Michael A. Carpenter

3.3k total citations · 1 hit paper
47 papers, 2.0k citations indexed

About

Michael A. Carpenter is a scholar working on Molecular Biology, Virology and Epidemiology. According to data from OpenAlex, Michael A. Carpenter has authored 47 papers receiving a total of 2.0k indexed citations (citations by other indexed papers that have themselves been cited), including 30 papers in Molecular Biology, 17 papers in Virology and 11 papers in Epidemiology. Recurrent topics in Michael A. Carpenter's work include HIV Research and Treatment (17 papers), CRISPR and Genetic Engineering (10 papers) and Cytomegalovirus and herpesvirus research (8 papers). Michael A. Carpenter is often cited by papers focused on HIV Research and Treatment (17 papers), CRISPR and Genetic Engineering (10 papers) and Cytomegalovirus and herpesvirus research (8 papers). Michael A. Carpenter collaborates with scholars based in United States, United Kingdom and Canada. Michael A. Carpenter's co-authors include Reuben S. Harris, William L. Brown, Emily K. Law, Anurag S. Rathore, Lela Lackey, Nuri A. Temiz, Allison M. Land, Rebecca M. McDougle, Brandon Leonard and Michael B. Burns and has published in prestigious journals such as Nature, Chemical Reviews and Proceedings of the National Academy of Sciences.

In The Last Decade

Michael A. Carpenter

44 papers receiving 2.0k citations

Hit Papers

APOBEC3B is an enzymatic source of mutation in breast cancer 2013 2026 2017 2021 2013 200 400 600

Peers

Michael A. Carpenter
Emily K. Law United States
Zuzana Keckesova United States
Brandon Leonard United States
Rebecca S. LaRue United States
Pierre Fiten Belgium
Jenifer Bear United States
Cristina Bergamaschi United States
Elizabeth Montabana United States
Thomas Eisenreich United States
Emily K. Law United States
Michael A. Carpenter
Citations per year, relative to Michael A. Carpenter Michael A. Carpenter (= 1×) peers Emily K. Law

Countries citing papers authored by Michael A. Carpenter

Since Specialization
Citations

This map shows the geographic impact of Michael A. Carpenter's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael A. Carpenter with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael A. Carpenter more than expected).

Fields of papers citing papers by Michael A. Carpenter

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael A. Carpenter. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael A. Carpenter. The network helps show where Michael A. Carpenter may publish in the future.

Co-authorship network of co-authors of Michael A. Carpenter

This figure shows the co-authorship network connecting the top 25 collaborators of Michael A. Carpenter. A scholar is included among the top collaborators of Michael A. Carpenter based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michael A. Carpenter. Michael A. Carpenter is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Hatton, Grace B., Sally James, Andrew S. Mason, et al.. (2025). Virus-induced APOBEC3 transmutagenesis in bladder cancer initiation. Science Advances. 11(49). eaea6124–eaea6124.
2.
Demir, Özlem, Surl-Hee Ahn, Carla Calvó‐Tusell, et al.. (2025). Regulatory Interactions between APOBEC3B N- and C-Terminal Domains. Journal of Chemical Information and Modeling. 65(7). 3593–3604. 1 indexed citations
4.
Carpenter, Michael A., et al.. (2023). APOBEC3A Catalytic Inactivity Mutation Induces Tertiary Structure Destabilization. ACS Medicinal Chemistry Letters. 14(3). 338–343. 4 indexed citations
5.
Argyris, Prokopios P., Michael A. Carpenter, Harshita B. Gupta, et al.. (2023). DNA Deamination Is Required for Human APOBEC3A-Driven Hepatocellular Carcinoma In Vivo. International Journal of Molecular Sciences. 24(11). 9305–9305. 8 indexed citations
6.
Carpenter, Michael A., et al.. (2022). The current toolbox for APOBEC drug discovery. Trends in Pharmacological Sciences. 43(5). 362–377. 16 indexed citations
7.
Shaban, Nadine M., Rui Yan, Ke Shi, et al.. (2022). Cryo-EM structure of the EBV ribonucleotide reductase BORF2 and mechanism of APOBEC3B inhibition. Science Advances. 8(17). eabm2827–eabm2827. 20 indexed citations
8.
Shi, Ke, Surajit Banerjee, Jennifer L. McCann, et al.. (2021). Structural basis for recognition of distinct deaminated DNA lesions by endonuclease Q. Proceedings of the National Academy of Sciences. 118(10). 13 indexed citations
9.
Law, Emily K., Rena Levin-Klein, Matthew C. Jarvis, et al.. (2020). APOBEC3A catalyzes mutation and drives carcinogenesis in vivo. The Journal of Experimental Medicine. 217(12). 83 indexed citations
10.
Jarvis, Matthew C., Teneale A. Stewart, Jennifer L. McCann, et al.. (2020). Characterization of the mechanism by which the RB/E2F pathway controls expression of the cancer genomic DNA deaminase APOBEC3B. eLife. 9. 24 indexed citations
11.
Wagner, Jeffrey, Özlem Demir, Michael A. Carpenter, et al.. (2018). Determinants of Oligonucleotide Selectivity of APOBEC3B. Journal of Chemical Information and Modeling. 59(5). 2264–2273. 11 indexed citations
12.
Ebrahimi, Diako, Christopher M. Richards, Michael A. Carpenter, et al.. (2018). Genetic and mechanistic basis for APOBEC3H alternative splicing, retrovirus restriction, and counteraction by HIV-1 protease. Nature Communications. 9(1). 4137–4137. 26 indexed citations
13.
Starrett, Gabriel J., Jelmar Quist, Nuri A. Temiz, et al.. (2016). Mutation Processes in 293-Based Clones Overexpressing the DNA Cytosine Deaminase APOBEC3B. PLoS ONE. 11(5). e0155391–e0155391. 23 indexed citations
14.
Starrett, Gabriel J., Elizabeth M. Luengas, Jennifer L. McCann, et al.. (2016). The DNA cytosine deaminase APOBEC3H haplotype I likely contributes to breast and lung cancer mutagenesis. Nature Communications. 7(1). 12918–12918. 121 indexed citations
15.
Shi, Ke, Michael A. Carpenter, Kayo Kurahashi, Reuben S. Harris, & Hideki Aihara. (2015). Crystal Structure of the DNA Deaminase APOBEC3B Catalytic Domain. Journal of Biological Chemistry. 290(47). 28120–28130. 76 indexed citations
16.
Li, Jinhui, Yan Chen, Ming Li, et al.. (2013). APOBEC3 Multimerization Correlates with HIV-1 Packaging and Restriction Activity in Living Cells. Journal of Molecular Biology. 426(6). 1296–1307. 60 indexed citations
17.
Burns, Michael B., Lela Lackey, Michael A. Carpenter, et al.. (2013). APOBEC3B is an enzymatic source of mutation in breast cancer. Nature. 494(7437). 366–370. 639 indexed citations breakdown →
18.
Carpenter, Michael A., et al.. (2010). Determinants of sequence-specificity within human AID and APOBEC3G. DNA repair. 9(5). 579–587. 76 indexed citations
19.
Szczepanowski, Roman H., Michael A. Carpenter, H. Czapinska, et al.. (2008). Central base pair flipping and discrimination by PspGI. Nucleic Acids Research. 36(19). 6109–6117. 27 indexed citations
20.
Carpenter, Michael A.. (2006). Sequence-dependent enhancement of hydrolytic deamination of cytosines in DNA by the restriction enzyme PspGI. Nucleic Acids Research. 34(13). 3762–3770. 14 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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