Mi Bai

1.3k total citations
40 papers, 1.0k citations indexed

About

Mi Bai is a scholar working on Nephrology, Molecular Biology and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, Mi Bai has authored 40 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Nephrology, 19 papers in Molecular Biology and 7 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in Mi Bai's work include Chronic Kidney Disease and Diabetes (8 papers), Renal Diseases and Glomerulopathies (8 papers) and Acute Kidney Injury Research (7 papers). Mi Bai is often cited by papers focused on Chronic Kidney Disease and Diabetes (8 papers), Renal Diseases and Glomerulopathies (8 papers) and Acute Kidney Injury Research (7 papers). Mi Bai collaborates with scholars based in China, United Kingdom and United States. Mi Bai's co-authors include Aihua Zhang, Zhanjun Jia, Songming Huang, Guixia Ding, Min Zhao, Mingzhu Jiang, Shuang Xu, Jiajia Ni, Yue Zhang and Li Yang and has published in prestigious journals such as Journal of Biological Chemistry, SHILAP Revista de lepidopterología and Applied Catalysis B: Environmental.

In The Last Decade

Mi Bai

39 papers receiving 1.0k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mi Bai China 17 531 361 124 123 120 40 1.0k
Yung‐Chien Hsu Taiwan 18 560 1.1× 425 1.2× 182 1.5× 154 1.3× 185 1.5× 41 1.2k
Tangli Xiao China 15 378 0.7× 534 1.5× 120 1.0× 119 1.0× 62 0.5× 29 1.1k
Miguel Fontecha‐Barriuso Spain 14 482 0.9× 277 0.8× 85 0.7× 86 0.7× 138 1.1× 23 932
José Luis Morgado‐Pascual Spain 14 478 0.9× 281 0.8× 80 0.6× 77 0.6× 84 0.7× 23 1.0k
Xiaofen Xiong China 16 592 1.1× 285 0.8× 123 1.0× 232 1.9× 107 0.9× 22 1.2k
Xi Qiao China 18 354 0.7× 321 0.9× 232 1.9× 86 0.7× 72 0.6× 47 1.0k
Pierre Galichon France 17 401 0.8× 341 0.9× 170 1.4× 79 0.6× 66 0.6× 47 991
Jianwei Tian China 24 506 1.0× 605 1.7× 145 1.2× 146 1.2× 173 1.4× 66 1.5k
Seon Ho Ahn South Korea 7 546 1.0× 446 1.2× 177 1.4× 199 1.6× 117 1.0× 11 1.2k
Guangyi Liu China 14 351 0.7× 183 0.5× 104 0.8× 95 0.8× 96 0.8× 30 871

Countries citing papers authored by Mi Bai

Since Specialization
Citations

This map shows the geographic impact of Mi Bai's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mi Bai with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mi Bai more than expected).

Fields of papers citing papers by Mi Bai

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mi Bai. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mi Bai. The network helps show where Mi Bai may publish in the future.

