Michael H. Lee

674 total citations · 1 hit paper
16 papers, 449 citations indexed

About

Michael H. Lee is a scholar working on Immunology, Oncology and Rheumatology. According to data from OpenAlex, Michael H. Lee has authored 16 papers receiving a total of 449 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Immunology, 6 papers in Oncology and 4 papers in Rheumatology. Recurrent topics in Michael H. Lee's work include Immunotherapy and Immune Responses (8 papers), T-cell and B-cell Immunology (8 papers) and Immune Cell Function and Interaction (6 papers). Michael H. Lee is often cited by papers focused on Immunotherapy and Immune Responses (8 papers), T-cell and B-cell Immunology (8 papers) and Immune Cell Function and Interaction (6 papers). Michael H. Lee collaborates with scholars based in United States, Canada and Mexico. Michael H. Lee's co-authors include Gudrun F. Debes, Stefania Gallucci, John B. Hay, R. Paul Wilson, Gerard C. L. Wong, Sarah A. Tursi, Ronald P. Wilson, Uma Sriram, Meghan Brown and Eugene C. Butcher and has published in prestigious journals such as Nature reviews. Immunology, The Journal of Immunology and Journal of Virology.

In The Last Decade

Michael H. Lee

15 papers receiving 444 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

How bile acids and the microbiota interact to shape host immunity

2024 52 citations Michael H. Lee, Sean‐Paul Nuccio et al. Nature reviews. Immunology profile →

Peers

Michael H. Lee

IMMUN

MB

ONCOL

PHYSI

RHEUM

Thomas Bickert Germany

Esther W. Taanman-Kueter Netherlands

Rabie Chelbi France

Kalyan C. Nallaparaju United States

Tamaki Sumi Japan

Stephanie Flaherty United States

Carla S. R. Lankford United States

Grefachew Workalemahu Germany

Kumarkrishna Raychaudhuri United States

Tim Vierbuchen Germany

Thomas Bickert Germany

Michael H. Lee

243 ×1.0

IMMUN

127 ×0.9

MB

68 ×0.7

ONCOL

41 ×0.9

PHYSI

27 ×0.7

RHEUM

Citations per year, relative to Michael H. Lee Michael H. Lee (= 1×) peers Thomas Bickert

Countries citing papers authored by Michael H. Lee

Since Specialization

Citations

This map shows the geographic impact of Michael H. Lee's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael H. Lee with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael H. Lee more than expected).

Fields of papers citing papers by Michael H. Lee

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael H. Lee. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael H. Lee. The network helps show where Michael H. Lee may publish in the future.

Co-authorship network of co-authors of Michael H. Lee

This figure shows the co-authorship network connecting the top 25 collaborators of Michael H. Lee. A scholar is included among the top collaborators of Michael H. Lee based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michael H. Lee. Michael H. Lee is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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16 of 16 papers shown

1.

Lee, Michael H., Araceli Pérez-López, Leigh A. Knodler, et al.. (2025). PgtE protease enables virulent Salmonella to evade C3-mediated serum and neutrophil killing. mBio. 16(8). e0380224–e0380224.

2.

Lee, Michael H., Sean‐Paul Nuccio, Ipsita Mohanty, et al.. (2024). How bile acids and the microbiota interact to shape host immunity. Nature reviews. Immunology. 24(11). 798–809. 52 indexed citations breakdown →

3.

Sato, Priscila Y., Connie Qiu, Michael H. Lee, et al.. (2019). Ethyl Pyruvate Modulates Murine Dendritic Cell Activation and Survival Through Their Immunometabolism. Frontiers in Immunology. 10. 30–30. 18 indexed citations

4.

Gallucci, Stefania, et al.. (2019). The metabolic modulator metformin affects the activation and survival of murine dendritic cell subsets. The Journal of Immunology. 202(1_Supplement). 180.17–180.17. 2 indexed citations

5.

Lee, Michael H., et al.. (2019). The cytokine network type I IFN-IL-27-IL-10 is augmented in murine and human lupus. Journal of Leukocyte Biology. 106(4). 967–975. 12 indexed citations

6.

Seliga, Alecia, Michael H. Lee, Nicole Fernandes, et al.. (2018). Kallikrein–Kinin System Suppresses Type I Interferon Responses: A Novel Pathway of Interferon Regulation. Frontiers in Immunology. 9. 156–156. 28 indexed citations

7.

Lee, Michael H., et al.. (2018). Conventional DCs from Male and Female Lupus-Prone B6.NZM Sle1/Sle2/Sle3 Mice Express an IFN Signature and Have a Higher Immunometabolism That Are Enhanced by Estrogen. Journal of Immunology Research. 2018. 1–21. 9 indexed citations

8.

Tursi, Sarah A., Ernest Y. Lee, Michael H. Lee, et al.. (2017). Bacterial amyloid curli acts as a carrier for DNA to elicit an autoimmune response via TLR2 and TLR9. PLoS Pathogens. 13(4). e1006315–e1006315. 88 indexed citations

9.

Qiu, Connie, Michael H. Lee, & Stefania Gallucci. (2017). Modulation of the energy metabolism inhibits plasmacytoid dendritic cell activation and delays onset of autoantibody production in murine models of systemic lupus erythematosus. The Journal of Immunology. 198(Supplement_1). 80.12–80.12. 4 indexed citations

10.

Lee, Michael H., et al.. (2016). The effects of Estrogen on the IFN signature in Lupus. The Journal of Immunology. 196(1_Supplement). 118.18–118.18. 1 indexed citations

11.

Xu, Jun, Michael H. Lee, Benjamin L. Green, et al.. (2016). STAT2 Is Required for TLR-Induced Murine Dendritic Cell Activation and Cross-Presentation. The Journal of Immunology. 197(1). 326–336. 33 indexed citations

12.

Lee, Michael H., et al.. (2013). Ovine skin-recirculating γδ T cells express IFN-γ and IL-17 and exit tissue independently of CCR7. Veterinary Immunology and Immunopathology. 155(1-2). 87–97. 20 indexed citations

13.

Lee, Michael H., et al.. (2012). Tissue Exit: a Novel Control Point in the Accumulation of Antigen-Specific CD8 T Cells in the Influenza A Virus-Infected Lung. Journal of Virology. 86(7). 3436–3445. 31 indexed citations

14.

Lee, Michael H., et al.. (2012). Tissue Exit: a Novel Control Point in the Accumulation of Antigen-Specific CD8 T Cells in the Influenza A Virus-Infected Lung. Journal of Virology. 86(16). 8918–8918. 1 indexed citations

15.

Lee, Michael H., R. Paul Wilson, Reema Patel, et al.. (2012). The Skin, a Novel Niche for Recirculating B Cells. The Journal of Immunology. 188(12). 6027–6035. 76 indexed citations

16.

Brown, Meghan, et al.. (2010). Chemoattractant Receptors and Lymphocyte Egress from Extralymphoid Tissue: Changing Requirements during the Course of Inflammation. The Journal of Immunology. 185(8). 4873–4882. 74 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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