Michael Oldridge

4.2k total citations · 1 hit paper
29 papers, 3.0k citations indexed

About

Michael Oldridge is a scholar working on Genetics, Molecular Biology and Pediatrics, Perinatology and Child Health. According to data from OpenAlex, Michael Oldridge has authored 29 papers receiving a total of 3.0k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Genetics, 10 papers in Molecular Biology and 3 papers in Pediatrics, Perinatology and Child Health. Recurrent topics in Michael Oldridge's work include Craniofacial Disorders and Treatments (14 papers), Cleft Lip and Palate Research (13 papers) and Connective tissue disorders research (9 papers). Michael Oldridge is often cited by papers focused on Craniofacial Disorders and Treatments (14 papers), Cleft Lip and Palate Research (13 papers) and Connective tissue disorders research (9 papers). Michael Oldridge collaborates with scholars based in United Kingdom, United States and India. Michael Oldridge's co-authors include Andrew O.M. Wilkie, Sarah F. Slaney, Richard Hayward, Paul Rutland, Robin M. Winter, William Reardon, Michael D. Poole, Louise J. Pulleyn, Steven A. Wall and David J. David and has published in prestigious journals such as The Lancet, Nature Genetics and Genome Research.

In The Last Decade

Michael Oldridge

28 papers receiving 2.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Apert syndrome results from localized mutations of FGFR2 and is allelic with Crouzon syndrome

1995 666 citations Andrew O.M. Wilkie, Sarah F. Slaney et al. Nature Genetics profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Michael Oldridge United Kingdom	19	2.1k	1.8k	260	231	190	29	3.0k
Mitsuo Masuno Japan	21	1.1k 0.5×	1.4k 0.8×	275 1.1×	239 1.0×	180 0.9×	86	2.3k
Elena Belloni Italy	20	971 0.5×	2.0k 1.1×	448 1.7×	198 0.9×	238 1.3×	41	2.8k
M.H. Breuning Netherlands	29	1.3k 0.6×	1.1k 0.6×	201 0.8×	82 0.4×	281 1.5×	60	2.6k
Armand Bottani Switzerland	24	1.3k 0.6×	1.2k 0.7×	178 0.7×	105 0.5×	302 1.6×	62	2.2k
Arie van Haeringen Netherlands	26	1.3k 0.7×	1.9k 1.1×	154 0.6×	70 0.3×	296 1.6×	69	3.0k
M.H. Breuning Netherlands	16	1.7k 0.8×	1.4k 0.8×	123 0.5×	85 0.4×	207 1.1×	26	2.1k
Luitgard M. Neumann Germany	20	1.0k 0.5×	708 0.4×	207 0.8×	91 0.4×	110 0.6×	46	1.7k
Jean‐Pierre Fryns Belgium	21	909 0.4×	1.2k 0.7×	206 0.8×	53 0.2×	202 1.1×	25	2.0k
Christèle Dubourg France	28	1.1k 0.6×	1.5k 0.8×	135 0.5×	137 0.6×	442 2.3×	67	1.9k
David J. Bunyan United Kingdom	23	979 0.5×	991 0.5×	208 0.8×	45 0.2×	113 0.6×	60	2.0k

Countries citing papers authored by Michael Oldridge

Since Specialization

Citations

This map shows the geographic impact of Michael Oldridge's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael Oldridge with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael Oldridge more than expected).

Fields of papers citing papers by Michael Oldridge

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael Oldridge. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael Oldridge. The network helps show where Michael Oldridge may publish in the future.

Co-authorship network of co-authors of Michael Oldridge

This figure shows the co-authorship network connecting the top 25 collaborators of Michael Oldridge. A scholar is included among the top collaborators of Michael Oldridge based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michael Oldridge. Michael Oldridge is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Gregson, Celia L., et al.. (2025). Identification of a novel TGFB1 variant in a patient with Camurati-Engelmann disease responsive to alendronate. Lara D. Veeken. 64(12). 6427–6429.