Co-authorship network of co-authors of Mi Bai

This figure shows the co-authorship network connecting the top 25 collaborators of Mi Bai. A scholar is included among the top collaborators of Mi Bai based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mi Bai. Mi Bai is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Wu, Mengqiu, Yuting Li, Jiaojiao Fan, et al.. (2025). Targeting renal tubular WWP2 to restore mitochondrial OXPHOS integrity retards the AKI-to-CKD transition. Molecular Therapy. 34(3). 1576–1596. 1 indexed citations
3.
Yang, Yunwen, Peipei Wang, Kaiqian Zhou, et al.. (2025). HUWE1‐Mediated Degradation of MUTYH Facilitates DNA Damage and Mitochondrial Dysfunction to Promote Acute Kidney Injury. Advanced Science. 12(13). e2412250–e2412250. 1 indexed citations
4.
Xu, Xinyue, Mi Bai, Jiaojiao Fan, et al.. (2024). Activation of LONP1 by 84-B10 alleviates aristolochic acid nephropathy via re-establishing mitochondrial and peroxisomal homeostasis. Chinese Journal of Natural Medicines. 22(9). 808–821. 2 indexed citations
5.
You, Ran, Yanwei Li, Wei Zhou, et al.. (2024). WWP2 deletion aggravates acute kidney injury by targeting CDC20/autophagy axis. Journal of Advanced Research. 71. 471–485. 4 indexed citations
6.
Song, Chang Myeon, Mi Bai, Wen Su, et al.. (2024). Genetic or pharmacologic blockade of mPGES-2 attenuates renal lipotoxicity and diabetic kidney disease by targeting Rev-Erbα/FABP5 signaling. Cell Reports. 43(4). 114075–114075. 4 indexed citations
7.
Jiang, Qingsong, Zhijie Chen, Mi Bai, et al.. (2024). Upcycling of waste polyethylene terephthalate (PET) into CoFe@C for highly efficient PV-driven bifunctional seawater splitting via a “waste materialization” strategy. Applied Catalysis B: Environmental. 362. 124756–124756. 10 indexed citations
8.
Bai, Mi, Shuang Xu, Mingzhu Jiang, et al.. (2024). Meis1 Targets Protein Tyrosine Phosphatase Receptor J in Fibroblast to Retard Chronic Kidney Disease Progression. Advanced Science. 11(39). e2309754–e2309754. 5 indexed citations
9.
He, Jia, Shuang Xu, Mingzhu Jiang, et al.. (2023). CDC20 inhibition alleviates fibrotic response of renal tubular epithelial cells and fibroblasts by regulating nuclear translocation of β-catenin. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1869(4). 166663–166663. 1 indexed citations
10.
Tang, Dan, Chun Loo Gan, Mi Bai, et al.. (2023). Novel variants in CRB2 targeting the malfunction of slit diaphragm related to focal segmental glomerulosclerosis. Pediatric Nephrology. 39(1). 149–165. 3 indexed citations
11.
Bai, Mi, Mengqiu Wu, Mingzhu Jiang, et al.. (2023). LONP1 targets HMGCS2 to protect mitochondrial function and attenuate chronic kidney disease. EMBO Molecular Medicine. 15(2). e16581–e16581. 48 indexed citations
12.
13.
Jiang, Mingzhu, Mi Bai, Shuang Xu, et al.. (2021). Blocking AURKA with MK-5108 attenuates renal fibrosis in chronic kidney disease. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1867(11). 166227–166227. 8 indexed citations
14.
Jiang, Mingzhu, Min Zhao, Mi Bai, et al.. (2021). SIRT1 Alleviates Aldosterone-Induced Podocyte Injury by Suppressing Mitochondrial Dysfunction and NLRP3 Inflammasome Activation. SHILAP Revista de lepidopterología. 7(4). 293–305. 14 indexed citations
15.
Bai, Mi, et al.. (2019). Risk factors associated with coronary heart disease in women: a systematic review. Herz. 45(S1). 52–57. 27 indexed citations
16.
Bai, Mi, Huimei Chen, Dan Ding, et al.. (2019). MicroRNA-214 promotes chronic kidney disease by disrupting mitochondrial oxidative phosphorylation. Kidney International. 95(6). 1389–1404. 80 indexed citations
17.
Zhao, Min, Mi Bai, Guixia Ding, et al.. (2018). Angiotensin II Stimulates the NLRP3 Inflammasome to Induce Podocyte Injury and Mitochondrial Dysfunction. Kidney Diseases. 4(2). 83–94. 65 indexed citations
18.
Guo, Yan, Jiajia Ni, Shuang Chen, et al.. (2017). MicroRNA-709 Mediates Acute Tubular Injury through Effects on Mitochondrial Function. Journal of the American Society of Nephrology. 29(2). 449–461. 90 indexed citations
19.
Bai, Mi, et al.. (2015). Reactive Oxygen Species-initiated Autophagy Opposes Aldosterone-induced Podocyte Injury. SHILAP Revista de lepidopterología. 17(2). S87–S87. 4 indexed citations
20.
Zhu, Chunhua, Ruochen Che, Guixia Ding, et al.. (2014). Dysfunction of the PGC-1α-mitochondria axis confers adriamycin-induced podocyte injury. American Journal of Physiology-Renal Physiology. 306(12). F1410–F1417. 33 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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