Michael Oldridge

4.2k citations

29 papers · 3.0k indexed · 1 hit paper · h-index 19

Impact in

Developmental Biology top 1%
- Congenital limb and hand anomalies
Genetics top 0.5%
- Craniofacial Disorders and Treatments
- Cleft Lip and Palate Research
- Connective tissue disorders research

Papers in ⓘ

Genetics 20
- Craniofacial Disorders and Treatments 14
- Cleft Lip and Palate Research 13
- Connective tissue disorders research 9
- Genomic variations and chromosomal abnormalities 4
Molecular Biology 10
- Wnt/β-catenin signaling in development and cancer 5
- Developmental Biology and Gene Regulation 4

Co-authors: Andrew O.M. Wilkie (21 shared papers)Sarah F. Slaney (3 shared papers)William Reardon (2 shared papers)Paul Rutland (2 shared papers)Robin M. Winter (2 shared papers)Louise J. Pulleyn (2 shared papers)Michael D. Poole (2 shared papers)Richard Hayward (2 shared papers)
Journals: The American Journal of Human Genetics (9 papers)Nature Genetics (5 papers)European Journal of Human Genetics (3 papers)Human Molecular Genetics (2 papers)Lara D. Veeken (1 paper)
Partner nations: United Kingdom United States India

In The Last Decade

Michael Oldridge

28 papers receiving 2.9k citations

Hit Papers

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Apert syndrome results from localized mutations of FGFR2 and is allelic with Crouzon syndrome 1995 · 666 citations

Peers

Countries citing papers authored by Michael Oldridge

Since Specialization

Citations

This map shows the geographic impact of Michael Oldridge's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael Oldridge with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael Oldridge more than expected).

Fields of papers citing papers by Michael Oldridge

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael Oldridge. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael Oldridge. The network helps show where Michael Oldridge may publish in the future.

Co-authors

The 25 scholars most cited alongside Michael Oldridge, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Michael Oldridge Line = papers co-authored together Michael Oldridge links everyone, so they are left out of the graph.

All Works

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20 of 20 papers shown

Showing the 20 most-cited of 29 papers — load more, or switch the sort, to bring in the rest.