Calder, Alistair, Tracy Lester, Helen Lord, et al.. (2018). A three-generation family with metaphyseal dysplasia, maxillary hypoplasia and brachydactyly (MDMHB) due to intragenic RUNX2 duplication. European Journal of Human Genetics. 26(9). 1288–1293. 9 indexed citations

Cossins, Judith, Wei Wei Liu, Katsiaryna Belaya, et al.. (2012). The spectrum of mutations that underlie the neuromuscular junction synaptopathy in DOK7 congenital myasthenic syndrome. Human Molecular Genetics. 21(17). 3765–3775. 50 indexed citations

Bowman, Michael, Michael Oldridge, A O'Rourke, et al.. (2012). Gross deletions in TCOF1 are a cause of Treacher–Collins–Franceschetti syndrome. European Journal of Human Genetics. 20(7). 769–777. 39 indexed citations

Writzl, Karin, Jera Jeruc, Michael Oldridge, Borut Peterlin, & Raoul C. M. Hennekam. (2008). Genital anomalies in a patient with Treacher Collins syndrome. American Journal of Medical Genetics Part A. 146A(16). 2169–2171. 3 indexed citations

Robertson, Stephen P., Sinéad Walsh, Michael Oldridge, et al.. (2001). Linkage of Otopalatodigital Syndrome Type 2 (OPD2) to Distal Xq28: Evidence for Allelism with OPD1. The American Journal of Human Genetics. 69(1). 223–227. 18 indexed citations

Oldridge, Michael, et al.. (2000). Autosomal recessive Robinow syndrome is caused by homozygous mutations in ROR2.. The American Journal of Human Genetics. 67. 389–389. 2 indexed citations

Afzal, Ali R., Anna Rajab, Christiane Fenske, et al.. (2000). Autosomal recessive Robinow syndrome is allelic to dominant brachydactyly type B and caused by loss of function mutations in ROR2. Journal of Medical Genetics. 37. 2 indexed citations

Schwabe, Georg C., Sigrid Tinschert, Peter Meinecke, et al.. (2000). Distinct Mutations in the Receptor Tyrosine Kinase Gene ROR2 Cause Brachydactyly Type B. The American Journal of Human Genetics. 67(4). 822–831. 134 indexed citations

10.

Oldridge, Michael, Ana María Fortuna, Monika Maringa, et al.. (2000). Dominant mutations in ROR2, encoding an orphan receptor tyrosine kinase, cause brachydactyly type B. Nature Genetics. 24(3). 275–278. 163 indexed citations

11.

Afzal, Ali R., Anna Rajab, Christiane Fenske, et al.. (2000). Recessive Robinow syndrome, allelic to dominant brachydactyly type B, is caused by mutation of ROR2. Nature Genetics. 25(4). 419–422. 232 indexed citations

12.

Glaser, Robert L., et al.. (1999). Paternal origin of FGFR2 mutations in sporadic cases of Crouzon and Pfeiffer syndromes.. The American Journal of Human Genetics. 65. 3 indexed citations

13.

Oldridge, Michael, I. Karen Temple, Heloísa G. dos Santos, et al.. (1999). Brachydactyly Type B: Linkage to Chromosome 9q22 and Evidence for Genetic Heterogeneity. The American Journal of Human Genetics. 64(2). 578–585. 20 indexed citations

14.

Oldridge, Michael, Elaine H. Zackai, Donna M. McDonald‐McGinn, et al.. (1999). De Novo Alu-Element Insertions in FGFR2 Identify a Distinct Pathological Basis for Apert Syndrome. The American Journal of Human Genetics. 64(2). 446–461. 179 indexed citations

15.

Wilkie, Andrew O.M., Sharon W. Horsley, Robert E. Kearney, et al.. (1998). Genes in craniosynostosis. European Journal of Human Genetics. 6. 27–27. 1 indexed citations

16.

Johnson, David, Sharon W. Horsley, Dominique M. Moloney, et al.. (1998). A Comprehensive Screen for TWIST Mutations in Patients with Craniosynostosis Identifies a New Microdeletion Syndrome of Chromosome Band 7p21.1. The American Journal of Human Genetics. 63(5). 1282–1293. 143 indexed citations

17.

Oldridge, Michael. (1997). Genotype-phenotype correlation for nucleotide substitutions in the IgII- IgIII linker of FGFR2. Human Molecular Genetics. 6(1). 137–143. 102 indexed citations

18.

Ravine, David, Nicola Ragge, Dafydd Stephens, Michael Oldridge, & Andrew O.M. Wilkie. (1997). Dominant coloboma-microphthalmos syndrome associated with sensorineural hearing loss, hematuria, and cleft lip/palate. American Journal of Medical Genetics. 72(2). 227–236. 16 indexed citations

19.

Wilkie, Andrew O.M., Sarah F. Slaney, Michael Oldridge, et al.. (1995). Apert syndrome results from localized mutations of FGFR2 and is allelic with Crouzon syndrome. Nature Genetics. 9(2). 165–172. 666 indexed citations breakdown →

20.

Rutland, Paul, Louise J. Pulleyn, William Reardon, et al.. (1995). Identical mutations in the FGFR2 gene cause both Pfeiffer and Crouzon syndrome phenotypes. Nature Genetics. 9(2). 173–176. 343 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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