Michael D. Schaid

916 total citations
19 papers, 584 citations indexed

About

Michael D. Schaid is a scholar working on Surgery, Molecular Biology and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, Michael D. Schaid has authored 19 papers receiving a total of 584 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Surgery, 8 papers in Molecular Biology and 8 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in Michael D. Schaid's work include Pancreatic function and diabetes (11 papers), Diabetes Treatment and Management (6 papers) and Diabetes and associated disorders (5 papers). Michael D. Schaid is often cited by papers focused on Pancreatic function and diabetes (11 papers), Diabetes Treatment and Management (6 papers) and Diabetes and associated disorders (5 papers). Michael D. Schaid collaborates with scholars based in United States, Australia and Germany. Michael D. Schaid's co-authors include Michelle E. Kimple, Rachel J. Fenske, Talia N. Lerner, Jaclyn A. Wisinski, Jillian L. Seiler, Matthew J. Merrins, Dudley W. Lamming, Dawn S. Sherman, Ildikó Kasza and Gabriella Geiger and has published in prestigious journals such as Journal of Biological Chemistry, The Journal of Physiology and Diabetes.

In The Last Decade

Michael D. Schaid

19 papers receiving 580 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Michael D. Schaid United States 13 274 209 166 121 91 19 584
Thomas F.J. Wagner Germany 8 368 1.3× 151 0.7× 114 0.7× 47 0.4× 205 2.3× 8 933
В. М. Бондарева Russia 12 169 0.6× 119 0.6× 120 0.7× 140 1.2× 137 1.5× 64 567
Elaine S. Coleman United States 15 274 1.0× 143 0.7× 40 0.2× 124 1.0× 133 1.5× 26 783
Teruko Takeo Japan 15 238 0.9× 56 0.3× 145 0.9× 79 0.7× 130 1.4× 28 621
Chi Kin Ip Australia 16 141 0.5× 166 0.8× 112 0.7× 50 0.4× 121 1.3× 23 526
Brandon L. Panaro United States 9 186 0.7× 183 0.9× 105 0.6× 111 0.9× 65 0.7× 10 612
Nicole E. Richardson United States 11 214 0.8× 346 1.7× 44 0.3× 48 0.4× 38 0.4× 15 606
Mulan He China 21 258 0.9× 78 0.4× 76 0.5× 95 0.8× 172 1.9× 47 946
Lynda Whiting New Zealand 13 238 0.9× 190 0.9× 118 0.7× 95 0.8× 131 1.4× 20 700
Claude Roberge Canada 14 140 0.5× 145 0.7× 40 0.2× 118 1.0× 62 0.7× 23 532

Countries citing papers authored by Michael D. Schaid

Since Specialization
Citations

This map shows the geographic impact of Michael D. Schaid's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael D. Schaid with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael D. Schaid more than expected).

Fields of papers citing papers by Michael D. Schaid

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael D. Schaid. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael D. Schaid. The network helps show where Michael D. Schaid may publish in the future.

