Michael Gabl
About
Michael Gabl is a scholar working on Molecular Biology, Immunology and Cancer Research.
According to data from OpenAlex, Michael Gabl has authored 26 papers receiving a total of 634 indexed citations (citations by other indexed papers that have themselves been cited), including 25 papers in Molecular Biology, 16 papers in Immunology and 8 papers in Cancer Research. Recurrent topics in Michael Gabl's work include S100 Proteins and Annexins (23 papers), Immune Response and Inflammation (15 papers) and Protease and Inhibitor Mechanisms (8 papers). Michael Gabl is often cited by papers focused on S100 Proteins and Annexins (23 papers), Immune Response and Inflammation (15 papers) and Protease and Inhibitor Mechanisms (8 papers). Michael Gabl collaborates with scholars based in Sweden, United States and Denmark. Michael Gabl's co-authors include Huamei Forsman, Cláes Dahlgren, André Holdfeldt, Malene Winther, Martina Sundqvist, Johan Bylund, Jonas Mårtensson, Karin Christenson, Lena Björkman and Sarah Line Skovbakke and has published in prestigious journals such as Journal of Biological Chemistry, The Journal of Immunology and PLoS ONE.
In The Last Decade
Michael Gabl
26 papers receiving 633 citations
Peers
Michael Gabl
André Holdfeldt Sweden
Malene Winther Sweden
Jeannie M. Gripentrog United States
Baoqun Li China
Natalii J. Paczkowski Australia
Jonas Mårtensson Sweden
I. Paegelow Germany
Cecilia Garrè Italy
Charles V. Olson United States
Cristhian J. Ildefonso United States
Citations per year, relative to Michael Gabl Michael Gabl (= 1×)
peers
André Holdfeldt
Countries citing papers authored by Michael Gabl
Since
Specialization
Citations
This map shows the geographic impact of Michael Gabl's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael Gabl with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael Gabl more than expected).
Fields of papers citing papers by Michael Gabl
Since
Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences
This network shows the impact of papers produced by Michael Gabl. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael Gabl. The network helps show where Michael Gabl may publish in the future.
Co-authorship network of co-authors of Michael Gabl
This figure shows the co-authorship network connecting the top 25 collaborators of Michael Gabl. A scholar is included among the top collaborators of Michael Gabl based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michael Gabl. Michael Gabl is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
1.
Venkatakrishnan, Vignesh, Michael Gabl, Otto Savolainen, et al.. (2021). Porphyromonas gingivalis Produce Neutrophil Specific Chemoattractants Including Short Chain Fatty Acids. Frontiers in Cellular and Infection Microbiology. 10. 620681–620681.
2.
Dahlgren, Cláes, André Holdfeldt, Jonas Mårtensson, et al.. (2020). Neutrophil Signaling That Challenges Dogmata of G Protein-Coupled Receptor Regulated Functions. ACS Pharmacology & Translational Science. 3(2). 203–220.
3.
Sundqvist, Martina, Karin Christenson, Michael Gabl, et al.. (2019). Staphylococcus aureus–Derived PSMα Peptides Activate Neutrophil FPR2 but Lack the Ability to Mediate β-Arrestin Recruitment and Chemotaxis. The Journal of Immunology. 203(12). 3349–3360.
4.
Holdfeldt, André, Sarah Line Skovbakke, Michael Gabl, et al.. (2019). Structure–Function Characteristics and Signaling Properties of Lipidated Peptidomimetic FPR2 Agonists: Peptoid Stereochemistry and Residues in the Vicinity of the Headgroup Affect Function. ACS Omega. 4(3). 5968–5982.
5.
Gabl, Michael, André Holdfeldt, Jonas Mårtensson, et al.. (2019). Identification of Residues Critical for FPR2 Activation by the Cryptic Peptide Mitocryptide-2 Originating from the Mitochondrial DNA–Encoded Cytochrome b. The Journal of Immunology. 202(9). 2710–2719.
6.
Venkatakrishnan, Vignesh, Médea Padra, Menno P.J. de Winther, et al.. (2019). Formyl peptide receptor 2 orchestrates mucosal protection againstCitrobacter rodentiuminfection. Virulence. 10(1). 610–624.