#	Work
1	Apert syndrome results from localized mutations of FGFR2 and is allelic with Crouzon syndrome Nature Genetics ·Andrew O.M. Wilkie, Sarah F. Slaney, Michael Oldridge, Michael D. Poole, Geraldine J. Ashworth, Anthony D. Hockley, Richard Hayward, David J. David, Louise J. Pulleyn, Paul Rutland, S Malcolm, Robin M. Winter, William Reardon Hit paper breakdown →	1995	666
2	Identical mutations in the FGFR2 gene cause both Pfeiffer and Crouzon syndrome phenotypes Nature Genetics ·Paul Rutland, Louise J. Pulleyn, William Reardon, M Baraitser, Richard Hayward, Barry M. Jones, Sue Malcolm, Robin M. Winter, Michael Oldridge, Sarah F. Slaney, Michael D. Poole, Andrew O.M. Wilkie	1995	343
3	Recessive Robinow syndrome, allelic to dominant brachydactyly type B, is caused by mutation of ROR2 Nature Genetics ·Ali R. Afzal, Anna Rajab, Christiane Fenske, Michael Oldridge, Navaratnam Elanko, E. Ternes-Pereira, Beyhan Tüysüz, Victoria A. Murday, Michael A. Patton, Andrew O.M. Wilkie, Steven Jeffery	2000	232
4	Exclusive paternal origin of new mutations in Apert syndrome Nature Genetics ·Dominique M. Moloney, Sarah R Slaney, Michael Oldridge, Steven A. Wall, Pelle Sahlin, Göran Stenman, Andrew O.M. Wilkie	1996	217
5	De Novo Alu-Element Insertions in FGFR2 Identify a Distinct Pathological Basis for Apert Syndrome The American Journal of Human Genetics ·Michael Oldridge, Elaine H. Zackai, Donna M. McDonald‐McGinn, Sachiko Iseki, Gillian Morriss‐Kay, Stephen R.F. Twigg, David Johnson, Steven A. Wall, Wen G. Jiang, Christiane Theda, Ethylin Wang Jabs, Andrew O.M. Wilkie	1999	179
6	Differential effects of FGFR2 mutations on syndactyly and cleft palate in Apert syndrome. PubMed ·Sarah F. Slaney, Michael Oldridge, J A Hurst, G M Moriss-Kay, C. Michael Hall, M D Poole, Andrew O.M. Wilkie	1996	174
7	Dominant mutations in ROR2, encoding an orphan receptor tyrosine kinase, cause brachydactyly type B Nature Genetics ·Michael Oldridge, Ana María Fortuna, Monika Maringa, Peter Propping, Sahar Mansour, C. Pollitt, Thomas M. DeChiara, Robert B. Kimble, David M. Valenzuela, George D. Yancopoulos, Andrew O.M. Wilkie	2000	163
8	A Comprehensive Screen for TWIST Mutations in Patients with Craniosynostosis Identifies a New Microdeletion Syndrome of Chromosome Band 7p21.1 The American Journal of Human Genetics ·David Johnson, Sharon W. Horsley, Dominique M. Moloney, Michael Oldridge, Stephen R.F. Twigg, Sinéad Walsh, Margaret Barrow, Pål R. Njølstad, Jürgen Kunz, Geraldine J. Ashworth, Steven A. Wall, Robert E. Kearney, Andrew O.M. Wilkie	1998	143
9	Distinct Mutations in the Receptor Tyrosine Kinase Gene ROR2 Cause Brachydactyly Type B The American Journal of Human Genetics ·Georg C. Schwabe, Sigrid Tinschert, Christian Buschow, Peter Meinecke, G. Wolff, Gabriele Gillessen-Kaesbach, Michael Oldridge, Andrew O.M. Wilkie, Reyhan Kömec, Stefan Mundlos	2000	134
10	Paternal Origin of FGFR2 Mutations in Sporadic Cases of Crouzon Syndrome and Pfeiffer Syndrome The American Journal of Human Genetics ·Rivka L. Glaser, Wen G. Jiang, Simeon A. Boyadjiev, A.K. Tran, Andrea A. Zachary, Lionel Van Maldergem, David Johnson, Sinéad Walsh, Michael Oldridge, Steven A. Wall, Andrew O.M. Wilkie, Ethylin Wang Jabs	2000	125
11	Prevalence of Pro250Arg mutation of fibroblast growth factor receptor 3 in coronal craniosynostosis The Lancet ·D M Moloney, SA Wall, GJ Ashworth, Michael Oldridge, IA Glass, CA Francomano, Maximilian Muenke, Andrew O.M. Wilkie	1997	108
12	Genotype-phenotype correlation for nucleotide substitutions in the IgII- IgIII linker of FGFR2 Human Molecular Genetics ·Michael Oldridge	1997	102
13	Potential Gene Conversion and Source Genes for Recently Integrated Alu Elements Genome Research ·Astrid M. Roy, Marion L. Carroll, Son Nguyen, Abdel Halim Salem, Michael Oldridge, Andrew O.M. Wilkie, Mark A. Batzer, Prescott L. Deininger	2000	90
14	Missense Mutations in the Homeodomain of HOXD13 Are Associated with Brachydactyly Types D and E The American Journal of Human Genetics ·David Johnson, Shih‐hsin Kan, Michael Oldridge, Richard C. Trembath, Philippe Roche, Robert Esnouf, Henk Giele, Andrew O.M. Wilkie	2003	76
15	The spectrum of mutations that underlie the neuromuscular junction synaptopathy in DOK7 congenital myasthenic syndrome Human Molecular Genetics ·Judith Cossins, Wei Wei Liu, Katsiaryna Belaya, Susan Maxwell, Michael Oldridge, Tracy Lester, S. Robb, David Beeson	2012	50
16	Gross deletions in TCOF1 are a cause of Treacher–Collins–Franceschetti syndrome European Journal of Human Genetics ·Michael Bowman, Michael Oldridge, Caroline N. S. Archer, A O'Rourke, Joanna McParland, Roel Brekelmans, A Seller, Tracy Lester	2012	39
17	Interstitial deletion of 2q associated with craniosynostosis, ocular coloboma, and limb abnormalities: Cytogenetic and molecular investigation American Journal of Medical Genetics ·John Nixon, Michael Oldridge, Andrew O.M. Wilkie, Kim Smith	1997	35
18	Craniosynostosis and Related Limb Anomalies Novartis Foundation symposium ·Andrew O.M. Wilkie, Michael Oldridge, Tony P. Tang, Robert E. Maxson	2001	34
19	Brachydactyly Type B: Linkage to Chromosome 9q22 and Evidence for Genetic Heterogeneity The American Journal of Human Genetics ·Michael Oldridge, I. Karen Temple, Heloísa G. dos Santos, Richard J. Gibbons, Zehra Mustafa, Kay Chapman, John Loughlin, Andrew O.M. Wilkie	1999	20
20	Linkage of Otopalatodigital Syndrome Type 2 (OPD2) to Distal Xq28: Evidence for Allelism with OPD1 The American Journal of Human Genetics ·Stephen P. Robertson, Sinéad Walsh, Michael Oldridge, Tania R. Gunn, D. M. O. Becroft, Andrew O.M. Wilkie	2001	18

About Michael Oldridge

Michael Oldridge is a scholar working on Genetics, Molecular Biology, Pediatrics, Perinatology and Child Health, Developmental Biology and Surgery, having authored 29 papers that have together received 3.0k indexed citations. Recurring topics across this work include Craniofacial Disorders and Treatments (14 papers), Cleft Lip and Palate Research (13 papers), Connective tissue disorders research (9 papers), Wnt/β-catenin signaling in development and cancer (5 papers), Developmental Biology and Gene Regulation (4 papers), Genomic variations and chromosomal abnormalities (4 papers), Prenatal Screening and Diagnostics (3 papers) and Congenital limb and hand anomalies (3 papers). The work is most often cited by research in Developmental Biology (231 citations), Genetics (2.1k citations), Molecular Biology (1.8k citations), Genetics (155 citations) and Pediatrics, Perinatology and Child Health (190 citations). Michael Oldridge has collaborated with scholars based in United Kingdom, United States and India. Frequent co-authors include Andrew O.M. Wilkie, Sarah F. Slaney, William Reardon, Paul Rutland, Robin M. Winter, Louise J. Pulleyn, Michael D. Poole, Richard Hayward, Steven A. Wall and S Malcolm. Their work appears in journals such as The American Journal of Human Genetics, Nature Genetics, European Journal of Human Genetics, Human Molecular Genetics and Lara D. Veeken.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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