Co-authorship network of co-authors of Michael D. Schaid

This figure shows the co-authorship network connecting the top 25 collaborators of Michael D. Schaid. A scholar is included among the top collaborators of Michael D. Schaid based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michael D. Schaid. Michael D. Schaid is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Schaid, Michael D., et al.. (2025). Region-specific nucleus accumbens dopamine signals encode distinct aspects of avoidance learning. Current Biology. 35(10). 2433–2443.e5. 3 indexed citations
2.
Neuman, Joshua C., Kathryn A. Carbajal, Michael D. Schaid, et al.. (2024). The prostaglandin E2 EP3 receptor has disparate effects on islet insulin secretion and content in β-cells in a high-fat diet-induced mouse model of obesity. American Journal of Physiology-Endocrinology and Metabolism. 326(5). E567–E576. 1 indexed citations
3.
Schaid, Michael D., et al.. (2023). Chronically dysregulated corticosterone impairs dopaminergic transmission in the dorsomedial striatum by sex-divergent mechanisms. Neuropsychopharmacology. 48(9). 1328–1337. 11 indexed citations
4.
5.
Seiler, Jillian L., et al.. (2022). Dopamine signaling in the dorsomedial striatum promotes compulsive behavior. Current Biology. 32(5). 1175–1188.e5. 40 indexed citations
6.
Tremmel, Daniel M., Sara Dutton Sackett, Austin K. Feeney, et al.. (2022). A human pancreatic ECM hydrogel optimized for 3-D modeling of the islet microenvironment. Scientific Reports. 12(1). 7188–7188. 28 indexed citations
7.
Schaid, Michael D., Yanlong Zhu, Nicole E. Richardson, et al.. (2021). Systemic Metabolic Alterations Correlate with Islet-Level Prostaglandin E2 Production and Signaling Mechanisms That Predict β-Cell Dysfunction in a Mouse Model of Type 2 Diabetes. Metabolites. 11(1). 58–58. 18 indexed citations
8.
Schaid, Michael D., et al.. (2021). GuPPy, a Python toolbox for the analysis of fiber photometry data. Scientific Reports. 11(1). 24212–24212. 58 indexed citations
9.
Sandhu, Harpreet, Joshua C. Neuman, Michael D. Schaid, et al.. (2021). Rat prostaglandin EP3 receptor is highly promiscuous and is the sole prostanoid receptor family member that regulates INS‐1 (832/3) cell glucose‐stimulated insulin secretion. Pharmacology Research & Perspectives. 9(2). e00736–e00736. 12 indexed citations
10.
Wisinski, Jaclyn A., et al.. (2021). Prostaglandin EP3 receptor signaling is required to prevent insulin hypersecretion and metabolic dysfunction in a non-obese mouse model of insulin resistance. American Journal of Physiology-Endocrinology and Metabolism. 321(4). E479–E489. 6 indexed citations
11.
Schaid, Michael D., Cara L. Green, Kathryn A. Carbajal, et al.. (2020). Agonist-independent Gαz activity negatively regulates beta-cell compensation in a diet-induced obesity model of type 2 diabetes. Journal of Biological Chemistry. 296. 100056–100056. 13 indexed citations
12.
Zhu, Yanlong, Benjamin Wancewicz, Michael D. Schaid, et al.. (2020). Ultrahigh-Resolution Mass Spectrometry-Based Platform for Plasma Metabolomics Applied to Type 2 Diabetes Research. Journal of Proteome Research. 20(1). 463–473. 16 indexed citations
13.
Koltes, James E., Itika Arora, R. C. Gupta, et al.. (2019). A gene expression network analysis of the pancreatic islets from lean and obese mice identifies complement 1q like-3 secreted protein as a regulator of β-cell function. Scientific Reports. 9(1). 10119–10119. 9 indexed citations
14.
Gupta, R. C., Dan C. Nguyen, Michael D. Schaid, et al.. (2018). Complement 1q-like-3 protein inhibits insulin secretion from pancreatic β-cells via the cell adhesion G protein–coupled receptor BAI3. Journal of Biological Chemistry. 293(47). 18086–18098. 30 indexed citations
15.
Cummings, Nicole E., Elizabeth M. Williams, Ildikó Kasza, et al.. (2017). Restoration of metabolic health by decreased consumption of branched‐chain amino acids. The Journal of Physiology. 596(4). 623–645. 247 indexed citations
16.
Schaid, Michael D., Jaclyn A. Wisinski, & Michelle E. Kimple. (2017). The EP3 Receptor/Gz Signaling Axis as a Therapeutic Target for Diabetes and Cardiovascular Disease. The AAPS Journal. 19(5). 1276–1283. 12 indexed citations
17.
Fenske, Rachel J., Kathryn A. Carbajal, Michael D. Schaid, et al.. (2017). The Inhibitory G Protein α-Subunit, Gαz, Promotes Type 1 Diabetes-Like Pathophysiology in NOD Mice. Endocrinology. 158(6). 1645–1658. 18 indexed citations
18.
Neuman, Joshua C., Michael D. Schaid, Allison L. Brill, et al.. (2017). Enriching Islet Phospholipids With Eicosapentaenoic Acid Reduces Prostaglandin E2 Signaling and Enhances Diabetic β-Cell Function. Diabetes. 66(6). 1572–1585. 37 indexed citations
19.
Brill, Allison L., Jaclyn A. Wisinski, Mary Thompson, et al.. (2016). Synergy Between Gαz Deficiency and GLP-1 Analog Treatment in Preserving Functional β-Cell Mass in Experimental Diabetes. Molecular Endocrinology. 30(5). 543–556. 21 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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