7.
Sundqvist, Martina, Karin Christenson, André Holdfeldt, et al.. (2018). Similarities and differences between the responses induced in human phagocytes through activation of the medium chain fatty acid receptor GPR84 and the short chain fatty acid receptor FFA2R. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1865(5). 695–708.
8.
Mårtensson, Jonas, André Holdfeldt, Martina Sundqvist, et al.. (2018). Neutrophil priming that turns natural FFA2R agonists into potent activators of the superoxide generating NADPH-oxidase. Journal of Leukocyte Biology. 104(6). 1117–1132.
9.
Winther, Malene, André Holdfeldt, Martina Sundqvist, et al.. (2017). Formyl peptide derived lipopeptides disclose differences between the receptors in mouse and men and call the pepducin concept in question. PLoS ONE. 12(9). e0185132–e0185132.
10.
Gabl, Michael, et al.. (2017). FPR2 signaling without β-arrestin recruitment alters the functional repertoire of neutrophils. Biochemical Pharmacology. 145. 114–122.
11.
Holdfeldt, André, Sarah Line Skovbakke, Malene Winther, et al.. (2016). The Lipidated Peptidomimetic Lau-((S)-Aoc)-(Lys-βNphe)6-NH2 Is a Novel Formyl Peptide Receptor 2 Agonist That Activates Both Human and Mouse Neutrophil NADPH Oxidase. Journal of Biological Chemistry. 291(38). 19888–19899.
12.
Dahlgren, Cláes, Michael Gabl, André Holdfeldt, Malene Winther, & Huamei Forsman. (2016). Basic characteristics of the neutrophil receptors that recognize formylated peptides, a danger-associated molecular pattern generated by bacteria and mitochondria. Biochemical Pharmacology. 114. 22–39.
13.
Skovbakke, Sarah Line, Malene Winther, Michael Gabl, et al.. (2016). The peptidomimetic Lau-(Lys-βNSpe)6-NH2 antagonizes formyl peptide receptor 2 expressed in mouse neutrophils. Biochemical Pharmacology. 119. 56–65.
14.
Gabl, Michael, André Holdfeldt, Malene Winther, et al.. (2016). A pepducin designed to modulate P2Y 2 R function interacts with FPR2 in human neutrophils and transfers ATP to an NADPH-oxidase-activating ligand through a receptor cross-talk mechanism. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1863(6). 1228–1237.
15.
Winther, Malene, André Holdfeldt, Michael Gabl, et al.. (2016). Formylated MHC Class Ib Binding Peptides Activate Both Human and Mouse Neutrophils Primarily through Formyl Peptide Receptor 1. PLoS ONE. 11(12). e0167529–e0167529.
16.
Gabl, Michael, Malene Winther, Amanda Welin, et al.. (2015). P2Y2 receptor signaling in neutrophils is regulated from inside by a novel cytoskeleton-dependent mechanism. Experimental Cell Research. 336(2). 242–252.
17.
Winther, Malene, Michael Gabl, Amanda Welin, Cláes Dahlgren, & Huamei Forsman. (2015). A neutrophil inhibitory pepducin derived from FPR1 expected to target FPR1 signaling hijacks the closely related FPR2 instead. FEBS Letters. 589(15). 1832–1839.
18.
Forsman, Huamei, Malene Winther, Michael Gabl, et al.. (2014). Structural changes of the ligand and of the receptor alters the receptor preference for neutrophil activating peptides starting with a formylmethionyl group. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1853(1). 192–200.
19.
Gabl, Michael, Malene Winther, Sarah Line Skovbakke, et al.. (2014). A Pepducin Derived from the Third Intracellular Loop of FPR2 Is a Partial Agonist for Direct Activation of This Receptor in Neutrophils But a Full Agonist for Cross-Talk Triggered Reactivation of FPR2. PLoS ONE. 9(10). e109516–e109516.
20.
Önnheim, Karin, Karin Christenson, Michael Gabl, et al.. (2014). A novel receptor cross-talk between the ATP receptor P2Y2 and formyl peptide receptors reactivates desensitized neutrophils to produce superoxide. Experimental Cell Research. 323(1). 209–217.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.
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