Mengdie Dong

484 total citations · 1 hit paper
11 papers, 282 citations indexed

About

Mengdie Dong is a scholar working on Molecular Biology, Immunology and Cancer Research. According to data from OpenAlex, Mengdie Dong has authored 11 papers receiving a total of 282 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 4 papers in Immunology and 4 papers in Cancer Research. Recurrent topics in Mengdie Dong's work include Ubiquitin and proteasome pathways (4 papers), RNA modifications and cancer (3 papers) and interferon and immune responses (3 papers). Mengdie Dong is often cited by papers focused on Ubiquitin and proteasome pathways (4 papers), RNA modifications and cancer (3 papers) and interferon and immune responses (3 papers). Mengdie Dong collaborates with scholars based in China, United Kingdom and South Korea. Mengdie Dong's co-authors include Quanwen Yin, Hong Jiang, Longbin Zheng, Hongshan Chen, Yunjia Zhang, Min Jiao, Minghong Chen, Xuesong Li, Minghong Chen and Xiang Chen and has published in prestigious journals such as Circulation, Biochemical and Biophysical Research Communications and European Heart Journal.

In The Last Decade

Mengdie Dong

11 papers receiving 281 citations

Hit Papers

TRAP1 drives smooth muscle cell senescence and promotes a... 2024 2026 2025 2024 20 40 60

Peers

Mengdie Dong
Ana R. Coelho Portugal
Rituparna Ganguly United States
Xiaoming Zou United States
Na Yao China
Mengdie Dong
Citations per year, relative to Mengdie Dong Mengdie Dong (= 1×) peers Christopher M. Schafer

Countries citing papers authored by Mengdie Dong

Since Specialization
Citations

This map shows the geographic impact of Mengdie Dong's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mengdie Dong with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mengdie Dong more than expected).

Fields of papers citing papers by Mengdie Dong

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mengdie Dong. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mengdie Dong. The network helps show where Mengdie Dong may publish in the future.

Co-authorship network of co-authors of Mengdie Dong

This figure shows the co-authorship network connecting the top 25 collaborators of Mengdie Dong. A scholar is included among the top collaborators of Mengdie Dong based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mengdie Dong. Mengdie Dong is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
1.
Dong, Mengdie, Minghong Chen, Min Jiao, et al.. (2024). Oscillatory shear stress promotes endothelial senescence and atherosclerosis via STING activation. Biochemical and Biophysical Research Communications. 715. 149979–149979. 10 indexed citations
2.
Dong, Mengdie, Yunjia Zhang, Minghong Chen, et al.. (2024). ASF1A-dependent P300-mediated histone H3 lysine 18 lactylation promotes atherosclerosis by regulating EndMT. Acta Pharmaceutica Sinica B. 14(7). 3027–3048. 45 indexed citations
3.
Zheng, Longbin, Xiang Chen, Xian He, et al.. (2024). METTL4-Mediated Mitochondrial DNA N6-Methyldeoxyadenosine Promoting Macrophage Inflammation and Atherosclerosis. Circulation. 151(13). 946–965. 19 indexed citations
4.
Zhang, Yunjia, Hong Jiang, Mengdie Dong, et al.. (2024). Macrophage MCT4 inhibition activates reparative genes and protects from atherosclerosis by histone H3 lysine 18 lactylation. Cell Reports. 43(5). 114180–114180. 51 indexed citations
5.
Li, Xuesong, Minghong Chen, Min Jiao, et al.. (2024). TRAP1 drives smooth muscle cell senescence and promotes atherosclerosis via HDAC3-primed histone H4 lysine 12 lactylation. European Heart Journal. 45(39). 4219–4235. 70 indexed citations breakdown →
6.
Li, Xuesong, Xiang Chen, Longbin Zheng, et al.. (2023). Non-canonical STING–PERK pathway dependent epigenetic regulation of vascular endothelial dysfunction via integrating IRF3 and NF-κB in inflammatory response. Acta Pharmaceutica Sinica B. 13(12). 4765–4784. 31 indexed citations
7.
Gu, Jiaming, Jiajing Chen, Quanwen Yin, et al.. (2023). lncRNA JPX-Enriched Chromatin Microenvironment Mediates Vascular Smooth Muscle Cell Senescence and Promotes Atherosclerosis. Arteriosclerosis Thrombosis and Vascular Biology. 44(1). 156–176. 17 indexed citations
8.
Zheng, Longbin, Xiang Chen, Quanwen Yin, et al.. (2022). RNA-m6A modification of HDGF mediated by Mettl3 aggravates the progression of atherosclerosis by regulating macrophages polarization via energy metabolism reprogramming. Biochemical and Biophysical Research Communications. 635. 120–127. 15 indexed citations
9.
Dong, Mengdie, et al.. (2022). Highly enantioselective δ-protonation and formal [3 + 3] annulation promoted by N-heterocyclic carbene. Organic Chemistry Frontiers. 9(11). 3039–3044. 7 indexed citations
10.
Duan, Xiaoyong, et al.. (2021). Highly Enantioselective Synthesis of Pyrroloindolones and Pyrroloquinolinones via an N-Heterocyclic Carbene-Catalyzed Cascade Reaction. Organic Letters. 23(9). 3777–3781. 14 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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