authored 35 papers receiving a total of 911 indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Cancer Research, 18 papers in Molecular Biology and 6 papers in Health, Toxicology and Mutagenesis. Recurrent topics in Michael R. O’Donovan's work include Carcinogens and Genotoxicity Assessment (20 papers), DNA Repair Mechanisms (6 papers) and Effects and risks of endocrine disrupting chemicals (5 papers). Michael R. O’Donovan is often cited by papers focused on Carcinogens and Genotoxicity Assessment (20 papers), DNA Repair Mechanisms (6 papers) and Effects and risks of endocrine disrupting chemicals (5 papers). Michael R. O’Donovan collaborates with scholars based in United Kingdom, Canada and Australia. Michael R. O’Donovan's co-authors include Michael Fellows, Nikolas J. Hodges, Richard M. Green, M. Graham, James Kevin Chipman, Pierre Lao‐Sirieix, J L Emery, Fiona M Walter, Jane Blazeby and Madhumita Das and has published in prestigious journals such as Oncogene, Radiology and Biosensors and Bioelectronics.

In The Last Decade

Michael R. O’Donovan

35 papers receiving 884 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Michael R. O’Donovan United Kingdom 16 292 278 220 158 138 35 911
Kousuke Saoo Japan 17 243 0.8× 69 0.2× 93 0.4× 172 1.1× 43 0.3× 48 729
Tomasz Dziaman Poland 19 539 1.8× 158 0.6× 49 0.2× 48 0.3× 82 0.6× 23 1.0k
Kranti A. Mapuskar United States 19 444 1.5× 186 0.7× 56 0.3× 108 0.7× 176 1.3× 41 1.2k
D Schmähl Germany 15 207 0.7× 194 0.7× 54 0.2× 65 0.4× 82 0.6× 75 693
Miloslav Dobrota United Kingdom 17 346 1.2× 61 0.2× 107 0.5× 30 0.2× 116 0.8× 44 1.0k
Wolfgang A. Schmalix Germany 20 464 1.6× 275 1.0× 52 0.2× 50 0.3× 66 0.5× 35 1.0k
E. Riklis Israel 17 536 1.8× 143 0.5× 68 0.3× 169 1.1× 24 0.2× 53 1.0k
Susumu Akasaka Japan 17 486 1.7× 202 0.7× 68 0.3× 35 0.2× 94 0.7× 34 885
H J Ahr Germany 16 533 1.8× 93 0.3× 104 0.5× 40 0.3× 94 0.7× 33 1.2k
H. Oßwald Germany 13 173 0.6× 96 0.3× 83 0.4× 60 0.4× 22 0.2× 66 715

Countries citing papers authored by Michael R. O’Donovan

Since Specialization
Citations

This map shows the geographic impact of Michael R. O’Donovan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael R. O’Donovan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael R. O’Donovan more than expected).

Fields of papers citing papers by Michael R. O’Donovan

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael R. O’Donovan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael R. O’Donovan. The network helps show where Michael R. O’Donovan may publish in the future.

Co-authorship network of co-authors of Michael R. O’Donovan

This figure shows the co-authorship network connecting the top 25 collaborators of Michael R. O’Donovan. A scholar is included among the top collaborators of Michael R. O’Donovan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michael R. O’Donovan. Michael R. O’Donovan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
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Manshian, Bella B., et al.. (2014). Chromosome Breakage Induced by the Genotoxic Agents Mitomycin C and Cytosine arabinoside is Concentration and p53 Dependent. Toxicological Sciences. 140(1). 94–102. 21 indexed citations
3.
Pontén, Ingrid, P. Jane Mutch, David Nicholls, et al.. (2013). Micronucleus induction in the bone marrow of rats by pharmacological mechanisms. II: long-acting beta-2 agonism. Mutagenesis. 28(2). 233–239. 2 indexed citations
4.
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Fellows, Michael, et al.. (2011). The ability of the mouse lymphoma TK assay to detect aneugens. Mutagenesis. 26(6). 771–781. 10 indexed citations
6.
7.
Priestley, Catherine, Richard M. Green, Michael Fellows, et al.. (2009). Anomalous genotoxic responses induced in mouse lymphoma L5178Y cells by potassium bromate. Toxicology. 267(1-3). 45–53. 12 indexed citations
8.
Fellows, Michael, Michael R. O’Donovan, Elisabeth Lorge, & David Kirkland. (2008). Comparison of different methods for an accurate assessment of cytotoxicity in the in vitro micronucleus test. Mutation Research/Genetic Toxicology and Environmental Mutagenesis. 655(1-2). 4–21. 55 indexed citations
9.
10.
Honeychurch, Kevin C., Michael R. O’Donovan, & John P. Hart. (2006). Voltammetric behaviour of DNA bases at a screen-printed carbon electrode and its application to a simple and rapid voltammetric method for the determination of oxidative damage in double stranded DNA. Biosensors and Bioelectronics. 22(9-10). 2057–2064. 34 indexed citations
11.
Tweats, David, David H. Blakey, Robert H. Heflich, et al.. (2006). Report of the IWGT working group on strategy/interpretation for regulatory in vivo tests. Mutation Research/Genetic Toxicology and Environmental Mutagenesis. 627(1). 92–105. 43 indexed citations
12.
O’Donovan, Michael R.. (2005). An evaluation of chromatin condensation and DNA integrity in the spermatozoa of men with cancer before and after therapy. Andrologia. 37(2-3). 83–90. 29 indexed citations
13.
Moore, Martha M., Masamitsu Honma, Julie Clements, et al.. (2000). Mouse lymphoma thymidine kinase locus gene mutation assay: International Workshop on Genotoxicity Test Procedures Workgroup report. Environmental and Molecular Mutagenesis. 35(3). 185–190. 22 indexed citations
14.
O’Donovan, Michael R., Sarah E. Johns, & Philip E. Wilcox. (1995). The effect of PHA stimulation on lymphocyte sub-populations in whole-blood cultures. Mutagenesis. 10(4). 371–374. 32 indexed citations
15.
O’Donovan, Michael R., et al.. (1994). Extended-term culture of human T lymphocytes: Material potentially useful in genotoxicity testing. Toxicology in Vitro. 8(4). 651–653. 1 indexed citations
16.
Beare, David, K E Aldridge, Michael R. O’Donovan, & Jane Cole. (1993). An improved procedure for the in vitro expansion of human T-lymphocyte clones for mutant analysis. Mutation Research/Environmental Mutagenesis and Related Subjects. 291(3). 207–212. 8 indexed citations
17.
O’Donovan, Michael R., et al.. (1993). Formaldehyde is a bacterial mutagen in a range of Salmonella and Escherichia indicator strains. Mutagenesis. 8(6). 577–581. 13 indexed citations
18.
Wilcox, Philip E., et al.. (1992). Sensitivity of Salmonella typhimurium TA97a to the type of agar used for preparation of Vogel-Bonner plates. Mutagenesis. 7(1). 13–18. 4 indexed citations
19.
Dykes, P.J., et al.. (1991). In vitro reconstruction of human skin: The use of skin equivalents as potential indicators of cutaneous toxicity. Toxicology in Vitro. 5(1). 1–8. 21 indexed citations
20.
O’Donovan, Michael R.. (1990). 1,8-Dinitropyrene: comparative mutagenicity in Chinese hamster V79 and CHO cells. Mutagenesis. 5(3). 275–278. 10 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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