Michael N. Davies

1.3k total citations
9 papers, 934 citations indexed

About

Michael N. Davies is a scholar working on Molecular Biology, Clinical Biochemistry and Surgery. According to data from OpenAlex, Michael N. Davies has authored 9 papers receiving a total of 934 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 4 papers in Clinical Biochemistry and 3 papers in Surgery. Recurrent topics in Michael N. Davies's work include Metabolism and Genetic Disorders (4 papers), Cholesterol and Lipid Metabolism (3 papers) and Metabolomics and Mass Spectrometry Studies (2 papers). Michael N. Davies is often cited by papers focused on Metabolism and Genetic Disorders (4 papers), Cholesterol and Lipid Metabolism (3 papers) and Metabolomics and Mass Spectrometry Studies (2 papers). Michael N. Davies collaborates with scholars based in United States, Netherlands and Germany. Michael N. Davies's co-authors include Deborah M. Muoio, Timothy R. Koves, Howard C. Towle, Brennon O’Callaghan, Olga Ilkayeva, Karen L. DeBalsi, Sarah E. Seiler, Robert C. Noland, Éric Ravussin and Sudip Bajpeyi and has published in prestigious journals such as Journal of Biological Chemistry, Molecular Cell and Cell Metabolism.

In The Last Decade

Michael N. Davies

9 papers receiving 926 citations

Peers

Michael N. Davies
Sivakama S. Bharathi United States
Radha Uppala United States
Vera Lemos Switzerland
Rebecca Quinn United States
Alison Burkart United States
Natalie Gleyzer United States
Ariane Pessentheiner United States
Sivakama S. Bharathi United States
Michael N. Davies
Citations per year, relative to Michael N. Davies Michael N. Davies (= 1×) peers Sivakama S. Bharathi

Countries citing papers authored by Michael N. Davies

Since Specialization
Citations

This map shows the geographic impact of Michael N. Davies's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael N. Davies with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael N. Davies more than expected).

Fields of papers citing papers by Michael N. Davies

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael N. Davies. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael N. Davies. The network helps show where Michael N. Davies may publish in the future.

Co-authorship network of co-authors of Michael N. Davies

This figure shows the co-authorship network connecting the top 25 collaborators of Michael N. Davies. A scholar is included among the top collaborators of Michael N. Davies based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michael N. Davies. Michael N. Davies is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

9 of 9 papers shown
1.
Seiler, Sarah E., Timothy R. Koves, Jessica Gooding, et al.. (2015). Carnitine Acetyltransferase Mitigates Metabolic Inertia and Muscle Fatigue during Exercise. Cell Metabolism. 22(1). 65–76. 76 indexed citations
2.
Davies, Michael N., J. Will Thompson, Laura G. Dubois, et al.. (2015). The Acetyl Group Buffering Action of Carnitine Acetyltransferase Offsets Macronutrient-Induced Lysine Acetylation of Mitochondrial Proteins. Cell Reports. 14(2). 243–254. 70 indexed citations
3.
Laurent, Gaëlle, Natalie J. German, Asish K. Saha, et al.. (2013). SIRT4 Coordinates the Balance between Lipid Synthesis and Catabolism by Repressing Malonyl CoA Decarboxylase. Molecular Cell. 50(5). 686–698. 304 indexed citations
4.
Muoio, Deborah M., Robert C. Noland, Jean-Paul Kovalik, et al.. (2012). Muscle-Specific Deletion of Carnitine Acetyltransferase Compromises Glucose Tolerance and Metabolic Flexibility. Cell Metabolism. 15(5). 764–777. 292 indexed citations
5.
Laurent, Gaëlle, Natalie J. German, Asish K. Saha, et al.. (2012). SIRT4 controls the balance between lipid synthesis and catabolism by repressing malonyl-CoA decarboxylase. BMC Proceedings. 6(S3). 5 indexed citations
6.
Davies, Michael N., Brennon O’Callaghan, & Howard C. Towle. (2010). Activation and repression of glucose-stimulated ChREBP requires the concerted action of multiple domains within the MondoA conserved region. American Journal of Physiology-Endocrinology and Metabolism. 299(4). E665–E674. 19 indexed citations
7.
Collier, Lara S., David J. Adams, Christopher S. Hackett, et al.. (2009). Whole-Body Sleeping Beauty Mutagenesis Can Cause Penetrant Leukemia/Lymphoma and Rare High-Grade Glioma without Associated Embryonic Lethality. Cancer Research. 69(21). 8429–8437. 59 indexed citations
8.
Davies, Michael N., Brennon O’Callaghan, & Howard C. Towle. (2008). Glucose Activates ChREBP by Increasing Its Rate of Nuclear Entry and Relieving Repression of Its Transcriptional Activity. Journal of Biological Chemistry. 283(35). 24029–24038. 79 indexed citations
9.
Davies, Michael N., et al.. (2008). Identification and function of phosphorylation in the glucose-regulated transcription factor ChREBP. Biochemical Journal. 411(2). 261–270. 30 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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